Format
Sort by

Send to

Choose Destination

Search results

Items: 4

1.

Cells isolated from residual intracranial tumors after treatment express iPSC genes and possess neural lineage differentiation plasticity.

Palanichamy K, Jacob JR, Litzenberg KT, Ray-Chaudhury A, Chakravarti A.

EBioMedicine. 2018 Oct;36:281-292. doi: 10.1016/j.ebiom.2018.09.019. Epub 2018 Sep 27.

2.

Lack of Constitutively Active DNA Repair Sensitizes Glioblastomas to Akt Inhibition and Induces Synthetic Lethality with Radiation Treatment in a p53-Dependent Manner.

Palanichamy K, Patel D, Jacob JR, Litzenberg KT, Gordon N, Acus K, Noda SE, Chakravarti A.

Mol Cancer Ther. 2018 Feb;17(2):336-346. doi: 10.1158/1535-7163.MCT-17-0429. Epub 2017 Aug 24.

3.

NNMT Silencing Activates Tumor Suppressor PP2A, Inactivates Oncogenic STKs, and Inhibits Tumor Forming Ability.

Palanichamy K, Kanji S, Gordon N, Thirumoorthy K, Jacob JR, Litzenberg KT, Patel D, Chakravarti A.

Clin Cancer Res. 2017 May 1;23(9):2325-2334. doi: 10.1158/1078-0432.CCR-16-1323. Epub 2016 Nov 3.

4.

Methionine and Kynurenine Activate Oncogenic Kinases in Glioblastoma, and Methionine Deprivation Compromises Proliferation.

Palanichamy K, Thirumoorthy K, Kanji S, Gordon N, Singh R, Jacob JR, Sebastian N, Litzenberg KT, Patel D, Bassett E, Ramasubramanian B, Lautenschlaeger T, Fischer SM, Ray-Chaudhury A, Chakravarti A.

Clin Cancer Res. 2016 Jul 15;22(14):3513-23. doi: 10.1158/1078-0432.CCR-15-2308. Epub 2016 Mar 2.

Supplemental Content

Loading ...
Support Center