Format
Sort by
Items per page

Send to

Choose Destination

Search results

Items: 7

1.

Open chromatin structures regulate the efficiencies of pre-RC formation and replication initiation in Epstein-Barr virus.

Papior P, Arteaga-Salas JM, G√ľnther T, Grundhoff A, Schepers A.

J Cell Biol. 2012 Aug 20;198(4):509-28. doi: 10.1083/jcb.201109105. Epub 2012 Aug 13.

2.

The latent origin of replication of Epstein-Barr virus directs viral genomes to active regions of the nucleus.

Deutsch MJ, Ott E, Papior P, Schepers A.

J Virol. 2010 Mar;84(5):2533-46. doi: 10.1128/JVI.01909-09. Epub 2009 Dec 23.

3.

Why are we where we are? Understanding replication origins and initiation sites in eukaryotes using ChIP-approaches.

Schepers A, Papior P.

Chromosome Res. 2010 Jan;18(1):63-77. doi: 10.1007/s10577-009-9087-1.

PMID:
19904620
4.

Nuclear signalling by tumour-associated antigen EpCAM.

Maetzel D, Denzel S, Mack B, Canis M, Went P, Benk M, Kieu C, Papior P, Baeuerle PA, Munz M, Gires O.

Nat Cell Biol. 2009 Feb;11(2):162-71. doi: 10.1038/ncb1824. Epub 2009 Jan 11.

PMID:
19136966
6.
7.

PML contributes to a cellular mechanism of repression of herpes simplex virus type 1 infection that is inactivated by ICP0.

Everett RD, Rechter S, Papior P, Tavalai N, Stamminger T, Orr A.

J Virol. 2006 Aug;80(16):7995-8005.

Supplemental Content

Loading ...
Support Center