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Items: 21

1.

Erratum: Oncostatin M drives intestinal inflammation and predicts response to tumor necrosis factor-neutralizing therapy in patients with inflammatory bowel disease.

West NR, Hegazy AN, Owens BMJ, Bullers SJ, Linggi B, Buonocore S, Coccia M, Görtz D, This S, Stockenhuber K, Pott J, Friedrich M, Ryzhakov G, Baribaud F, Brodmerkel C, Cieluch C, Rahman N, Müller-Newen G, Owens RJ, Kühl AA, Maloy KJ, Plevy SE; Oxford IBD Cohort Investigators, Keshav S, Travis SPL, Powrie F.

Nat Med. 2017 Jun 6;23(6):788. doi: 10.1038/nm0617-788d. No abstract available.

PMID:
28586341
2.

Oncostatin M drives intestinal inflammation and predicts response to tumor necrosis factor-neutralizing therapy in patients with inflammatory bowel disease.

West NR, Hegazy AN, Owens BMJ, Bullers SJ, Linggi B, Buonocore S, Coccia M, Görtz D, This S, Stockenhuber K, Pott J, Friedrich M, Ryzhakov G, Baribaud F, Brodmerkel C, Cieluch C, Rahman N, Müller-Newen G, Owens RJ, Kühl AA, Maloy KJ, Plevy SE; Oxford IBD Cohort Investigators, Keshav S, Travis SPL, Powrie F.

Nat Med. 2017 May;23(5):579-589. doi: 10.1038/nm.4307. Epub 2017 Apr 3. Erratum in: Nat Med. 2017 Jun 6;23 (6):788.

3.

CO2 exposure at pressure impacts metabolism and stress responses in the model sulfate-reducing bacterium Desulfovibrio vulgaris strain Hildenborough.

Wilkins MJ, Hoyt DW, Marshall MJ, Alderson PA, Plymale AE, Markillie LM, Tucker AE, Walter ED, Linggi BE, Dohnalkova AC, Taylor RC.

Front Microbiol. 2014 Sep 25;5:507. doi: 10.3389/fmicb.2014.00507. eCollection 2014.

4.

Inference of interactions in cyanobacterial-heterotrophic co-cultures via transcriptome sequencing.

Beliaev AS, Romine MF, Serres M, Bernstein HC, Linggi BE, Markillie LM, Isern NG, Chrisler WB, Kucek LA, Hill EA, Pinchuk GE, Bryant DA, Wiley HS, Fredrickson JK, Konopka A.

ISME J. 2014 Nov;8(11):2243-55. doi: 10.1038/ismej.2014.69. Epub 2014 Apr 29.

5.

DNA methylation as a biomarker for preeclampsia.

Anderson CM, Ralph JL, Wright ML, Linggi B, Ohm JE.

Biol Res Nurs. 2014 Oct;16(4):409-20. doi: 10.1177/1099800413508645. Epub 2013 Oct 28.

PMID:
24165327
6.

Changes in translational efficiency is a dominant regulatory mechanism in the environmental response of bacteria.

Taylor RC, Webb Robertson BJ, Markillie LM, Serres MH, Linggi BE, Aldrich JT, Hill EA, Romine MF, Lipton MS, Wiley HS.

Integr Biol (Camb). 2013 Nov;5(11):1393-406. doi: 10.1039/c3ib40120k. Epub 2013 Sep 30.

7.

Coregulation of Terpenoid Pathway Genes and Prediction of Isoprene Production in Bacillus subtilis Using Transcriptomics.

Hess BM, Xue J, Markillie LM, Taylor RC, Wiley HS, Ahring BK, Linggi B.

PLoS One. 2013 Jun 19;8(6):e66104. doi: 10.1371/journal.pone.0066104. Print 2013.

8.

Improving RNA-Seq Precision with MapAl.

Labaj PP, Linggi BE, Wiley HS, Kreil DP.

Front Genet. 2012 Apr 2;3:28. doi: 10.3389/fgene.2012.00028. eCollection 2012.

9.

Characterization and improvement of RNA-Seq precision in quantitative transcript expression profiling.

Łabaj PP, Leparc GG, Linggi BE, Markillie LM, Wiley HS, Kreil DP.

