Sort by
Items per page

Send to

Choose Destination

Search results

Items: 9


The stromal cell-derived factor-1alpha/CXCR4 ligand-receptor axis is critical for progenitor survival and migration in the pancreas.

Kayali AG, Van Gunst K, Campbell IL, Stotland A, Kritzik M, Liu G, Flodström-Tullberg M, Zhang YQ, Sarvetnick N.

J Cell Biol. 2003 Nov 24;163(4):859-69.


H-2D end confers dominant protection from IL-10-mediated acceleration of autoimmune diabetes in the nonobese diabetic mouse.

La Cava A, Balasa B, Good A, van Gunst K, Jung N, Sarvetnick N.

J Immunol. 2001 Jul 15;167(2):1066-71.


Germ line deletion of the CD1 locus exacerbates diabetes in the NOD mouse.

Shi FD, Flodstrom M, Balasa B, Kim SH, Van Gunst K, Strominger JL, Wilson SB, Sarvetnick N.

Proc Natl Acad Sci U S A. 2001 Jun 5;98(12):6777-82.


Vaccination with glutamic acid decarboxylase plasmid DNA protects mice from spontaneous autoimmune diabetes and B7/CD28 costimulation circumvents that protection.

Balasa B, Boehm BO, Fortnagel A, Karges W, Van Gunst K, Jung N, Camacho SA, Webb SR, Sarvetnick N.

Clin Immunol. 2001 May;99(2):241-52.


A mechanism for IL-10-mediated diabetes in the nonobese diabetic (NOD) mouse: ICAM-1 deficiency blocks accelerated diabetes.

Balasa B, La Cava A, Van Gunst K, Mocnik L, Balakrishna D, Nguyen N, Tucker L, Sarvetnick N.

J Immunol. 2000 Dec 15;165(12):7330-7.


Islet-specific expression of IL-10 promotes diabetes in nonobese diabetic mice independent of Fas, perforin, TNF receptor-1, and TNF receptor-2 molecules.

Balasa B, Van Gunst K, Jung N, Balakrishna D, Santamaria P, Hanafusa T, Itoh N, Sarvetnick N.

J Immunol. 2000 Sep 1;165(5):2841-9.


IL-10 deficiency does not inhibit insulitis and accelerates cyclophosphamide-induced diabetes in the nonobese diabetic mouse.

Balasa B, Van Gunst K, Jung N, Katz JD, Sarvetnick N.

Cell Immunol. 2000 Jun 15;202(2):97-102.


Supplemental Content

Loading ...
Support Center