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Items: 5

1.

The NADase CD38 is induced by factors secreted from senescent cells providing a potential link between senescence and age-related cellular NAD+ decline.

Chini C, Hogan KA, Warner GM, Tarragó MG, Peclat TR, Tchkonia T, Kirkland JL, Chini E.

Biochem Biophys Res Commun. 2019 May 28;513(2):486-493. doi: 10.1016/j.bbrc.2019.03.199. Epub 2019 Apr 8.

2.

A Potent and Specific CD38 Inhibitor Ameliorates Age-Related Metabolic Dysfunction by Reversing Tissue NAD+ Decline.

Tarragó MG, Chini CCS, Kanamori KS, Warner GM, Caride A, de Oliveira GC, Rud M, Samani A, Hein KZ, Huang R, Jurk D, Cho DS, Boslett JJ, Miller JD, Zweier JL, Passos JF, Doles JD, Becherer DJ, Chini EN.

Cell Metab. 2018 May 1;27(5):1081-1095.e10. doi: 10.1016/j.cmet.2018.03.016.

3.

NAD and the aging process: Role in life, death and everything in between.

Chini CCS, Tarragó MG, Chini EN.

Mol Cell Endocrinol. 2017 Nov 5;455:62-74. doi: 10.1016/j.mce.2016.11.003. Epub 2016 Nov 5. Review.

4.

A High Dose of Isoniazid Disturbs Endobiotic Homeostasis in Mouse Liver.

Li F, Wang P, Liu K, Tarrago MG, Lu J, Chini EN, Ma X.

Drug Metab Dispos. 2016 Nov;44(11):1742-1751. Epub 2016 Aug 16.

5.

CD38 Dictates Age-Related NAD Decline and Mitochondrial Dysfunction through an SIRT3-Dependent Mechanism.

Camacho-Pereira J, Tarragó MG, Chini CCS, Nin V, Escande C, Warner GM, Puranik AS, Schoon RA, Reid JM, Galina A, Chini EN.

Cell Metab. 2016 Jun 14;23(6):1127-1139. doi: 10.1016/j.cmet.2016.05.006.

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