Format
Sort by
Items per page

Send to

Choose Destination

Search results

Items: 1 to 50 of 65

1.

Retraction.

Berret E, Kintscher M, Palchaudhuri S, Tang W, Osypenko D, Kochubey O, Schneggenburger R.

Science. 2019 Dec 20;366(6472):1460. doi: 10.1126/science.aba2173. No abstract available.

PMID:
31857474
2.

Insular cortex processes aversive somatosensory information and is crucial for threat learning.

Berret E, Kintscher M, Palchaudhuri S, Tang W, Osypenko D, Kochubey O, Schneggenburger R.

Science. 2019 May 31;364(6443). pii: eaaw0474. doi: 10.1126/science.aaw0474. Epub 2019 May 16. Retraction in: Science. 2019 Dec 20;366(6472):1460.

PMID:
31097492
3.

Role of BMP Signaling for the Formation of Auditory Brainstem Nuclei and Large Auditory Relay Synapses.

Kronander E, Clark C, Schneggenburger R.

Dev Neurobiol. 2019 Feb;79(2):155-174. doi: 10.1002/dneu.22661. Epub 2019 Jan 2.

4.

Parvalbumin-Interneuron Output Synapses Show Spike-Timing-Dependent Plasticity that Contributes to Auditory Map Remodeling.

Vickers ED, Clark C, Osypenko D, Fratzl A, Kochubey O, Bettler B, Schneggenburger R.

Neuron. 2018 Aug 22;99(4):720-735.e6. doi: 10.1016/j.neuron.2018.07.018. Epub 2018 Aug 2.

5.

Ultrastructural basis of strong unitary inhibition in a binaural neuron.

Gjoni E, Aguet C, Sahlender DA, Knott G, Schneggenburger R.

J Physiol. 2018 Oct;596(20):4969-4982. doi: 10.1113/JP276015. Epub 2018 Sep 2.

6.

Specific synaptic input strengths determine the computational properties of excitation-inhibition integration in a sound localization circuit.

Gjoni E, Zenke F, Bouhours B, Schneggenburger R.

J Physiol. 2018 Oct;596(20):4945-4967. doi: 10.1113/JP276012. Epub 2018 Aug 28.

7.

An organotypic slice culture to study the formation of calyx of Held synapses in-vitro.

Kronander E, Michalski N, Lebrand C, Hornung JP, Schneggenburger R.

PLoS One. 2017 Apr 18;12(4):e0175964. doi: 10.1371/journal.pone.0175964. eCollection 2017.

8.

Synaptotagmin2 (Syt2) Drives Fast Release Redundantly with Syt1 at the Output Synapses of Parvalbumin-Expressing Inhibitory Neurons.

Bouhours B, Gjoni E, Kochubey O, Schneggenburger R.

J Neurosci. 2017 Apr 26;37(17):4604-4617. doi: 10.1523/JNEUROSCI.3736-16.2017. Epub 2017 Mar 31.

9.

A Synaptotagmin Isoform Switch during the Development of an Identified CNS Synapse.

Kochubey O, Babai N, Schneggenburger R.

Neuron. 2016 Sep 7;91(5):1183. doi: 10.1016/j.neuron.2016.08.024. Epub 2016 Sep 7. No abstract available.

10.

A Synaptotagmin Isoform Switch during the Development of an Identified CNS Synapse.

Kochubey O, Babai N, Schneggenburger R.

Neuron. 2016 Jun 1;90(5):984-99. doi: 10.1016/j.neuron.2016.04.038. Epub 2016 May 19. Erratum in: Neuron. 2016 Sep 7;91(5):1183.

11.

Ca2+ Uncaging in Nerve Terminals: A Three-Point Calibration Procedure.

Kochubey O, Schneggenburger R.

Cold Spring Harb Protoc. 2015 Aug 3;2015(8):761-8. doi: 10.1101/pdb.prot087650.

PMID:
26240411
12.

Molecular mechanisms governing Ca(2+) regulation of evoked and spontaneous release.

Schneggenburger R, Rosenmund C.

Nat Neurosci. 2015 Jul;18(7):935-41. doi: 10.1038/nn.4044. Review.

PMID:
26108721
13.

An Exclusion Zone for Ca2+ Channels around Docked Vesicles Explains Release Control by Multiple Channels at a CNS Synapse.

Keller D, Babai N, Kochubey O, Han Y, Markram H, Schürmann F, Schneggenburger R.

