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Items: 17

1.

Epstein-Barr virus-encoded EBNA1 and ZEBRA: targets for therapeutic strategies against EBV-carrying cancers.

Daskalogianni C, Pyndiah S, Apcher S, Mazars A, Manoury B, Ammari N, Nylander K, Voisset C, Blondel M, Fåhraeus R.

J Pathol. 2015 Jan;235(2):334-41. doi: 10.1002/path.4431. Review.

PMID:
25186125
2.

A yeast-based assay identifies drugs that interfere with immune evasion of the Epstein-Barr virus.

Voisset C, Daskalogianni C, Contesse MA, Mazars A, Arbach H, Le Cann M, Soubigou F, Apcher S, Fåhraeus R, Blondel M.

Dis Model Mech. 2014 Apr;7(4):435-44. doi: 10.1242/dmm.014308. Epub 2014 Feb 20.

3.

The p53 mRNA-Mdm2 interaction controls Mdm2 nuclear trafficking and is required for p53 activation following DNA damage.

Gajjar M, Candeias MM, Malbert-Colas L, Mazars A, Fujita J, Olivares-Illana V, Fåhraeus R.

Cancer Cell. 2012 Jan 17;21(1):25-35. doi: 10.1016/j.ccr.2011.11.016.

4.

Using BRET to study chemical compound-induced disruptions of the p53-HDM2 interactions in live cells.

Mazars A, Fåhraeus R.

Biotechnol J. 2010 Apr;5(4):377-84. doi: 10.1002/biot.200900272.

5.

Designing focused chemical libraries enriched in protein-protein interaction inhibitors using machine-learning methods.

Reynès C, Host H, Camproux AC, Laconde G, Leroux F, Mazars A, Deprez B, Fahraeus R, Villoutreix BO, Sperandio O.

PLoS Comput Biol. 2010 Mar 5;6(3):e1000695. doi: 10.1371/journal.pcbi.1000695.

6.

Upregulation of the CDC25A phosphatase down-stream of the NPM/ALK oncogene participates to anaplastic large cell lymphoma enhanced proliferation.

Fernandez-Vidal A, Mazars A, Gautier EF, Prévost G, Payrastre B, Manenti S.

Cell Cycle. 2009 May 1;8(9):1373-9. Epub 2009 May 26.

PMID:
19305144
7.

CDC25A: a rebel within the CDC25 phosphatases family?

Fernandez-Vidal A, Mazars A, Manenti S.

Anticancer Agents Med Chem. 2008 Dec;8(8):825-31. Review.

PMID:
19075564
8.

A caspase-dependent cleavage of CDC25A generates an active fragment activating cyclin-dependent kinase 2 during apoptosis.

Mazars A, Fernandez-Vidal A, Mondesert O, Lorenzo C, Prévost G, Ducommun B, Payrastre B, Racaud-Sultan C, Manenti S.

Cell Death Differ. 2009 Feb;16(2):208-18. doi: 10.1038/cdd.2008.142. Epub 2008 Oct 17.

9.

[The Bcl-2 family of proteins as drug targets].

Mazars A, Geneste O, Hickman J.

J Soc Biol. 2005;199(3):253-65. Review. French.

PMID:
16471266
10.

Transforming growth factor-beta inhibition of insulin-like growth factor-binding protein-5 synthesis in skeletal muscle cells involves a c-Jun N-terminal kinase-dependent pathway.

Rousse S, Lallemand F, Montarras D, Pinset C, Mazars A, Prunier C, Atfi A, Dubois C.

J Biol Chem. 2001 Dec 14;276(50):46961-7. Epub 2001 Oct 11.

11.

Evidence for a role of the JNK cascade in Smad7-mediated apoptosis.

Mazars A, Lallemand F, Prunier C, Marais J, Ferrand N, Pessah M, Cherqui G, Atfi A.

J Biol Chem. 2001 Sep 28;276(39):36797-803. Epub 2001 Jul 26.

12.

c-Jun interacts with the corepressor TG-interacting factor (TGIF) to suppress Smad2 transcriptional activity.

Pessah M, Prunier C, Marais J, Ferrand N, Mazars A, Lallemand F, Gauthier JM, Atfi A.

Proc Natl Acad Sci U S A. 2001 May 22;98(11):6198-203.

13.

Smad7 inhibits the survival nuclear factor kappaB and potentiates apoptosis in epithelial cells.

Lallemand F, Mazars A, Prunier C, Bertrand F, Kornprost M, Gallea S, Roman-Roman S, Cherqui G, Atfi A.

Oncogene. 2001 Feb 15;20(7):879-84.

14.

Differential roles of JNK and Smad2 signaling pathways in the inhibition of c-Myc-induced cell death by TGF-beta.

Mazars A, Tournigand C, Mollat P, Prunier C, Ferrand N, Bourgeade MF, Gespach C, Atfi A.

Oncogene. 2000 Mar 2;19(10):1277-87.

15.

The oncogenic TEL/PDGFR beta fusion protein induces cell death through JNK/SAPK pathway.

Atfi A, Prunier C, Mazars A, Défachelles AS, Cayre Y, Gespach C, Bourgeade MF.

Oncogene. 1999 Jul 1;18(26):3878-85.

16.

Evidence that Smad2 is a tumor suppressor implicated in the control of cellular invasion.

Prunier C, Mazars A, Noë V, Bruyneel E, Mareel M, Gespach C, Atfi A.

J Biol Chem. 1999 Aug 13;274(33):22919-22.

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