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Items: 21

1.

Drosophila studies support a role for a presynaptic synaptotagmin mutation in a human congenital myasthenic syndrome.

Shields MC, Bowers MR, Fulcer MM, Bollig MK, Rock PJ, Sutton BR, Vrailas-Mortimer AD, Lochm├╝ller H, Whittaker RG, Horvath R, Reist NE.

PLoS One. 2017 Sep 27;12(9):e0184817. doi: 10.1371/journal.pone.0184817. eCollection 2017.

2.

Epsin 1 Promotes Synaptic Growth by Enhancing BMP Signal Levels in Motoneuron Nuclei.

Vanlandingham PA, Fore TR, Chastain LR, Royer SM, Bao H, Reist NE, Zhang B.

PLoS One. 2013 Jun 19;8(6):e65997. doi: 10.1371/journal.pone.0065997. Print 2013.

3.

Calcium binding by synaptotagmin's C2A domain is an essential element of the electrostatic switch that triggers synchronous synaptic transmission.

Striegel AR, Biela LM, Evans CS, Wang Z, Delehoy JB, Sutton RB, Chapman ER, Reist NE.

J Neurosci. 2012 Jan 25;32(4):1253-60. doi: 10.1523/JNEUROSCI.4652-11.2012.

4.

Membrane penetration by synaptotagmin is required for coupling calcium binding to vesicle fusion in vivo.

Paddock BE, Wang Z, Biela LM, Chen K, Getzy MD, Striegel A, Richmond JE, Chapman ER, Featherstone DE, Reist NE.

J Neurosci. 2011 Feb 9;31(6):2248-57. doi: 10.1523/JNEUROSCI.3153-09.2011.

5.

Synaptotagmin I stabilizes synaptic vesicles via its C(2)A polylysine motif.

Mace KE, Biela LM, Sares AG, Reist NE.

Genesis. 2009 May;47(5):337-45. doi: 10.1002/dvg.20502.

PMID:
19358157
6.

The Drosophila epsin 1 is required for ubiquitin-dependent synaptic growth and function but not for synaptic vesicle recycling.

Bao H, Reist NE, Zhang B.

Traffic. 2008 Dec;9(12):2190-205. doi: 10.1111/j.1600-0854.2008.00832.x. Epub 2008 Sep 15.

7.

Ca2+-dependent, phospholipid-binding residues of synaptotagmin are critical for excitation-secretion coupling in vivo.

Paddock BE, Striegel AR, Hui E, Chapman ER, Reist NE.

J Neurosci. 2008 Jul 23;28(30):7458-66. doi: 10.1523/JNEUROSCI.0197-08.2008.

8.
9.

C2B polylysine motif of synaptotagmin facilitates a Ca2+-independent stage of synaptic vesicle priming in vivo.

Loewen CA, Lee SM, Shin YK, Reist NE.

Mol Biol Cell. 2006 Dec;17(12):5211-26. Epub 2006 Sep 20.

10.
11.

The C(2)B Ca(2+)-binding motif of synaptotagmin is required for synaptic transmission in vivo.

Mackler JM, Drummond JA, Loewen CA, Robinson IM, Reist NE.

Nature. 2002 Jul 18;418(6895):340-4. Epub 2002 Jul 7.

PMID:
12110842
12.

Drosophila synaptotagmin I null mutants survive to early adulthood.

Loewen CA, Mackler JM, Reist NE.

Genesis. 2001 Sep;31(1):30-6.

PMID:
11668675
13.
14.

Morphologically docked synaptic vesicles are reduced in synaptotagmin mutants of Drosophila.

Reist NE, Buchanan J, Li J, DiAntonio A, Buxton EM, Schwarz TL.

J Neurosci. 1998 Oct 1;18(19):7662-73.

15.

Neurally evoked calcium transients in terminal Schwann cells at the neuromuscular junction.

Reist NE, Smith SJ.

Proc Natl Acad Sci U S A. 1992 Aug 15;89(16):7625-9.

16.
17.

Agrin-like molecules at synaptic sites in normal, denervated, and damaged skeletal muscles.

Reist NE, Magill C, McMahan UJ.

J Cell Biol. 1987 Dec;105(6 Pt 1):2457-69.

18.

Identification of agrin in electric organ extracts and localization of agrin-like molecules in muscle and central nervous system.

Smith MA, Yao YM, Reist NE, Magill C, Wallace BG, McMahan UJ.

J Exp Biol. 1987 Sep;132:223-30.

19.

Agrin.

Magill C, Reist NE, Fallon JR, Nitkin RM, Wallace BG, McMahan UJ.

Prog Brain Res. 1987;71:391-6. No abstract available.

PMID:
3035610
20.

Aggregates of acetylcholinesterase induced by acetylcholine receptor-aggregating factor.

Wallace BG, Nitkin RM, Reist NE, Fallon JR, Moayeri NN, McMahan UJ.

Nature. 1985 Jun 13-19;315(6020):574-7.

PMID:
4010772
21.

Acetylcholine receptor-aggregating factor is similar to molecules concentrated at neuromuscular junctions.

Fallon JR, Nitkin RM, Reist NE, Wallace BG, McMahan UJ.

Nature. 1985 Jun 13-19;315(6020):571-4.

PMID:
3892302

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