Format
Sort by
Items per page

Send to

Choose Destination

Search results

Items: 17

1.

Activity of a nitric oxide-generating wound treatment system against wound pathogen biofilms.

Waite RD, Stewart JE, Stephen AS, Allaker RP.

Int J Antimicrob Agents. 2018 Sep;52(3):338-343. doi: 10.1016/j.ijantimicag.2018.04.009. Epub 2018 Apr 14.

PMID:
29665443
2.
3.
4.

Synergy between Colistin and the Signal Peptidase Inhibitor MD3 Is Dependent on the Mechanism of Colistin Resistance in Acinetobacter baumannii.

Martínez-Guitián M, Vázquez-Ucha JC, Odingo J, Parish T, Poza M, Waite RD, Bou G, Wareham DW, Beceiro A.

Antimicrob Agents Chemother. 2016 Jun 20;60(7):4375-9. doi: 10.1128/AAC.00510-16. Print 2016 Jul.

5.

Environment and colonisation sequence are key parameters driving cooperation and competition between Pseudomonas aeruginosa cystic fibrosis strains and oral commensal streptococci.

Whiley RA, Fleming EV, Makhija R, Waite RD.

PLoS One. 2015 Feb 24;10(2):e0115513. doi: 10.1371/journal.pone.0115513. eCollection 2015.

6.

Activity of the type I signal peptidase inhibitor MD3 against multidrug-resistant Gram-negative bacteria alone and in combination with colistin.

Personne Y, Curtis MA, Wareham DW, Waite RD.

J Antimicrob Chemother. 2014 Dec;69(12):3236-43. doi: 10.1093/jac/dku309. Epub 2014 Aug 18.

PMID:
25134721
7.

Differential potentiation of the virulence of the Pseudomonas aeruginosa cystic fibrosis liverpool epidemic strain by oral commensal Streptococci.

Whiley RA, Sheikh NP, Mushtaq N, Hagi-Pavli E, Personne Y, Javaid D, Waite RD.

J Infect Dis. 2014 Mar 1;209(5):769-80. doi: 10.1093/infdis/jit568. Epub 2013 Oct 24.

PMID:
24158959
8.

LacR mutations are frequently observed in Streptococcus intermedius and are responsible for increased intermedilysin production and virulence.

Tomoyasu T, Imaki H, Masuda S, Okamoto A, Kim H, Waite RD, Whiley RA, Kikuchi K, Hiramatsu K, Tabata A, Nagamune H.

Infect Immun. 2013 Sep;81(9):3276-86. doi: 10.1128/IAI.00638-13. Epub 2013 Jun 24.

9.

Pseudomonas aeruginosa possesses two putative type I signal peptidases, LepB and PA1303, each with distinct roles in physiology and virulence.

Waite RD, Rose RS, Rangarajan M, Aduse-Opoku J, Hashim A, Curtis MA.

J Bacteriol. 2012 Sep;194(17):4521-36. doi: 10.1128/JB.06678-11. Epub 2012 Jun 22.

10.

A simple, semiselective medium for anaerobic isolation of anginosus group streptococci from patients with chronic lung disease.

Waite RD, Wareham DW, Gardiner S, Whiley RA.

J Clin Microbiol. 2012 Apr;50(4):1430-2. doi: 10.1128/JCM.06184-11. Epub 2012 Jan 11.

11.

Comparative microarray analysis reveals that the core biofilm-associated transcriptome of Pseudomonas aeruginosa comprises relatively few genes.

Patell S, Gu M, Davenport P, Givskov M, Waite RD, Welch M.

Environ Microbiol Rep. 2010 Jun;2(3):440-8. doi: 10.1111/j.1758-2229.2010.00158.x. Epub 2010 Mar 30.

PMID:
23766118
12.

Pseudomonas aeruginosa PAO1 pyocin production affects population dynamics within mixed-culture biofilms.

Waite RD, Curtis MA.

J Bacteriol. 2009 Feb;191(4):1349-54. doi: 10.1128/JB.01458-08. Epub 2008 Dec 5.

13.

Topographic distribution of bacteria associated with oral malodour on the tongue.

Allaker RP, Waite RD, Hickling J, North M, McNab R, Bosma MP, Hughes FJ.

Arch Oral Biol. 2008 Apr;53 Suppl 1:S8-S12. doi: 10.1016/S0003-9969(08)70003-7.

PMID:
18460402
14.

Clustering of Pseudomonas aeruginosa transcriptomes from planktonic cultures, developing and mature biofilms reveals distinct expression profiles.

Waite RD, Paccanaro A, Papakonstantinopoulou A, Hurst JM, Saqi M, Littler E, Curtis MA.

BMC Genomics. 2006 Jun 26;7:162.

15.
16.

Supplemental Content

Loading ...
Support Center