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Items: 28


Ptr/CTL0175 Is Required for the Efficient Recovery of Chlamydia trachomatis From Stress Induced by Gamma-Interferon.

Panzetta ME, Luján AL, Bastidas RJ, Damiani MT, Valdivia RH, Saka HA.

Front Microbiol. 2019 Apr 10;10:756. doi: 10.3389/fmicb.2019.00756. eCollection 2019.


Chlamydia Persistence: A Survival Strategy to Evade Antimicrobial Effects in-vitro and in-vivo.

Panzetta ME, Valdivia RH, Saka HA.

Front Microbiol. 2018 Dec 12;9:3101. doi: 10.3389/fmicb.2018.03101. eCollection 2018. Review.


c-Jun Proto-Oncoprotein Plays a Protective Role in Lung Epithelial Cells Exposed to Staphylococcal α-Toxin.

Moyano AJ, Racca AC, Soria G, Saka HA, Andreoli V, Smania AM, Sola C, Bocco JL.

Front Cell Infect Microbiol. 2018 May 25;8:170. doi: 10.3389/fcimb.2018.00170. eCollection 2018.


Erratum: Chlamydia trachomatis neither exerts deleterious effects on spermatozoa nor impairs male fertility.

Puerta Suárez J, Sánchez LR, Salazar FC, Saka HA, Molina R, Tissera A, Rivero VE, Cardona Maya WD, Motrich RD.

Sci Rep. 2017 Nov 21;7(1):16312. doi: 10.1038/s41598-017-12316-4.


Male genital tract immune response against Chlamydia trachomatis infection.

Mackern-Oberti JP, Motrich RD, Damiani MT, Saka HA, Quintero CA, Sánchez LR, Moreno-Sosa T, Olivera C, Cuffini C, Rivero VE.

Reproduction. 2017 Oct;154(4):R99-R110. doi: 10.1530/REP-16-0561. Review.


Chlamydia trachomatis neither exerts deleterious effects on spermatozoa nor impairs male fertility.

Puerta Suarez J, Sanchez LR, Salazar FC, Saka HA, Molina R, Tissera A, Rivero VE, Cardona Maya WD, Motrich RD.

Sci Rep. 2017 Apr 25;7(1):1126. doi: 10.1038/s41598-017-01262-w. Erratum in: Sci Rep. 2017 Nov 21;7(1):16312.


Chlamydia trachomatis Infection Leads to Defined Alterations to the Lipid Droplet Proteome in Epithelial Cells.

Saka HA, Thompson JW, Chen YS, Dubois LG, Haas JT, Moseley A, Valdivia RH.

PLoS One. 2015 Apr 24;10(4):e0124630. doi: 10.1371/journal.pone.0124630. eCollection 2015.


New patterns of methicillin-resistant Staphylococcus aureus (MRSA) clones, community-associated MRSA genotypes behave like healthcare-associated MRSA genotypes within hospitals, Argentina.

Egea AL, Gagetti P, Lamberghini R, Faccone D, Lucero C, Vindel A, Tosoroni D, Garnero A, Saka HA, Galas M; S. aureus Study Group-Argentina, Bocco JL, Corso A, Sola C.

Int J Med Microbiol. 2014 Nov;304(8):1086-99. doi: 10.1016/j.ijmm.2014.08.002. Epub 2014 Aug 12.


Search for microRNAs expressed by intracellular bacterial pathogens in infected mammalian cells.

Furuse Y, Finethy R, Saka HA, Xet-Mull AM, Sisk DM, Smith KL, Lee S, Coers J, Valdivia RH, Tobin DM, Cullen BR.

PLoS One. 2014 Sep 3;9(9):e106434. doi: 10.1371/journal.pone.0106434. eCollection 2014.


Reassessing the role of the secreted protease CPAF in Chlamydia trachomatis infection through genetic approaches.

Snavely EA, Kokes M, Dunn JD, Saka HA, Nguyen BD, Bastidas RJ, McCafferty DG, Valdivia RH.

Pathog Dis. 2014 Aug;71(3):336-51. doi: 10.1111/2049-632X.12179. Epub 2014 May 16.


The Chlamydia trachomatis type III secretion chaperone Slc1 engages multiple early effectors, including TepP, a tyrosine-phosphorylated protein required for the recruitment of CrkI-II to nascent inclusions and innate immune signaling.

Chen YS, Bastidas RJ, Saka HA, Carpenter VK, Richards KL, Plano GV, Valdivia RH.

PLoS Pathog. 2014 Feb 20;10(2):e1003954. doi: 10.1371/journal.ppat.1003954. eCollection 2014 Feb. Erratum in: PLoS Pathog. 2014 Mar;10(3):e1004094.


IRG and GBP host resistance factors target aberrant, "non-self" vacuoles characterized by the missing of "self" IRGM proteins.

