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Items: 5

1.

A20, a modulator of smooth muscle cell proliferation and apoptosis, prevents and induces regression of neointimal hyperplasia.

Patel VI, Daniel S, Longo CR, Shrikhande GV, Scali ST, Czismadia E, Groft CM, Shukri T, Motley-Dore C, Ramsey HE, Fisher MD, Grey ST, Arvelo MB, Ferran C.

FASEB J. 2006 Jul;20(9):1418-30.

PMID:
16816117
2.

Combined expression of A1 and A20 achieves optimal protection of renal proximal tubular epithelial cells.

Kunter U, Daniel S, Arvelo MB, Choi J, Shukri T, Patel VI, Longo CR, Scali ST, Shrikhande G, Rocha E, Czismadia E, Mottley C, Grey ST, Floege J, Ferran C.

Kidney Int. 2005 Oct;68(4):1520-32.

3.

A20 protects endothelial cells from TNF-, Fas-, and NK-mediated cell death by inhibiting caspase 8 activation.

Daniel S, Arvelo MB, Patel VI, Longo CR, Shrikhande G, Shukri T, Mahiou J, Sun DW, Mottley C, Grey ST, Ferran C.

Blood. 2004 Oct 15;104(8):2376-84. Epub 2004 Jul 13.

PMID:
15251990
4.

Role for activating transcription factor 3 in stress-induced beta-cell apoptosis.

Hartman MG, Lu D, Kim ML, Kociba GJ, Shukri T, Buteau J, Wang X, Frankel WL, Guttridge D, Prentki M, Grey ST, Ron D, Hai T.

Mol Cell Biol. 2004 Jul;24(13):5721-32.

5.

Genetic engineering of a suboptimal islet graft with A20 preserves beta cell mass and function.

Grey ST, Longo C, Shukri T, Patel VI, Csizmadia E, Daniel S, Arvelo MB, Tchipashvili V, Ferran C.

J Immunol. 2003 Jun 15;170(12):6250-6.

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