PLoS Comput Biol. 2015 May 7;11(5):e1004253. doi: 10.1371/journal.pcbi.1004253. eCollection 2015 May.

14.

RIM1 and RIM2 redundantly determine Ca2+ channel density and readily releasable pool size at a large hindbrain synapse.

Han Y, Babai N, Kaeser P, Südhof TC, Schneggenburger R.

J Neurophysiol. 2015 Jan 1;113(1):255-63. doi: 10.1152/jn.00488.2014. Epub 2014 Oct 15.

15.

An alien divalent ion reveals a major role for Ca²⁺ buffering in controlling slow transmitter release.

Babai N, Kochubey O, Keller D, Schneggenburger R.

J Neurosci. 2014 Sep 17;34(38):12622-35. doi: 10.1523/JNEUROSCI.1990-14.2014.

16.

Munc18-1 is a dynamically regulated PKC target during short-term enhancement of transmitter release.

Genc O, Kochubey O, Toonen RF, Verhage M, Schneggenburger R.

Elife. 2014 Feb 11;3:e01715. doi: 10.7554/eLife.01715.

17.

BMP signaling specifies the development of a large and fast CNS synapse.

Xiao L, Michalski N, Kronander E, Gjoni E, Genoud C, Knott G, Schneggenburger R.

Nat Neurosci. 2013 Jul;16(7):856-64. doi: 10.1038/nn.3414. Epub 2013 May 26.

PMID:
23708139
18.

Robo3-driven axon midline crossing conditions functional maturation of a large commissural synapse.

Michalski N, Babai N, Renier N, Perkel DJ, Chédotal A, Schneggenburger R.

Neuron. 2013 Jun 5;78(5):855-68. doi: 10.1016/j.neuron.2013.04.006. Epub 2013 May 9.

19.

Ca(2+) channels and transmitter release at the active zone.

Schneggenburger R, Han Y, Kochubey O.

Cell Calcium. 2012 Sep-Oct;52(3-4):199-207. doi: 10.1016/j.ceca.2012.04.011. Epub 2012 Jun 8. Review.

PMID:
22682961
20.

Nigrostriatal overabundance of α-synuclein leads to decreased vesicle density and deficits in dopamine release that correlate with reduced motor activity.

Gaugler MN, Genc O, Bobela W, Mohanna S, Ardah MT, El-Agnaf OM, Cantoni M, Bensadoun JC, Schneggenburger R, Knott GW, Aebischer P, Schneider BL.

Acta Neuropathol. 2012 May;123(5):653-69. doi: 10.1007/s00401-012-0963-y. Epub 2012 Feb 24.

PMID:
22361813
21.

Regulation of transmitter release by Ca(2+) and synaptotagmin: insights from a large CNS synapse.

Kochubey O, Lou X, Schneggenburger R.

Trends Neurosci. 2011 May;34(5):237-46. doi: 10.1016/j.tins.2011.02.006. Epub 2011 Mar 24. Review.

PMID:
21439657
22.

Synaptotagmin increases the dynamic range of synapses by driving Ca²+-evoked release and by clamping a near-linear remaining Ca²+ sensor.

Kochubey O, Schneggenburger R.

Neuron. 2011 Feb 24;69(4):736-48. doi: 10.1016/j.neuron.2011.01.013.

23.

RIM determines Ca²+ channel density and vesicle docking at the presynaptic active zone.

Han Y, Kaeser PS, Südhof TC, Schneggenburger R.

Neuron. 2011 Jan 27;69(2):304-16. doi: 10.1016/j.neuron.2010.12.014.

24.

Control of exocytosis by synaptotagmins and otoferlin in auditory hair cells.

Beurg M, Michalski N, Safieddine S, Bouleau Y, Schneggenburger R, Chapman ER, Petit C, Dulon D.

J Neurosci. 2010 Oct 6;30(40):13281-90. doi: 10.1523/JNEUROSCI.2528-10.2010.

25.

Developmental expression of Synaptotagmin isoforms in single calyx of Held-generating neurons.

Xiao L, Han Y, Runne H, Murray H, Kochubey O, Luthi-Carter R, Schneggenburger R.

Mol Cell Neurosci. 2010 Aug;44(4):374-85. doi: 10.1016/j.mcn.2010.05.002. Epub 2010 May 12.

PMID:
20470891
26.

Interaction between facilitation and depression at a large CNS synapse reveals mechanisms of short-term plasticity.

Müller M, Goutman JD, Kochubey O, Schneggenburger R.

