Format
Sort by
Items per page

Send to

Choose Destination

Search results

Items: 11

1.

The enigma of an interconnected mitochondrial reticulum: new insights into mitochondrial fusion.

Mattie S, Krols M, McBride HM.

Curr Opin Cell Biol. 2019 Aug;59:159-166. doi: 10.1016/j.ceb.2019.05.004. Epub 2019 Jun 25. Review.

PMID:
31252211
2.

Visualization of SNARE-Mediated Organelle Membrane Hemifusion by Electron Microscopy.

Mattie S, Kazmirchuk T, Mui J, Vali H, Brett CL.

Methods Mol Biol. 2019;1860:361-377. doi: 10.1007/978-1-4939-8760-3_24.

PMID:
30317518
3.

Rab-Effector-Kinase Interplay Modulates Intralumenal Fragment Formation during Vacuole Fusion.

Karim MA, McNally EK, Samyn DR, Mattie S, Brett CL.

Dev Cell. 2018 Oct 8;47(1):80-97.e6. doi: 10.1016/j.devcel.2018.09.002. Epub 2018 Sep 27.

PMID:
30269949
4.

A new mitofusin topology places the redox-regulated C terminus in the mitochondrial intermembrane space.

Mattie S, Riemer J, Wideman JG, McBride HM.

J Cell Biol. 2018 Feb 5;217(2):507-515. doi: 10.1083/jcb.201611194. Epub 2017 Dec 6.

5.

Distinct features of multivesicular body-lysosome fusion revealed by a new cell-free content-mixing assay.

Karim MA, Samyn DR, Mattie S, Brett CL.

Traffic. 2018 Feb;19(2):138-149. doi: 10.1111/tra.12543. Epub 2017 Dec 10.

6.

Newly born peroxisomes are a hybrid of mitochondrial and ER-derived pre-peroxisomes.

Sugiura A, Mattie S, Prudent J, McBride HM.

Nature. 2017 Feb 9;542(7640):251-254. doi: 10.1038/nature21375. Epub 2017 Feb 1.

PMID:
28146471
7.

How and why intralumenal membrane fragments form during vacuolar lysosome fusion.

Mattie S, McNally EK, Karim MA, Vali H, Brett CL.

Mol Biol Cell. 2017 Jan 15;28(2):309-321. doi: 10.1091/mbc.E15-11-0759. Epub 2016 Nov 23.

8.

MAPL SUMOylation of Drp1 Stabilizes an ER/Mitochondrial Platform Required for Cell Death.

Prudent J, Zunino R, Sugiura A, Mattie S, Shore GC, McBride HM.

Mol Cell. 2015 Sep 17;59(6):941-55. doi: 10.1016/j.molcel.2015.08.001.

9.

Macromitophagy, neutral lipids synthesis, and peroxisomal fatty acid oxidation protect yeast from "liponecrosis", a previously unknown form of programmed cell death.

Sheibani S, Richard VR, Beach A, Leonov A, Feldman R, Mattie S, Khelghatybana L, Piano A, Greenwood M, Vali H, Titorenko VI.

Cell Cycle. 2014;13(1):138-47. doi: 10.4161/cc.26885. Epub 2013 Oct 28.

10.

Macromitophagy is a longevity assurance process that in chronologically aging yeast limited in calorie supply sustains functional mitochondria and maintains cellular lipid homeostasis.

Richard VR, Leonov A, Beach A, Burstein MT, Koupaki O, Gomez-Perez A, Levy S, Pluska L, Mattie S, Rafesh R, Iouk T, Sheibani S, Greenwood M, Vali H, Titorenko VI.

Aging (Albany NY). 2013 Apr;5(4):234-69.

11.

The Saccharomyces cerevisiae enolase-related regions encode proteins that are active enolases.

Kornblatt MJ, Richard Albert J, Mattie S, Zakaib J, Dayanandan S, Hanic-Joyce PJ, Joyce PB.

Yeast. 2013 Feb;30(2):55-69. doi: 10.1002/yea.2940. Epub 2013 Jan 28.

Supplemental Content

Loading ...
Support Center