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Items: 43

1.

The PARP inhibitor olaparib exerts beneficial effects in mice subjected to cecal ligature and puncture and in cells subjected to oxidative stress without impairing DNA integrity: A potential opportunity for repurposing a clinically used oncological drug for the experimental therapy of sepsis.

Ahmad A, Vieira JC, de Mello AH, de Lima TM, Ariga SK, Barbeiro DF, Barbeiro HV, Szczesny B, Törö G, Druzhyna N, Randi EB, Marcatti M, Toliver-Kinsky T, Kiss A, Liaudet L, Salomao R, Soriano FG, Szabo C.

Pharmacol Res. 2019 Jul;145:104263. doi: 10.1016/j.phrs.2019.104263. Epub 2019 May 6.

PMID:
31071432
2.

H2S, a Bacterial Defense Mechanism against the Host Immune Response.

Toliver-Kinsky T, Cui W, Törö G, Lee SJ, Shatalin K, Nudler E, Szabo C.

Infect Immun. 2018 Dec 19;87(1). pii: e00272-18. doi: 10.1128/IAI.00272-18. Print 2019 Jan.

3.

Fms-Like Tyrosine Kinase-3 Ligand Attenuates Local and Systemic Infection in a Model of Post-Burn Pneumonia.

Hundeshagen G, Cui W, Musgrove L, Cherry A, Lee SJ, Cox RA, Toliver-Kinsky T.

Shock. 2018 Jun;49(6):721-727. doi: 10.1097/SHK.0000000000000964.

PMID:
28796661
4.

Burn Trauma Acutely Increases the Respiratory Capacity and Function of Liver Mitochondria.

Bohanon FJ, Nunez Lopez O, Herndon DN, Wang X, Bhattarai N, Ayadi AE, Prasai A, Jay JW, Rojas-Khalil Y, Toliver-Kinsky TE, Finnerty CC, Radhakrishnan RS, Porter C.

Shock. 2018 Apr;49(4):466-473. doi: 10.1097/SHK.0000000000000935.

5.

Fms-like tyrosine kinase-3 ligand increases resistance to burn wound infection through effects on plasmacytoid dendritic cells.

Bae L, Bohannon JK, Cui W, Vinish M, Toliver-Kinsky T.

BMC Immunol. 2017 Feb 22;18(1):9. doi: 10.1186/s12865-016-0188-2.

6.

Human and Mouse Brown Adipose Tissue Mitochondria Have Comparable UCP1 Function.

Porter C, Herndon DN, Chondronikola M, Chao T, Annamalai P, Bhattarai N, Saraf MK, Capek KD, Reidy PT, Daquinag AC, Kolonin MG, Rasmussen BB, Borsheim E, Toliver-Kinsky T, Sidossis LS.

Cell Metab. 2016 Aug 9;24(2):246-55. doi: 10.1016/j.cmet.2016.07.004.

7.

Time-Dependent and Organ-Specific Changes in Mitochondrial Function, Mitochondrial DNA Integrity, Oxidative Stress and Mononuclear Cell Infiltration in a Mouse Model of Burn Injury.

Szczesny B, Brunyánszki A, Ahmad A, Oláh G, Porter C, Toliver-Kinsky T, Sidossis L, Herndon DN, Szabo C.

PLoS One. 2015 Dec 2;10(12):e0143730. doi: 10.1371/journal.pone.0143730. eCollection 2015.

8.

Differential acute and chronic effects of burn trauma on murine skeletal muscle bioenergetics.

Porter C, Herndon DN, Bhattarai N, Ogunbileje JO, Szczesny B, Szabo C, Toliver-Kinsky T, Sidossis LS.

Burns. 2016 Feb;42(1):112-122. doi: 10.1016/j.burns.2015.10.026. Epub 2015 Nov 23.

9.

Dendritic cells modulate burn wound healing by enhancing early proliferation.

Vinish M, Cui W, Stafford E, Bae L, Hawkins H, Cox R, Toliver-Kinsky T.

Wound Repair Regen. 2016 Jan-Feb;24(1):6-13. doi: 10.1111/wrr.12388. Epub 2016 Jan 19.

PMID:
26609910
10.

Morphological Changes in Subcutaneous White Adipose Tissue After Severe Burn Injury.

Saraf MK, Herndon DN, Porter C, Toliver-Kinsky T, Radhakrishnan R, Chao T, Chondronikola M, Sidossis LS.

J Burn Care Res. 2016 Mar-Apr;37(2):e96-103. doi: 10.1097/BCR.0000000000000292.

11.