J Neurosci. 2010 Feb 10;30(6):2007-16. doi: 10.1523/JNEUROSCI.4378-09.2010.

27.

Developmental regulation of the intracellular Ca2+ sensitivity of vesicle fusion and Ca2+-secretion coupling at the rat calyx of Held.

Kochubey O, Han Y, Schneggenburger R.

J Physiol. 2009 Jun 15;587(Pt 12):3009-23. doi: 10.1113/jphysiol.2009.172387. Epub 2009 Apr 29.

28.

A limited contribution of Ca2+ current facilitation to paired-pulse facilitation of transmitter release at the rat calyx of Held.

Müller M, Felmy F, Schneggenburger R.

J Physiol. 2008 Nov 15;586(22):5503-20. doi: 10.1113/jphysiol.2008.155838. Epub 2008 Oct 2.

29.

Phorbol esters modulate spontaneous and Ca2+-evoked transmitter release via acting on both Munc13 and protein kinase C.

Lou X, Korogod N, Brose N, Schneggenburger R.

J Neurosci. 2008 Aug 13;28(33):8257-67. doi: 10.1523/JNEUROSCI.0550-08.2008.

30.

Posttetanic potentiation critically depends on an enhanced Ca(2+) sensitivity of vesicle fusion mediated by presynaptic PKC.

Korogod N, Lou X, Schneggenburger R.

Proc Natl Acad Sci U S A. 2007 Oct 2;104(40):15923-8. Epub 2007 Sep 20.

31.
32.
33.

The calyx of Held.

Schneggenburger R, Forsythe ID.

Cell Tissue Res. 2006 Nov;326(2):311-37. Epub 2006 Aug 8. Review.

PMID:
16896951
34.

A Munc13/RIM/Rab3 tripartite complex: from priming to plasticity?

Dulubova I, Lou X, Lu J, Huryeva I, Alam A, Schneggenburger R, Südhof TC, Rizo J.

EMBO J. 2005 Aug 17;24(16):2839-50. Epub 2005 Jul 28.

35.

Interplay between Na+/Ca2+ exchangers and mitochondria in Ca2+ clearance at the calyx of Held.

Kim MH, Korogod N, Schneggenburger R, Ho WK, Lee SH.

J Neurosci. 2005 Jun 29;25(26):6057-65.

36.

Presynaptic calcium and control of vesicle fusion.

Schneggenburger R, Neher E.

Curr Opin Neurobiol. 2005 Jun;15(3):266-74. Review.

PMID:
15919191
37.

Allosteric modulation of the presynaptic Ca2+ sensor for vesicle fusion.

Lou X, Scheuss V, Schneggenburger R.

Nature. 2005 May 26;435(7041):497-501.

PMID:
15917809
38.
39.
40.

The synaptic vesicle protein CSP alpha prevents presynaptic degeneration.

Fernández-Chacón R, Wölfel M, Nishimune H, Tabares L, Schmitz F, Castellano-Muñoz M, Rosenmund C, Montesinos ML, Sanes JR, Schneggenburger R, Südhof TC.

Neuron. 2004 Apr 22;42(2):237-51.

41.

The timing of phasic transmitter release is Ca2+-dependent and lacks a direct influence of presynaptic membrane potential.

Felmy F, Neher E, Schneggenburger R.

Proc Natl Acad Sci U S A. 2003 Dec 9;100(25):15200-5. Epub 2003 Nov 20.

43.

Probing the intracellular calcium sensitivity of transmitter release during synaptic facilitation.

Felmy F, Neher E, Schneggenburger R.

Neuron. 2003 Mar 6;37(5):801-11.

44.

Heterogeneous presynaptic release probabilities: functional relevance for short-term plasticity.

Trommershäuser J, Schneggenburger R, Zippelius A, Neher E.

Biophys J. 2003 Mar;84(3):1563-79.

45.

Estimation of quantal parameters at the calyx of Held synapse.

Sakaba T, Schneggenburger R, Neher E.

Neurosci Res. 2002 Dec;44(4):343-56. Review.

PMID:
12445623
46.

Vesicle pools and short-term synaptic depression: lessons from a large synapse.

Schneggenburger R, Sakaba T, Neher E.

Trends Neurosci. 2002 Apr;25(4):206-12. Review.

PMID:
11998689
48.
49.

Intracellular calcium dependence of transmitter release rates at a fast central synapse.

Schneggenburger R, Neher E.

Nature. 2000 Aug 24;406(6798):889-93.

PMID:
10972290
50.

Supplemental Content

Loading ...
Support Center