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Items: 6

1.

Protein kinase N controls a lysosomal lipid switch to facilitate nutrient signalling via mTORC1.

Wallroth A, Koch PA, Marat AL, Krause E, Haucke V.

Nat Cell Biol. 2019 Sep;21(9):1093-1101. doi: 10.1038/s41556-019-0377-3. Epub 2019 Aug 26.

PMID:
31451768
2.

[Components of the joint-sparing, combined bony and soft tissue correction of the cavovarus foot].

Wallroth A, Dreher T.

Oper Orthop Traumatol. 2018 Aug;30(4):286-292. doi: 10.1007/s00064-018-0552-x. Epub 2018 Jun 21. Review. German.

PMID:
29931378
3.

Phosphoinositide conversion in endocytosis and the endolysosomal system.

Wallroth A, Haucke V.

J Biol Chem. 2018 Feb 2;293(5):1526-1535. doi: 10.1074/jbc.R117.000629. Epub 2017 Dec 27. Review.

4.

A lipid off-switch for mTORC1.

Wallroth A, Haucke V.

Mol Cell Oncol. 2017 Aug 2;4(5):e1356899. doi: 10.1080/23723556.2017.1356899. eCollection 2017.

5.

mTORC1 activity repression by late endosomal phosphatidylinositol 3,4-bisphosphate.

Marat AL, Wallroth A, Lo WT, Müller R, Norata GD, Falasca M, Schultz C, Haucke V.

Science. 2017 Jun 2;356(6341):968-972. doi: 10.1126/science.aaf8310.

PMID:
28572395
6.

Cavovarus deformity in Charcot-Marie-Tooth disease: is there a hindfoot equinus deformity that needs treatment?

Beckmann NA, Wolf SI, Heitzmann D, Wallroth A, Müller S, Dreher T.

J Foot Ankle Res. 2015 Nov 26;8:65. doi: 10.1186/s13047-015-0121-6. eCollection 2015.

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