Browning of Subcutaneous White Adipose Tissue in Humans after Severe Adrenergic Stress.

Sidossis LS, Porter C, Saraf MK, Børsheim E, Radhakrishnan RS, Chao T, Ali A, Chondronikola M, Mlcak R, Finnerty CC, Hawkins HK, Toliver-Kinsky T, Herndon DN.

Cell Metab. 2015 Aug 4;22(2):219-27. doi: 10.1016/j.cmet.2015.06.022.

12.

Severe Burn Injury Induces Thermogenically Functional Mitochondria in Murine White Adipose Tissue.

Porter C, Herndon DN, Bhattarai N, Ogunbileje JO, Szczesny B, Szabo C, Toliver-Kinsky T, Sidossis LS.

Shock. 2015 Sep;44(3):258-64. doi: 10.1097/SHK.0000000000000410.

13.

The role of CXCL10 in the pathogenesis of experimental septic shock.

Herzig DS, Luan L, Bohannon JK, Toliver-Kinsky TE, Guo Y, Sherwood ER.

Crit Care. 2014 Jun 2;18(3):R113. doi: 10.1186/cc13902.

14.

Therapeutic efficacy of CXCR3 blockade in an experimental model of severe sepsis.

Herzig DS, Guo Y, Fang G, Toliver-Kinsky TE, Sherwood ER.

Crit Care. 2012 Sep 19;16(5):R168. doi: 10.1186/cc11642.

15.

STAT1-deficient mice are resistant to cecal ligation and puncture-induced septic shock.

Herzig D, Fang G, Toliver-Kinsky TE, Guo Y, Bohannon J, Sherwood ER.

Shock. 2012 Oct;38(4):395-402. doi: 10.1097/SHK.0b013e318265a2ab.

16.

Regulation of lymphocyte trafficking by CXC chemokine receptor 3 during septic shock.

Herzig DS, Driver BR, Fang G, Toliver-Kinsky TE, Shute EN, Sherwood ER.

Am J Respir Crit Care Med. 2012 Feb 1;185(3):291-300. doi: 10.1164/rccm.201108-1560OC. Epub 2011 Dec 1.

17.

Dendritic cell modification of neutrophil responses to infection after burn injury.

Bohannon J, Cui W, Sherwood E, Toliver-Kinsky T.

J Immunol. 2010 Sep 1;185(5):2847-53. doi: 10.4049/jimmunol.0903619. Epub 2010 Aug 2.

18.

The role of interferon-γ in the pathogenesis of acute intra-abdominal sepsis.

Romero CR, Herzig DS, Etogo A, Nunez J, Mahmoudizad R, Fang G, Murphey ED, Toliver-Kinsky T, Sherwood ER.

J Leukoc Biol. 2010 Oct;88(4):725-35. doi: 10.1189/jlb.0509307. Epub 2010 Jul 13.

19.

Insulin increases resistance to burn wound infection-associated sepsis.

Gauglitz GG, Toliver-Kinsky TE, Williams FN, Song J, Cui W, Herndon DN, Jeschke MG.

Crit Care Med. 2010 Jan;38(1):202-8. doi: 10.1097/CCM.0b013e3181b43236.

20.

Fms-like tyrosine kinase-3 ligand alters antigen-specific responses to infections after severe burn injury.

Bohannon J, Fang G, Cui W, Sherwood E, Toliver-Kinsky T.

Shock. 2009 Oct;32(4):435-41. doi: 10.1097/SHK.0b013e31819e2937.

21.

Endogenous Fms-like tyrosine kinase-3 ligand levels are not altered in mice after a severe burn and infection.

Bohannon JK, Cui W, Toliver-Kinsky T.

BMC Immunol. 2009 Aug 28;10:47. doi: 10.1186/1471-2172-10-47.

22.

NK but not CD1-restricted NKT cells facilitate systemic inflammation during polymicrobial intra-abdominal sepsis.

Etogo AO, Nunez J, Lin CY, Toliver-Kinsky TE, Sherwood ER.

J Immunol. 2008 May 1;180(9):6334-45.

23.

Prophylactic treatment with fms-like tyrosine kinase-3 ligand after burn injury enhances global immune responses to infection.

Bohannon J, Cui W, Cox R, Przkora R, Sherwood E, Toliver-Kinsky T.

J Immunol. 2008 Mar 1;180(5):3038-48.

24.

Mice depleted of alphabeta but not gammadelta T cells are resistant to mortality caused by cecal ligation and puncture.

Enoh VT, Lin SH, Lin CY, Toliver-Kinsky T, Murphey ED, Varma TK, Sherwood ER.

Shock. 2007 May;27(5):507-19.

