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Items: 5

1.

B56γ tumor-associated mutations provide new mechanisms for B56γ-PP2A tumor suppressor activity.

Nobumori Y, Shouse GP, Wu Y, Lee KJ, Shen B, Liu X.

Mol Cancer Res. 2013 Sep;11(9):995-1003. doi: 10.1158/1541-7786.MCR-12-0633. Epub 2013 May 30.

2.

Structure of the C-terminal half of human XPB helicase and the impact of the disease-causing mutation XP11BE.

Hilario E, Li Y, Nobumori Y, Liu X, Fan L.

Acta Crystallogr D Biol Crystallogr. 2013 Feb;69(Pt 2):237-46. doi: 10.1107/S0907444912045040. Epub 2013 Jan 19.

PMID:
23385459
3.

HEAT repeat 1 motif is required for B56γ-containing protein phosphatase 2A (B56γ-PP2A) holoenzyme assembly and tumor-suppressive function.

Nobumori Y, Shouse GP, Fan L, Liu X.

J Biol Chem. 2012 Mar 30;287(14):11030-6. doi: 10.1074/jbc.M111.334094. Epub 2012 Feb 7.

4.

ATM-mediated phosphorylation activates the tumor-suppressive function of B56γ-PP2A.

Shouse GP, Nobumori Y, Panowicz MJ, Liu X.

Oncogene. 2011 Sep 1;30(35):3755-65. doi: 10.1038/onc.2011.95. Epub 2011 Apr 4.

PMID:
21460856
5.

A B56gamma mutation in lung cancer disrupts the p53-dependent tumor-suppressor function of protein phosphatase 2A.

Shouse GP, Nobumori Y, Liu X.

Oncogene. 2010 Jul 8;29(27):3933-41. doi: 10.1038/onc.2010.161. Epub 2010 May 17.

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