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Items: 3

1.

Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome.

Bohnert BN, Daiminger S, Wörn M, Sure F, Staudner T, Ilyaskin AV, Batbouta F, Janessa A, Schneider JC, Essigke D, Kanse S, Haerteis S, Korbmacher C, Artunc F.

Acta Physiol (Oxf). 2019 Dec;227(4):e13286. doi: 10.1111/apha.13286. Epub 2019 May 20.

PMID:
31006168
2.

Bile acids inhibit human purinergic receptor P2X4 in a heterologous expression system.

Ilyaskin AV, Sure F, Nesterov V, Haerteis S, Korbmacher C.

J Gen Physiol. 2019 Jun 3;151(6):820-833. doi: 10.1085/jgp.201812291. Epub 2019 Apr 15.

PMID:
30988062
3.

Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome.

Bohnert BN, Menacher M, Janessa A, Wörn M, Schork A, Daiminger S, Kalbacher H, Häring HU, Daniel C, Amann K, Sure F, Bertog M, Haerteis S, Korbmacher C, Artunc F.

Kidney Int. 2018 Jan;93(1):159-172. doi: 10.1016/j.kint.2017.07.023. Epub 2017 Oct 14.

PMID:
29042083

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