PMID:
17438456
25.

Effect of transforming growth factor-beta neutralization on survival and bacterial clearance in a murine model of Pseudomonas aeruginosa burn wound infection.

Huang Z, Pereira C, Toliver-Kinsky T, Murphey ED, Varma TK, Lin CY, Herndon DN, Sherwood ER.

J Burn Care Res. 2006 Sep-Oct;27(5):682-7.

PMID:
16998401
26.

Endotoxin priming improves clearance of Pseudomonas aeruginosa in wild-type and interleukin-10 knockout mice.

Varma TK, Durham M, Murphey ED, Cui W, Huang Z, Lin CY, Toliver-Kinsky T, Sherwood ER.

Infect Immun. 2005 Nov;73(11):7340-7.

27.

Glucan phosphate treatment attenuates burn-induced inflammation and improves resistance to Pseudomonas aeruginosa burn wound infection.

Lyuksutova OI, Murphey ED, Toliver-Kinsky TE, Lin CY, Cui W, Williams DL, Sherwood ER.

Shock. 2005 Mar;23(3):224-32.

PMID:
15718919
28.
29.

Mechanisms of the inflammatory response.

Sherwood ER, Toliver-Kinsky T.

Best Pract Res Clin Anaesthesiol. 2004 Sep;18(3):385-405. Review.

PMID:
15212335
30.
31.
32.

Hyperoxia increases AP-1 DNA binding in rat brain.

Tong L, Toliver-Kinsky T, Rassin D, Werrbach-Perez K, Perez-Polo JR.

Neurochem Res. 2003 Jan;28(1):111-5.

PMID:
12587669
33.

NF-kappaB activity decreases in basal forebrain of young and aged rats after hyperoxia.

Toliver-Kinsky T, Rassin D, Perez-Polo JR.

Neurobiol Aging. 2002 Sep-Oct;23(5):899-905.

PMID:
12392794
34.

Interferon-gamma production is suppressed in thermally injured mice: decreased production of regulatory cytokines and corresponding receptors.

Toliver-Kinsky TE, Varma TK, Lin CY, Herndon DN, Sherwood ER.

Shock. 2002 Oct;18(4):322-30.

PMID:
12392275
35.

Attenuated transcriptional responses to oxidative stress in the aged rat brain.

Tong L, Toliver-Kinsky T, Edwards M, Rassin DK, Werrbach-Perez K, Perez-Polo JR.

J Neurosci Res. 2002 Nov 1;70(3):318-26.

PMID:
12391591
36.

Endotoxin-induced gamma interferon production: contributing cell types and key regulatory factors.

Varma TK, Lin CY, Toliver-Kinsky TE, Sherwood ER.

Clin Diagn Lab Immunol. 2002 May;9(3):530-43.

37.

Glucan phosphate potentiates endotoxin-induced interferon-gamma expression in immunocompetent mice, but attenuates induction of endotoxin tolerance.

Sherwood ER, Varma TK, Fram RY, Lin CY, Koutrouvelis AP, Toliver-Kinsky TE.

Clin Sci (Lond). 2001 Dec;101(6):541-50.

PMID:
11724637
38.

Cellular mechanisms that cause suppressed gamma interferon secretion in endotoxin-tolerant mice.

Varma TK, Toliver-Kinsky TE, Lin CY, Koutrouvelis AP, Nichols JE, Sherwood ER.

Infect Immun. 2001 Sep;69(9):5249-63.

40.

NGF-mediated alteration of NF-kappaB binding activity after partial immunolesions to rat cholinergic basal forebrain neurons.

Gu Z, Toliver-Kinsky T, Glasgow J, Werrbach-Perez K, Perez-Polo JR.

Int J Dev Neurosci. 2000 Jul-Aug;18(4-5):455-68.

PMID:
10817930
41.

Signal transduction in neuronal death.

Tong L, Toliver-Kinsky T, Taglialatela G, Werrbach-Perez K, Wood T, Perez-Polo JR.

J Neurochem. 1998 Aug;71(2):447-59. Review.

42.

Responses of young and aged rat CNS to partial cholinergic immunolesions and NGF treatment.

Wörtwein G, Yu J, Toliver-Kinsky T, Perez-Polo JR.

J Neurosci Res. 1998 May 1;52(3):322-33.

PMID:
9590440
43.

Age-associated alterations in hippocampal and basal forebrain nuclear factor kappa B activity.

Toliver-Kinsky T, Papaconstantinou J, Perez-Polo JR.

J Neurosci Res. 1997 Jun 15;48(6):580-7.

PMID:
9210528

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