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Items: 7


Keratin 18 overexpression but not phosphorylation or filament organization blocks mouse Mallory body formation.

Harada M, Strnad P, Resurreccion EZ, Ku NO, Omary MB.

Hepatology. 2007 Jan;45(1):88-96.


Keratin 8 and 18 hyperphosphorylation is a marker of progression of human liver disease.

Toivola DM, Ku NO, Resurreccion EZ, Nelson DR, Wright TL, Omary MB.

Hepatology. 2004 Aug;40(2):459-66.


Organ-specific stress induces mouse pancreatic keratin overexpression in association with NF-kappaB activation.

Zhong B, Zhou Q, Toivola DM, Tao GZ, Resurreccion EZ, Omary MB.

J Cell Sci. 2004 Apr 1;117(Pt 9):1709-19.


Keratin-8 null mice have different gallbladder and liver susceptibility to lithogenic diet-induced injury.

Tao GZ, Toivola DM, Zhong B, Michie SA, Resurreccion EZ, Tamai Y, Taketo MM, Omary MB.

J Cell Sci. 2003 Nov 15;116(Pt 22):4629-38.


Keratin binding to 14-3-3 proteins modulates keratin filaments and hepatocyte mitotic progression.

Ku NO, Michie S, Resurreccion EZ, Broome RL, Omary MB.

Proc Natl Acad Sci U S A. 2002 Apr 2;99(7):4373-8. Epub 2002 Mar 26.


Mutation of a major keratin phosphorylation site predisposes to hepatotoxic injury in transgenic mice.

Ku NO, Michie SA, Soetikno RM, Resurreccion EZ, Broome RL, Omary MB.

J Cell Biol. 1998 Dec 28;143(7):2023-32.


Susceptibility to hepatotoxicity in transgenic mice that express a dominant-negative human keratin 18 mutant.

Ku NO, Michie SA, Soetikno RM, Resurreccion EZ, Broome RL, Oshima RG, Omary MB.

J Clin Invest. 1996 Aug 15;98(4):1034-46.

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