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Items: 8


Incomplete prophage tolerance by type III-A CRISPR-Cas systems reduces the fitness of lysogenic hosts.

Goldberg GW, McMillan EA, Varble A, Modell JW, Samai P, Jiang W, Marraffini LA.

Nat Commun. 2018 Jan 4;9(1):61. doi: 10.1038/s41467-017-02557-2.


CRISPR-Cas systems exploit viral DNA injection to establish and maintain adaptive immunity.

Modell JW, Jiang W, Marraffini LA.

Nature. 2017 Apr 6;544(7648):101-104. doi: 10.1038/nature21719. Epub 2017 Mar 29.


Cas9 specifies functional viral targets during CRISPR-Cas adaptation.

Heler R, Samai P, Modell JW, Weiner C, Goldberg GW, Bikard D, Marraffini LA.

Nature. 2015 Mar 12;519(7542):199-202. doi: 10.1038/nature14245. Epub 2015 Feb 18.


A DNA damage-induced, SOS-independent checkpoint regulates cell division in Caulobacter crescentus.

Modell JW, Kambara TK, Perchuk BS, Laub MT.

PLoS Biol. 2014 Oct 28;12(10):e1001977. doi: 10.1371/journal.pbio.1001977. eCollection 2014 Oct.


A DNA damage checkpoint in Caulobacter crescentus inhibits cell division through a direct interaction with FtsW.

Modell JW, Hopkins AC, Laub MT.

Genes Dev. 2011 Jun 15;25(12):1328-43. doi: 10.1101/gad.2038911. Erratum in: Genes Dev. 2011 Aug 1;25(15):1662.


Mitochondrial gradients and p38 activity in early sea urchin embryos.

Modell JW, Bradham CA.

Mol Reprod Dev. 2011 Apr;78(4):225. doi: 10.1002/mrd.21302. Epub 2011 Mar 8. No abstract available.


Chordin is required for neural but not axial development in sea urchin embryos.

Bradham CA, Oikonomou C, Kühn A, Core AB, Modell JW, McClay DR, Poustka AJ.

Dev Biol. 2009 Apr 15;328(2):221-33. doi: 10.1016/j.ydbio.2009.01.027. Epub 2009 Jan 29.


The Connectivity Map: using gene-expression signatures to connect small molecules, genes, and disease.

Lamb J, Crawford ED, Peck D, Modell JW, Blat IC, Wrobel MJ, Lerner J, Brunet JP, Subramanian A, Ross KN, Reich M, Hieronymus H, Wei G, Armstrong SA, Haggarty SJ, Clemons PA, Wei R, Carr SA, Lander ES, Golub TR.

Science. 2006 Sep 29;313(5795):1929-35.

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