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Items: 14

1.

Transmission of amyloid-β protein pathology from cadaveric pituitary growth hormone.

Purro SA, Farrow MA, Linehan J, Nazari T, Thomas DX, Chen Z, Mengel D, Saito T, Saido T, Rudge P, Brandner S, Walsh DM, Collinge J.

Nature. 2018 Dec;564(7736):415-419. doi: 10.1038/s41586-018-0790-y. Epub 2018 Dec 13.

2.

Reply to: Intrinsic Toxicity of Antibodies to the Globular Domain of the Prion Protein.

Purro SA, Mead S, Khalili-Shirazi A, Nicoll AJ, Collinge J.

Biol Psychiatry. 2018 Oct 1;84(7):e53-e54. doi: 10.1016/j.biopsych.2018.04.002. Epub 2018 Apr 12. No abstract available.

PMID:
29752071
3.

Prion Protein as a Toxic Acceptor of Amyloid-β Oligomers.

Purro SA, Nicoll AJ, Collinge J.

Biol Psychiatry. 2018 Feb 15;83(4):358-368. doi: 10.1016/j.biopsych.2017.11.020. Epub 2017 Nov 21. Review.

PMID:
29331212
4.

Post-Translational Incorporation of L-Phenylalanine into the C-Terminus of α-Tubulin as a Possible Cause of Neuronal Dysfunction.

Ditamo Y, Dentesano YM, Purro SA, Arce CA, Bisig CG.

Sci Rep. 2016 Dec 1;6:38140. doi: 10.1038/srep38140.

5.

mGlu5 receptors and cellular prion protein mediate amyloid-β-facilitated synaptic long-term depression in vivo.

Hu NW, Nicoll AJ, Zhang D, Mably AJ, O'Malley T, Purro SA, Terry C, Collinge J, Walsh DM, Rowan MJ.

Nat Commun. 2014 Mar 4;5:3374. doi: 10.1038/ncomms4374.

6.

Dysfunction of Wnt signaling and synaptic disassembly in neurodegenerative diseases.

Purro SA, Galli S, Salinas PC.

J Mol Cell Biol. 2014 Feb;6(1):75-80. doi: 10.1093/jmcb/mjt049. Epub 2014 Jan 20. Review.

7.

The secreted Wnt antagonist Dickkopf-1 is required for amyloid β-mediated synaptic loss.

Purro SA, Dickins EM, Salinas PC.

J Neurosci. 2012 Mar 7;32(10):3492-8. doi: 10.1523/JNEUROSCI.4562-11.2012.

8.

Phytoremediation of 2,4-dichlorophenol using wild type and transgenic tobacco plants.

Talano MA, Busso DC, Paisio CE, González PS, Purro SA, Medina MI, Agostini E.

Environ Sci Pollut Res Int. 2012 Jul;19(6):2202-11. doi: 10.1007/s11356-011-0724-9. Epub 2012 Jan 11.

PMID:
22234851
9.

Lack of stabilized microtubules as a result of the absence of major maps in CAD cells does not preclude neurite formation.

Bisig CG, Chesta ME, Zampar GG, Purro SA, Santander VS, Arce CA.

FEBS J. 2009 Dec;276(23):7110-23. doi: 10.1111/j.1742-4658.2009.07422.x. Epub 2009 Oct 29.

10.

Wnt regulates axon behavior through changes in microtubule growth directionality: a new role for adenomatous polyposis coli.

Purro SA, Ciani L, Hoyos-Flight M, Stamatakou E, Siomou E, Salinas PC.

J Neurosci. 2008 Aug 20;28(34):8644-54. doi: 10.1523/JNEUROSCI.2320-08.2008.

11.

Tubulin must be acetylated in order to form a complex with membrane Na(+),K (+)-ATPase and to inhibit its enzyme activity.

Santander VS, Bisig CG, Purro SA, Casale CH, Arce CA, Barra HS.

Mol Cell Biochem. 2006 Oct;291(1-2):167-74. Epub 2006 May 30.

PMID:
16733802
12.

Inhibitors of protein phosphatase 1 and 2A decrease the level of tubulin carboxypeptidase activity associated with microtubules.

Contín MA, Purro SA, Bisig CG, Barra HS, Arce CA.

Eur J Biochem. 2003 Dec;270(24):4921-9.

13.

Post-translational incorporation of the antiproliferative agent azatyrosine into the C-terminus of alpha-tubulin.

Purro SA, Bisig CG, Contin MA, Barra HS, Arce CA.

Biochem J. 2003 Oct 1;375(Pt 1):121-9.

14.

Incorporation of 3-nitrotyrosine into the C-terminus of alpha-tubulin is reversible and not detrimental to dividing cells.

Bisig CG, Purro SA, Contín MA, Barra HS, Arce CA.

Eur J Biochem. 2002 Oct;269(20):5037-45.

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