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Items: 5


Human-like Cmah inactivation in mice increases running endurance and decreases muscle fatigability: implications for human evolution.

Okerblom J, Fletes W, Patel HH, Schenk S, Varki A, Breen EC.

Proc Biol Sci. 2018 Sep 12;285(1886). pii: 20181656. doi: 10.1098/rspb.2018.1656.


Biochemical, Cellular, Physiological, and Pathological Consequences of Human Loss of N-Glycolylneuraminic Acid.

Okerblom J, Varki A.

Chembiochem. 2017 Jul 4;18(13):1155-1171. doi: 10.1002/cbic.201700077. Epub 2017 Jun 9. Review.


Loss of CMAH during Human Evolution Primed the Monocyte-Macrophage Lineage toward a More Inflammatory and Phagocytic State.

Okerblom JJ, Schwarz F, Olson J, Fletes W, Ali SR, Martin PT, Glass CK, Nizet V, Varki A.

J Immunol. 2017 Mar 15;198(6):2366-2373. doi: 10.4049/jimmunol.1601471. Epub 2017 Feb 1.


Interaction of heat shock protein 70 with membranes depends on the lipid environment.

Armijo G, Okerblom J, Cauvi DM, Lopez V, Schlamadinger DE, Kim J, Arispe N, De Maio A.

Cell Stress Chaperones. 2014 Nov;19(6):877-86. doi: 10.1007/s12192-014-0511-x. Epub 2014 May 1.


A comparative study of N-glycolylneuraminic acid (Neu5Gc) and cytotoxic T cell (CT) carbohydrate expression in normal and dystrophin-deficient dog and human skeletal muscle.

Martin PT, Golden B, Okerblom J, Camboni M, Chandrasekharan K, Xu R, Varki A, Flanigan KM, Kornegay JN.

PLoS One. 2014 Feb 5;9(2):e88226. doi: 10.1371/journal.pone.0088226. eCollection 2014.

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