Bioinformatics. 2011 Jul 1;27(13):i383-91. doi: 10.1093/bioinformatics/btr247.

10.

Therapeutically targeting ErbB3: a key node in ligand-induced activation of the ErbB receptor-PI3K axis.

Schoeberl B, Pace EA, Fitzgerald JB, Harms BD, Xu L, Nie L, Linggi B, Kalra A, Paragas V, Bukhalid R, Grantcharova V, Kohli N, West KA, Leszczyniecka M, Feldhaus MJ, Kudla AJ, Nielsen UB.

Sci Signal. 2009 Jun 30;2(77):ra31. doi: 10.1126/scisignal.2000352.

11.

ErbB receptors: new insights on mechanisms and biology.

Linggi B, Carpenter G.

Trends Cell Biol. 2006 Dec;16(12):649-56. Epub 2006 Nov 7. Review.

PMID:
17085050
12.

ErbB-4 s80 intracellular domain abrogates ETO2-dependent transcriptional repression.

Linggi B, Carpenter G.

J Biol Chem. 2006 Sep 1;281(35):25373-80. Epub 2006 Jun 30.

13.

The ErbB-4 s80 intracellular domain is a constitutively active tyrosine kinase.

Linggi B, Cheng QC, Rao AR, Carpenter G.

Oncogene. 2006 Jan 5;25(1):160-3.

PMID:
16170367
14.

Translating the histone code into leukemia.

Linggi BE, Brandt SJ, Sun ZW, Hiebert SW.

J Cell Biochem. 2005 Dec 1;96(5):938-50. Review.

PMID:
16167339
15.

Specific protein redirection as a transcriptional therapy approach for t(8;21) leukemia.

Steffen B, Serve H, Berdel WE, Agrawal S, Linggi B, Büchner T, Hiebert SW, Müller-Tidow C.

Proc Natl Acad Sci U S A. 2003 Jul 8;100(14):8448-53. Epub 2003 Jun 20.

16.

The t(8;21) fusion protein contacts co-repressors and histone deacetylases to repress the transcription of the p14ARF tumor suppressor.

Hiebert SW, Reed-Inderbitzin EF, Amann J, Irvin B, Durst K, Linggi B.

Blood Cells Mol Dis. 2003 Mar-Apr;30(2):177-83. Review.

PMID:
12732181
17.

The t(8;21) fusion protein, AML1 ETO, specifically represses the transcription of the p14(ARF) tumor suppressor in acute myeloid leukemia.

Linggi B, Müller-Tidow C, van de Locht L, Hu M, Nip J, Serve H, Berdel WE, van der Reijden B, Quelle DE, Rowley JD, Cleveland J, Jansen JH, Pandolfi PP, Hiebert SW.

Nat Med. 2002 Jul;8(7):743-50. Epub 2002 Jun 24.

PMID:
12091906
18.

Mechanisms of transcriptional repression by the t(8;21)-, t(12;21)-, and inv(16)-encoded fusion proteins.

Heibert SW, Lutterbach B, Durst K, Wang L, Linggi B, Wu S, Wood L, Amann J, King D, Hou Y.

Cancer Chemother Pharmacol. 2001 Aug;48 Suppl 1:S31-4. Review.

PMID:
11587363
19.

Expression of the AML-1 oncogene shortens the G(1) phase of the cell cycle.

Strom DK, Nip J, Westendorf JJ, Linggi B, Lutterbach B, Downing JR, Lenny N, Hiebert SW.

J Biol Chem. 2000 Feb 4;275(5):3438-45.

20.

A mechanism of repression by acute myeloid leukemia-1, the target of multiple chromosomal translocations in acute leukemia.

Lutterbach B, Westendorf JJ, Linggi B, Isaac S, Seto E, Hiebert SW.

J Biol Chem. 2000 Jan 7;275(1):651-6.

21.

ETO, a target of t(8;21) in acute leukemia, interacts with the N-CoR and mSin3 corepressors.

Lutterbach B, Westendorf JJ, Linggi B, Patten A, Moniwa M, Davie JR, Huynh KD, Bardwell VJ, Lavinsky RM, Rosenfeld MG, Glass C, Seto E, Hiebert SW.

Mol Cell Biol. 1998 Dec;18(12):7176-84.

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