Haldar AK, Saka HA, Piro AS, Dunn JD, Henry SC, Taylor GA, Frickel EM, Valdivia RH, Coers J.

PLoS Pathog. 2013;9(6):e1003414. doi: 10.1371/journal.ppat.1003414. Epub 2013 Jun 13.


Emerging roles for lipid droplets in immunity and host-pathogen interactions.

Saka HA, Valdivia R.

Annu Rev Cell Dev Biol. 2012;28:411-37. doi: 10.1146/annurev-cellbio-092910-153958. Epub 2012 May 11. Review.


Quantitative proteomics reveals metabolic and pathogenic properties of Chlamydia trachomatis developmental forms.

Saka HA, Thompson JW, Chen YS, Kumar Y, Dubois LG, Moseley MA, Valdivia RH.

Mol Microbiol. 2011 Dec;82(5):1185-203. doi: 10.1111/j.1365-2958.2011.07877.x. Epub 2011 Nov 7.


On multi-strain model for Hepatitis C.

Ahmed E, El-Saka H.

Nonlinear Biomed Phys. 2011 Aug 3;5:6. doi: 10.1186/1753-4631-5-6.


On modeling two immune effectors two strain antigen interaction.

Ahmed el-SM, El-Saka HA.

Nonlinear Biomed Phys. 2010 Nov 25;4:6. doi: 10.1186/1753-4631-4-6.


On fractional order models for Hepatitis C.

Ahmed E, El-Saka HA.

Nonlinear Biomed Phys. 2010 Mar 18;4(1):1. doi: 10.1186/1753-4631-4-1.


Acquisition of nutrients by Chlamydiae: unique challenges of living in an intracellular compartment.

Saka HA, Valdivia RH.

Curr Opin Microbiol. 2010 Feb;13(1):4-10. doi: 10.1016/j.mib.2009.11.002. Epub 2009 Dec 16. Review.


Vibrio cholerae cytolysin is essential for high enterotoxicity and apoptosis induction produced by a cholera toxin gene-negative V. cholerae non-O1, non-O139 strain.

Saka HA, Bidinost C, Sola C, Carranza P, Collino C, Ortiz S, Echenique JR, Bocco JL.

Microb Pathog. 2008 Feb;44(2):118-28. Epub 2007 Aug 31.


The autophagic pathway: a cell survival strategy against the bacterial pore-forming toxin Vibrio cholerae cytolysin.

Saka HA, Gutiérrez MG, Bocco JL, Colombo MI.

Autophagy. 2007 Jul-Aug;3(4):363-5. Epub 2007 Jul 19.


High frequency of Panton-Valentine leukocidin genes in invasive methicillin-susceptible Staphylococcus aureus strains and the relationship with methicillin-resistant Staphylococcus aureus in Córdoba, Argentina.

Sola C, Saka HA, Vindel A, Bocco JL; Córdoba S. aureus Collaborative Study Group.

Eur J Clin Microbiol Infect Dis. 2007 Apr;26(4):281-6.


Protective role of autophagy against Vibrio cholerae cytolysin, a pore-forming toxin from V. cholerae.

Gutierrez MG, Saka HA, Chinen I, Zoppino FC, Yoshimori T, Bocco JL, Colombo MI.

Proc Natl Acad Sci U S A. 2007 Feb 6;104(6):1829-34. Epub 2007 Jan 31.


Virulence factors of non-O1 non-O139 Vibrio cholerae isolated in Córdoba, Argentina.

Bidinost C, Saka HA, Aliendro O, Sola C, Panzetta-Duttari G, Carranza P, Echenique J, Patrito E, Bocco JL.

Rev Argent Microbiol. 2004 Oct-Dec;36(4):158-63.


CARB-9, a carbenicillinase encoded in the VCR region of Vibrio cholerae non-O1, non-O139 belongs to a family of cassette-encoded beta-lactamases.

Petroni A, Melano RG, Saka HA, Garutti A, Mange L, Pasterán F, Rapoport M, Miranda M, Faccone D, Rossi A, Hoffman PS, Galas MF.

Antimicrob Agents Chemother. 2004 Oct;48(10):4042-6.


[Extended-spectrum beta-lactamases in Klebsiella pneumoniae isolated at the Cordoba Children's Hospital, Argentina].

Saka HA, Egea M, Culasso C, Rollán R, Avaro A, Carvajal L.

Rev Argent Microbiol. 2003 Jan-Mar;35(1):1-7. Spanish.


New carbenicillin-hydrolyzing beta-lactamase (CARB-7) from Vibrio cholerae non-O1, non-O139 strains encoded by the VCR region of the V. cholerae genome.

Melano R, Petroni A, Garutti A, Saka HA, Mange L, Pasterán F, Rapoport M, Rossi A, Galas M.

Antimicrob Agents Chemother. 2002 Jul;46(7):2162-8.

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