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Items: 10

1.

A CD40 Agonist and PD-1 Antagonist Antibody Reprogram the Microenvironment of Nonimmunogenic Tumors to Allow T-cell-Mediated Anticancer Activity.

Ma HS, Poudel B, Torres ER, Sidhom JW, Robinson TM, Christmas B, Scott B, Cruz K, Woolman S, Wall VZ, Armstrong T, Jaffee EM.

Cancer Immunol Res. 2019 Mar;7(3):428-442. doi: 10.1158/2326-6066.CIR-18-0061. Epub 2019 Jan 14.

PMID:
30642833
2.

Smooth muscle glucose metabolism promotes monocyte recruitment and atherosclerosis in a mouse model of metabolic syndrome.

Wall VZ, Barnhart S, Kanter JE, Kramer F, Shimizu-Albergine M, Adhikari N, Wight TN, Hall JL, Bornfeldt KE.

JCI Insight. 2018 Jun 7;3(11). pii: 96544. doi: 10.1172/jci.insight.96544. eCollection 2018 Jun 7.

3.

Inflammatory stimuli induce acyl-CoA thioesterase 7 and remodeling of phospholipids containing unsaturated long (≥C20)-acyl chains in macrophages.

Wall VZ, Barnhart S, Kramer F, Kanter JE, Vivekanandan-Giri A, Pennathur S, Bolego C, Ellis JM, Gijón MA, Wolfgang MJ, Bornfeldt KE.

J Lipid Res. 2017 Jun;58(6):1174-1185. doi: 10.1194/jlr.M076489. Epub 2017 Apr 17.

4.

Arterial smooth muscle.

Wall VZ, Bornfeldt KE.

Arterioscler Thromb Vasc Biol. 2014 Oct;34(10):2175-9. doi: 10.1161/ATVBAHA.114.304441. No abstract available.

5.

Testing the role of myeloid cell glucose flux in inflammation and atherosclerosis.

Nishizawa T, Kanter JE, Kramer F, Barnhart S, Shen X, Vivekanandan-Giri A, Wall VZ, Kowitz J, Devaraj S, O'Brien KD, Pennathur S, Tang J, Miyaoka RS, Raines EW, Bornfeldt KE.

Cell Rep. 2014 Apr 24;7(2):356-365. doi: 10.1016/j.celrep.2014.03.028. Epub 2014 Apr 13.

6.

mTOR inhibition alleviates mitochondrial disease in a mouse model of Leigh syndrome.

Johnson SC, Yanos ME, Kayser EB, Quintana A, Sangesland M, Castanza A, Uhde L, Hui J, Wall VZ, Gagnidze A, Oh K, Wasko BM, Ramos FJ, Palmiter RD, Rabinovitch PS, Morgan PG, Sedensky MM, Kaeberlein M.

Science. 2013 Dec 20;342(6165):1524-8. doi: 10.1126/science.1244360. Epub 2013 Nov 14.

7.

Acyl-CoA synthetase 1 is induced by Gram-negative bacteria and lipopolysaccharide and is required for phospholipid turnover in stimulated macrophages.

Rubinow KB, Wall VZ, Nelson J, Mar D, Bomsztyk K, Askari B, Lai MA, Smith KD, Han MS, Vivekanandan-Giri A, Pennathur S, Albert CJ, Ford DA, Davis RJ, Bornfeldt KE.

J Biol Chem. 2013 Apr 5;288(14):9957-70. doi: 10.1074/jbc.M113.458372. Epub 2013 Feb 20.

8.

The MDT-15 subunit of mediator interacts with dietary restriction to modulate longevity and fluoranthene toxicity in Caenorhabditis elegans.

Schleit J, Wall VZ, Simko M, Kaeberlein M.

PLoS One. 2011;6(11):e28036. doi: 10.1371/journal.pone.0028036. Epub 2011 Nov 21.

9.

A behavioral genetics approach to understanding D1 receptor involvement in phasic dopamine signaling.

Wall VZ, Parker JG, Fadok JP, Darvas M, Zweifel L, Palmiter RD.

Mol Cell Neurosci. 2011 Jan;46(1):21-31. doi: 10.1016/j.mcn.2010.09.011. Epub 2010 Oct 1. Review.

10.

Disruption of NMDAR-dependent burst firing by dopamine neurons provides selective assessment of phasic dopamine-dependent behavior.

Zweifel LS, Parker JG, Lobb CJ, Rainwater A, Wall VZ, Fadok JP, Darvas M, Kim MJ, Mizumori SJ, Paladini CA, Phillips PE, Palmiter RD.

Proc Natl Acad Sci U S A. 2009 May 5;106(18):7281-8. doi: 10.1073/pnas.0813415106. Epub 2009 Apr 2.

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