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Items: 21


Virus transcript levels and cell growth rates after naturally occurring HPV16 integration events in basal cervical keratinocytes.

Scarpini CG, Groves IJ, Pett MR, Ward D, Coleman N.

J Pathol. 2014 Jul;233(3):281-93. doi: 10.1002/path.4358. Epub 2014 May 21.


Depletion of HPV16 early genes induces autophagy and senescence in a cervical carcinogenesis model, regardless of viral physical state.

Hanning JE, Saini HK, Murray MJ, Caffarel MM, van Dongen S, Ward D, Barker EM, Scarpini CG, Groves IJ, Stanley MA, Enright AJ, Pett MR, Coleman N.

J Pathol. 2013 Nov;231(3):354-66. doi: 10.1002/path.4244.


LIN28 Expression in malignant germ cell tumors downregulates let-7 and increases oncogene levels.

Murray MJ, Saini HK, Siegler CA, Hanning JE, Barker EM, van Dongen S, Ward DM, Raby KL, Groves IJ, Scarpini CG, Pett MR, Thornton CM, Enright AJ, Nicholson JC, Coleman N; CCLG.

Cancer Res. 2013 Aug 1;73(15):4872-84. doi: 10.1158/0008-5472.CAN-12-2085. Epub 2013 Jun 17.


Lack of correlation between predicted and actual off-target effects of short-interfering RNAs targeting the human papillomavirus type 16 E7 oncogene.

Hanning JE, Saini HK, Murray MJ, van Dongen S, Davis MP, Barker EM, Ward DM, Scarpini CG, Enright AJ, Pett MR, Coleman N.

Br J Cancer. 2013 Feb 5;108(2):450-60. doi: 10.1038/bjc.2012.564. Epub 2013 Jan 8.


Lipoprotein lipase is frequently overexpressed or translocated in cervical squamous cell carcinoma and promotes invasiveness through the non-catalytic C terminus.

Carter SA, Foster NA, Scarpini CG, Chattopadhyay A, Pett MR, Roberts I, Coleman N.

Br J Cancer. 2012 Aug 7;107(4):739-47. doi: 10.1038/bjc.2012.301. Epub 2012 Jul 10.


Overexpression of the oncostatin M receptor in cervical squamous cell carcinoma cells is associated with a pro-angiogenic phenotype and increased cell motility and invasiveness.

Winder DM, Chattopadhyay A, Muralidhar B, Bauer J, English WR, Zhang X, Karagavriilidou K, Roberts I, Pett MR, Murphy G, Coleman N.

J Pathol. 2011 Nov;225(3):448-62. doi: 10.1002/path.2968. Epub 2011 Sep 26.


Development of peptide aptamer microarrays for detection of HPV16 oncoproteins in cell extracts.

Laurenson S, Pett MR, Hoppe-Seyler K, Denk C, Hoppe-Seyler F, Coleman N, Ko Ferrigno P.

Anal Biochem. 2011 Mar 15;410(2):161-70. doi: 10.1016/j.ab.2010.10.038. Epub 2010 Nov 6.


The two most common histological subtypes of malignant germ cell tumour are distinguished by global microRNA profiles, associated with differential transcription factor expression.

Murray MJ, Saini HK, van Dongen S, Palmer RD, Muralidhar B, Pett MR, Piipari M, Thornton CM, Nicholson JC, Enright AJ, Coleman N.

Mol Cancer. 2010 Nov 8;9:290. doi: 10.1186/1476-4598-9-290.


In vitro progression of human papillomavirus 16 episome-associated cervical neoplasia displays fundamental similarities to integrant-associated carcinogenesis.

Gray E, Pett MR, Ward D, Winder DM, Stanley MA, Roberts I, Scarpini CG, Coleman N.

Cancer Res. 2010 May 15;70(10):4081-91. doi: 10.1158/0008-5472.CAN-09-3335. Epub 2010 May 4.


