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Items: 5

1.

Epithelial cells augment barrier function via activation of the Toll-like receptor 2/phosphatidylinositol 3-kinase pathway upon recognition of Salmonella enterica serovar Typhimurium curli fibrils in the gut.

Oppong GO, Rapsinski GJ, Newman TN, Nishimori JH, Biesecker SG, Tükel Ç.

Infect Immun. 2013 Feb;81(2):478-86. doi: 10.1128/IAI.00453-12. Epub 2012 Dec 3.

2.

Microbial amyloids induce interleukin 17A (IL-17A) and IL-22 responses via Toll-like receptor 2 activation in the intestinal mucosa.

Nishimori JH, Newman TN, Oppong GO, Rapsinski GJ, Yen JH, Biesecker SG, Wilson RP, Butler BP, Winter MG, Tsolis RM, Ganea D, Tükel Ç.

Infect Immun. 2012 Dec;80(12):4398-408. doi: 10.1128/IAI.00911-12. Epub 2012 Oct 1.

3.

The Vi capsular polysaccharide prevents complement receptor 3-mediated clearance of Salmonella enterica serotype Typhi.

Wilson RP, Winter SE, Spees AM, Winter MG, Nishimori JH, Sanchez JF, Nuccio SP, Crawford RW, Tükel Ç, Bäumler AJ.

Infect Immun. 2011 Feb;79(2):830-7. doi: 10.1128/IAI.00961-10. Epub 2010 Nov 22.

4.

Toll-like receptors 1 and 2 cooperatively mediate immune responses to curli, a common amyloid from enterobacterial biofilms.

Tükel C, Nishimori JH, Wilson RP, Winter MG, Keestra AM, van Putten JP, Bäumler AJ.

Cell Microbiol. 2010 Oct;12(10):1495-505. doi: 10.1111/j.1462-5822.2010.01485.x.

5.

Responses to amyloids of microbial and host origin are mediated through toll-like receptor 2.

Tükel C, Wilson RP, Nishimori JH, Pezeshki M, Chromy BA, Bäumler AJ.

Cell Host Microbe. 2009 Jul 23;6(1):45-53. doi: 10.1016/j.chom.2009.05.020.

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