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Items: 16


Immune- and Nonimmune-Compartment-Specific Interferon Responses Are Critical Determinants of Herpes Simplex Virus-Induced Generalized Infections and Acute Liver Failure.

Parker ZM, Pasieka TJ, Parker GA, Leib DA.

J Virol. 2016 Nov 14;90(23):10789-10799. doi: 10.1128/JVI.01473-16. Print 2016 Dec 1.


Immortalized human cerebral microvascular endothelial cells maintain the properties of primary cells in an in vitro model of immune migration across the blood brain barrier.

Daniels BP, Cruz-Orengo L, Pasieka TJ, Couraud PO, Romero IA, Weksler B, Cooper JA, Doering TL, Klein RS.

J Neurosci Methods. 2013 Jan 15;212(1):173-9. doi: 10.1016/j.jneumeth.2012.10.001. Epub 2012 Oct 13.


Corneal replication is an interferon response-independent bottleneck for virulence of herpes simplex virus 1 in the absence of virion host shutoff.

Pasieka TJ, Menachery VD, Rosato PC, Leib DA.

J Virol. 2012 Jul;86(14):7692-5. doi: 10.1128/JVI.00761-12. Epub 2012 May 2.


Functional genomics reveals an essential and specific role for Stat1 in protection of the central nervous system following herpes simplex virus corneal infection.

Pasieka TJ, Cilloniz C, Carter VS, Rosato P, Katze MG, Leib DA.

J Virol. 2011 Dec;85(24):12972-81. doi: 10.1128/JVI.06032-11. Epub 2011 Oct 12.


Bioluminescent imaging reveals divergent viral pathogenesis in two strains of Stat1-deficient mice, and in αßγ interferon receptor-deficient mice.

Pasieka TJ, Collins L, O'Connor MA, Chen Y, Parker ZM, Berwin BL, Piwnica-Worms DR, Leib DA.

PLoS One. 2011;6(9):e24018. doi: 10.1371/journal.pone.0024018. Epub 2011 Sep 7.


Interferon regulatory factor 3-dependent pathways are critical for control of herpes simplex virus type 1 central nervous system infection.

Menachery VD, Pasieka TJ, Leib DA.

J Virol. 2010 Oct;84(19):9685-94. doi: 10.1128/JVI.00706-10. Epub 2010 Jul 21.


Host responses to wild-type and attenuated herpes simplex virus infection in the absence of Stat1.

Pasieka TJ, Cilloniz C, Lu B, Teal TH, Proll SC, Katze MG, Leib DA.

J Virol. 2009 Mar;83(5):2075-87. doi: 10.1128/JVI.02007-08. Epub 2008 Dec 24.


Enhanced pathogenesis of an attenuated herpes simplex virus for mice lacking Stat1.

Pasieka TJ, Lu B, Leib DA.

J Virol. 2008 Jun;82(12):6052-5. doi: 10.1128/JVI.00297-08. Epub 2008 Apr 9.


Herpes simplex virus virion host shutoff attenuates establishment of the antiviral state.

Pasieka TJ, Lu B, Crosby SD, Wylie KM, Morrison LA, Alexander DE, Menachery VD, Leib DA.

J Virol. 2008 Jun;82(11):5527-35. doi: 10.1128/JVI.02047-07. Epub 2008 Mar 26.


Functional genomic analysis of herpes simplex virus type 1 counteraction of the host innate response.

Pasieka TJ, Baas T, Carter VS, Proll SC, Katze MG, Leib DA.

J Virol. 2006 Aug;80(15):7600-12.


Incorporation of three endocytosed varicella-zoster virus glycoproteins, gE, gH, and gB, into the virion envelope.

Maresova L, Pasieka TJ, Homan E, Gerday E, Grose C.

J Virol. 2005 Jan;79(2):997-1007.


Viral induced fusion and syncytium formation: measurement by the Kolmogorov-Smirnov statistical test.

Pasieka TJ, Woolson RF, Grose C.

J Virol Methods. 2003 Aug;111(2):157-61.


Multimeric humanized varicella-zoster virus antibody fragments to gH neutralize virus while monomeric fragments do not.

Drew PD, Moss MT, Pasieka TJ, Grose C, Harris WJ, Porter AJ.

J Gen Virol. 2001 Aug;82(Pt 8):1959-63.


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