Malignant germ cell tumors display common microRNA profiles resulting in global changes in expression of messenger RNA targets.

Palmer RD, Murray MJ, Saini HK, van Dongen S, Abreu-Goodger C, Muralidhar B, Pett MR, Thornton CM, Nicholson JC, Enright AJ, Coleman N; Children's Cancer and Leukaemia Group.

Cancer Res. 2010 Apr 1;70(7):2911-23. doi: 10.1158/0008-5472.CAN-09-3301. Epub 2010 Mar 23.


Characterization of naturally occurring HPV16 integration sites isolated from cervical keratinocytes under noncompetitive conditions.

Dall KL, Scarpini CG, Roberts I, Winder DM, Stanley MA, Muralidhar B, Herdman MT, Pett MR, Coleman N.

Cancer Res. 2008 Oct 15;68(20):8249-59. doi: 10.1158/0008-5472.CAN-08-1741.


Critical evaluation of HPV16 gene copy number quantification by SYBR green PCR.

Roberts I, Ng G, Foster N, Stanley M, Herdman MT, Pett MR, Teschendorff A, Coleman N.

BMC Biotechnol. 2008 Jul 24;8:57. doi: 10.1186/1472-6750-8-57.


Pediatric malignant germ cell tumors show characteristic transcriptome profiles.

Palmer RD, Barbosa-Morais NL, Gooding EL, Muralidhar B, Thornton CM, Pett MR, Roberts I, Schneider DT, Thorne N, Tavaré S, Nicholson JC, Coleman N; Children's Cancer and Leukaemia Group.

Cancer Res. 2008 Jun 1;68(11):4239-47. doi: 10.1158/0008-5472.CAN-07-5560.


HPV: from infection to cancer.

Stanley MA, Pett MR, Coleman N.

Biochem Soc Trans. 2007 Dec;35(Pt 6):1456-60. Review.


An increase in DNA double-strand breaks, induced by Ku70 depletion, is associated with human papillomavirus 16 episome loss and de novo viral integration events.

Winder DM, Pett MR, Foster N, Shivji MK, Herdman MT, Stanley MA, Venkitaraman AR, Coleman N.

J Pathol. 2007 Sep;213(1):27-34.


Global microRNA profiles in cervical squamous cell carcinoma depend on Drosha expression levels.

Muralidhar B, Goldstein LD, Ng G, Winder DM, Palmer RD, Gooding EL, Barbosa-Morais NL, Mukherjee G, Thorne NP, Roberts I, Pett MR, Coleman N.

J Pathol. 2007 Aug;212(4):368-77.


Interferon-beta treatment of cervical keratinocytes naturally infected with human papillomavirus 16 episomes promotes rapid reduction in episome numbers and emergence of latent integrants.

Herdman MT, Pett MR, Roberts I, Alazawi WO, Teschendorff AE, Zhang XY, Stanley MA, Coleman N.

Carcinogenesis. 2006 Nov;27(11):2341-53. Epub 2006 Sep 14.


Selection of cervical keratinocytes containing integrated HPV16 associates with episome loss and an endogenous antiviral response.

Pett MR, Herdman MT, Palmer RD, Yeo GS, Shivji MK, Stanley MA, Coleman N.

Proc Natl Acad Sci U S A. 2006 Mar 7;103(10):3822-7. Epub 2006 Feb 27.


Acquisition of high-level chromosomal instability is associated with integration of human papillomavirus type 16 in cervical keratinocytes.

Pett MR, Alazawi WO, Roberts I, Dowen S, Smith DI, Stanley MA, Coleman N.

Cancer Res. 2004 Feb 15;64(4):1359-68.


Amplification of chromosome 5p correlates with increased expression of Skp2 in HPV-immortalized keratinocytes.

Dowen SE, Neutze DM, Pett MR, Cottage A, Stern P, Coleman N, Stanley MA.

Oncogene. 2003 Apr 24;22(16):2531-40.


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