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Items: 7

1.

Ligand-mediated cytoplasmic retention of the Ah receptor inhibits macrophage-mediated acute inflammatory responses.

Muku GE, Lahoti TS, Murray IA, Podolsky MA, Smith KJ, Hubbard TD, Kuzu G, Gowda K, Amin SG, Perdew GH.

Lab Invest. 2017 Dec;97(12):1471-1487. doi: 10.1038/labinvest.2017.92. Epub 2017 Sep 11.

2.

ER stress and distinct outputs of the IRE1α RNase control proliferation and senescence in response to oncogenic Ras.

Blazanin N, Son J, Craig-Lucas AB, John CL, Breech KJ, Podolsky MA, Glick AB.

Proc Natl Acad Sci U S A. 2017 Sep 12;114(37):9900-9905. doi: 10.1073/pnas.1701757114. Epub 2017 Aug 28.

3.

Differentiated State of Initiating Tumor Cells Is Key to Distinctive Immune Responses Seen in H-RasG12V-Induced Squamous Tumors.

Podolsky MA, Bailey JT, Gunderson AJ, Oakes CJ, Breech K, Glick AB.

Cancer Immunol Res. 2017 Mar;5(3):198-210. doi: 10.1158/2326-6066.CIR-16-0304. Epub 2017 Jan 30.

4.

Genetic and pharmacological analysis identifies a physiological role for the AHR in epidermal differentiation.

van den Bogaard EH, Podolsky MA, Smits JP, Cui X, John C, Gowda K, Desai D, Amin SG, Schalkwijk J, Perdew GH, Glick AB.

J Invest Dermatol. 2015 May;135(5):1320-1328. doi: 10.1038/jid.2015.6. Epub 2015 Jan 20.

5.

The TGFβ1 pathway is required for NFκB dependent gene expression in mouse keratinocytes.

Hogan KA, Ravindran A, Podolsky MA, Glick AB.

Cytokine. 2013 Dec;64(3):652-9. doi: 10.1016/j.cyto.2013.09.004. Epub 2013 Sep 24.

6.

CD8(+) T cells mediate RAS-induced psoriasis-like skin inflammation through IFN-γ.

Gunderson AJ, Mohammed J, Horvath FJ, Podolsky MA, Anderson CR, Glick AB.

J Invest Dermatol. 2013 Apr;133(4):955-63. doi: 10.1038/jid.2012.390. Epub 2012 Nov 15.

7.

Extended JAK activation and delayed STAT1 dephosphorylation contribute to the distinct signaling profile of CNS neurons exposed to interferon-gamma.

Podolsky MA, Solomos AC, Durso LC, Evans SM, Rall GF, Rose RW.

J Neuroimmunol. 2012 Oct 15;251(1-2):33-8. doi: 10.1016/j.jneuroim.2012.06.006. Epub 2012 Jul 4.

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