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CXCL10 and trafficking of virus-specific T cells during coronavirus-induced demyelination.

Stiles LN, Liu MT, Kane JA, Lane TE.

Autoimmunity. 2009 Sep;42(6):484-91. doi: 10.1080/08916930902810708.


T-cell function is partially maintained in the absence of class IA phosphoinositide 3-kinase signaling.

Deane JA, Kharas MG, Oak JS, Stiles LN, Luo J, Moore TI, Ji H, Rommel C, Cantley LC, Lane TE, Fruman DA.

Blood. 2007 Apr 1;109(7):2894-902.


T cell antiviral effector function is not dependent on CXCL10 following murine coronavirus infection.

Stiles LN, Hardison JL, Schaumburg CS, Whitman LM, Lane TE.

J Immunol. 2006 Dec 15;177(12):8372-80.


Differential roles for CXCR3 in CD4+ and CD8+ T cell trafficking following viral infection of the CNS.

Stiles LN, Hosking MP, Edwards RA, Strieter RM, Lane TE.

Eur J Immunol. 2006 Mar;36(3):613-22.


CXC chemokine ligand 10 controls viral infection in the central nervous system: evidence for a role in innate immune response through recruitment and activation of natural killer cells.

Trifilo MJ, Montalto-Morrison C, Stiles LN, Hurst KR, Hardison JL, Manning JE, Masters PS, Lane TE.

J Virol. 2004 Jan;78(2):585-94.

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