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Items: 5

1.

Rare homozygosity in amyotrophic lateral sclerosis suggests the contribution of recessive variants to disease genetics.

Goldstein O, Kedmi M, Gana-Weisz M, Twito S, Nefussy B, Vainer B, Fainmesser Y, Abraham A, Nayshool O, Orr-Urtreger A, Drory VE.

J Neurol Sci. 2019 Jul 15;402:62-68. doi: 10.1016/j.jns.2019.05.006. Epub 2019 May 8.

PMID:
31108397
2.

MiR-1275 promotes non-small cell lung cancer cell proliferation and metastasis by regulating LZTS3 expression.

He J, Yu L, Wang CM, Zhou XF.

Eur Rev Med Pharmacol Sci. 2018 May;22(9):2680-2687. doi: 10.26355/eurrev_201805_14964.

3.

The Shank3 Interaction Partner ProSAPiP1 Regulates Postsynaptic SPAR Levels and the Maturation of Dendritic Spines in Hippocampal Neurons.

Reim D, Weis TM, Halbedl S, Delling JP, Grabrucker AM, Boeckers TM, Schmeisser MJ.

Front Synaptic Neurosci. 2016 May 24;8:13. doi: 10.3389/fnsyn.2016.00013. eCollection 2016.

4.

Sipa1l3/SPAR3 is targeted to postsynaptic specializations and interacts with the Fezzin ProSAPiP1/Lzts3.

Dolnik A, Kanwal N, Mackert S, Halbedl S, Proepper C, Bockmann J, Schoen M, Boeckers TM, Kühl SJ, Schmeisser MJ.

J Neurochem. 2016 Jan;136(1):28-35. doi: 10.1111/jnc.13353. Epub 2015 Oct 8.

5.

In silico characterization of LZTS3, a potential tumor suppressor.

Teufel A, Weinmann A, Galle PR, Lohse AW.

Oncol Rep. 2005 Aug;14(2):547-51.

PMID:
16012743

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