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Items: 4

1.

SLX4IP Antagonizes Promiscuous BLM Activity during ALT Maintenance.

Panier S, Maric M, Hewitt G, Mason-Osann E, Gali H, Dai A, Labadorf A, Guervilly JH, Ruis P, Segura-Bayona S, Belan O, Marzec P, Gaillard PHL, Flynn RL, Boulton SJ.

Mol Cell. 2019 Oct 3;76(1):27-43.e11. doi: 10.1016/j.molcel.2019.07.010. Epub 2019 Aug 22.

2.

Erratum: Molecular basis of telomere dysfunction in human genetic diseases.

Sarek G, Marzec P, Margalef P, Boulton SJ.

Nat Struct Mol Biol. 2017 Jun 6;24(6):553. doi: 10.1038/nsmb0617-553a. No abstract available.

PMID:
28586326
3.

Molecular basis of telomere dysfunction in human genetic diseases.

Sarek G, Marzec P, Margalef P, Boulton SJ.

Nat Struct Mol Biol. 2015 Nov;22(11):867-74. doi: 10.1038/nsmb.3093. Review. Erratum in: Nat Struct Mol Biol. 2017 Jun 6;24(6):553.

PMID:
26581521
4.

Nuclear-receptor-mediated telomere insertion leads to genome instability in ALT cancers.

Marzec P, Armenise C, Pérot G, Roumelioti FM, Basyuk E, Gagos S, Chibon F, Déjardin J.

Cell. 2015 Feb 26;160(5):913-927. doi: 10.1016/j.cell.2015.01.044.

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