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Items: 17


PARP2 controls double-strand break repair pathway choice by limiting 53BP1 accumulation at DNA damage sites and promoting end-resection.

Fouquin A, Guirouilh-Barbat J, Lopez B, Hall J, Amor-Guéret M, Pennaneach V.

Nucleic Acids Res. 2017 Dec 1;45(21):12325-12339. doi: 10.1093/nar/gkx881.


Cdk5 promotes DNA replication stress checkpoint activation through RPA-32 phosphorylation, and impacts on metastasis free survival in breast cancer patients.

Chiker S, Pennaneach V, Loew D, Dingli F, Biard D, Cordelières FP, Gemble S, Vacher S, Bieche I, Hall J, Fernet M.

Cell Cycle. 2015;14(19):3066-78. doi: 10.1080/15384101.2015.1078020.


[PARP inhibitors and radiotherapy: rational and prospects for a clinical use].

Pernin V, Mégnin-Chanet F, Pennaneach V, Fourquet A, Kirova Y, Hall J.

Cancer Radiother. 2014 Dec;18(8):790-8; quiz 799-802. doi: 10.1016/j.canrad.2014.05.012. Epub 2014 Oct 17. Review. French.


PARP-2 depletion results in lower radiation cell survival but cell line-specific differences in poly(ADP-ribose) levels.

Boudra MT, Bolin C, Chiker S, Fouquin A, Zaremba T, Vaslin L, Biard D, Cordelières FP, Mégnin-Chanet F, Favaudon V, Fernet M, Pennaneach V, Hall J.

Cell Mol Life Sci. 2015 Apr;72(8):1585-97. doi: 10.1007/s00018-014-1765-2. Epub 2014 Oct 22.


Variations in the mRNA expression of poly(ADP-ribose) polymerases, poly(ADP-ribose) glycohydrolase and ADP-ribosylhydrolase 3 in breast tumors and impact on clinical outcome.

Bieche I, Pennaneach V, Driouch K, Vacher S, Zaremba T, Susini A, Lidereau R, Hall J.

Int J Cancer. 2013 Dec 15;133(12):2791-800. doi: 10.1002/ijc.28304. Epub 2013 Aug 9.


Partial complementation of a DNA ligase I deficiency by DNA ligase III and its impact on cell survival and telomere stability in mammalian cells.

Le Chalony C, Hoffschir F, Gauthier LR, Gross J, Biard DS, Boussin FD, Pennaneach V.

Cell Mol Life Sci. 2012 Sep;69(17):2933-49. doi: 10.1007/s00018-012-0975-8. Epub 2012 Mar 30.


The impact of cyclin-dependent kinase 5 depletion on poly(ADP-ribose) polymerase activity and responses to radiation.

Bolin C, Boudra MT, Fernet M, Vaslin L, Pennaneach V, Zaremba T, Biard D, Cordelières FP, Favaudon V, Mégnin-Chanet F, Hall J.

Cell Mol Life Sci. 2012 Mar;69(6):951-62. doi: 10.1007/s00018-011-0811-6. Epub 2011 Sep 16.


Stabilization of dicentric translocations through secondary rearrangements mediated by multiple mechanisms in S. cerevisiae.

Pennaneach V, Kolodner RD.

PLoS One. 2009 Jul 28;4(7):e6389. doi: 10.1371/journal.pone.0006389.


Analysis of gross-chromosomal rearrangements in Saccharomyces cerevisiae.

Schmidt KH, Pennaneach V, Putnam CD, Kolodner RD.

Methods Enzymol. 2006;409:462-76.


Chromosome healing by de novo telomere addition in Saccharomyces cerevisiae.

Pennaneach V, Putnam CD, Kolodner RD.

Mol Microbiol. 2006 Mar;59(5):1357-68. Review.


Saccharomyces cerevisiae as a model system to define the chromosomal instability phenotype.

Putnam CD, Pennaneach V, Kolodner RD.

Mol Cell Biol. 2005 Aug;25(16):7226-38.


Chromosome healing through terminal deletions generated by de novo telomere additions in Saccharomyces cerevisiae.

Putnam CD, Pennaneach V, Kolodner RD.

Proc Natl Acad Sci U S A. 2004 Sep 7;101(36):13262-7. Epub 2004 Aug 24.


Rb inhibits E2F-1-induced cell death in a LXCXE-dependent manner by active repression.

Pennaneach V, Barbier V, Regazzoni K, Fotedar R, Fotedar A.

J Biol Chem. 2004 May 28;279(22):23376-83. Epub 2004 Mar 11.


Phosphorylation of the PCNA binding domain of the large subunit of replication factor C on Thr506 by cyclin-dependent kinases regulates binding to PCNA.

Salles-Passador I, Munshi A, Cannella D, Pennaneach V, Koundrioukoff S, Jaquinod M, Forest E, Podust V, Fotedar A, Fotedar R.

Nucleic Acids Res. 2003 Sep 1;31(17):5202-11. Erratum in: Nucleic Acids Res. 2003 Oct 15;31(20):6062. Jacquinod, M [corrected to Jaquinod, M].


Saccharomyces cerevisiae chromatin-assembly factors that act during DNA replication function in the maintenance of genome stability.

Myung K, Pennaneach V, Kats ES, Kolodner RD.

Proc Natl Acad Sci U S A. 2003 May 27;100(11):6640-5. Epub 2003 May 15.


The large subunit of replication factor C promotes cell survival after DNA damage in an LxCxE motif- and Rb-dependent manner.

Pennaneach V, Salles-Passador I, Munshi A, Brickner H, Regazzoni K, Dick F, Dyson N, Chen TT, Wang JY, Fotedar R, Fotedar A.

Mol Cell. 2001 Apr;7(4):715-27.

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