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Items: 6

1.

Calcium dysregulation, functional calpainopathy, and endoplasmic reticulum stress in sporadic inclusion body myositis.

Amici DR, Pinal-Fernandez I, Mázala DA, Lloyd TE, Corse AM, Christopher-Stine L, Mammen AL, Chin ER.

Acta Neuropathol Commun. 2017 Mar 22;5(1):24. doi: 10.1186/s40478-017-0427-7.

2.

NAD+ repletion improves muscle function in muscular dystrophy and counters global PARylation.

Ryu D, Zhang H, Ropelle ER, Sorrentino V, Mázala DA, Mouchiroud L, Marshall PL, Campbell MD, Ali AS, Knowels GM, Bellemin S, Iyer SR, Wang X, Gariani K, Sauve AA, Cantó C, Conley KE, Walter L, Lovering RM, Chin ER, Jasmin BJ, Marcinek DJ, Menzies KJ, Auwerx J.

Sci Transl Med. 2016 Oct 19;8(361):361ra139.

3.

The SH3 and cysteine-rich domain 3 (Stac3) gene is important to growth, fiber composition, and calcium release from the sarcoplasmic reticulum in postnatal skeletal muscle.

Cong X, Doering J, Mazala DA, Chin ER, Grange RW, Jiang H.

Skelet Muscle. 2016 Apr 11;6:17. doi: 10.1186/s13395-016-0088-4. eCollection 2016.

4.

SERCA1 overexpression minimizes skeletal muscle damage in dystrophic mouse models.

Mázala DA, Pratt SJP, Chen D, Molkentin JD, Lovering RM, Chin ER.

Am J Physiol Cell Physiol. 2015 May 1;308(9):C699-709. doi: 10.1152/ajpcell.00341.2014. Epub 2015 Feb 4.

5.

The role of proteases in excitation-contraction coupling failure in muscular dystrophy.

Mázala DA, Grange RW, Chin ER.

Am J Physiol Cell Physiol. 2015 Jan 1;308(1):C33-40. doi: 10.1152/ajpcell.00267.2013. Epub 2014 Oct 8.

6.

Perturbations in intracellular Ca2+ handling in skeletal muscle in the G93A*SOD1 mouse model of amyotrophic lateral sclerosis.

Chin ER, Chen D, Bobyk KD, Mázala DA.

Am J Physiol Cell Physiol. 2014 Dec 1;307(11):C1031-8. doi: 10.1152/ajpcell.00237.2013. Epub 2014 Sep 24.

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