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Items: 4

1.

Publisher Correction: Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug.

Barak B, Zhang Z, Liu Y, Nir A, Trangle SS, Ennis M, Levandowski KM, Wang D, Quast K, Boulting GL, Li Y, Bayarsaihan D, He Z, Feng G.

Nat Neurosci. 2019 Jul;22(7):1197. doi: 10.1038/s41593-019-0441-0.

PMID:
31171854
2.

Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug.

Barak B, Zhang Z, Liu Y, Nir A, Trangle SS, Ennis M, Levandowski KM, Wang D, Quast K, Boulting GL, Li Y, Bayarsaihan D, He Z, Feng G.

Nat Neurosci. 2019 May;22(5):700-708. doi: 10.1038/s41593-019-0380-9. Epub 2019 Apr 22. Erratum in: Nat Neurosci. 2019 Jul;22(7):1197.

PMID:
31011227
3.

Homeostatic circuits selectively gate food cue responses in insular cortex.

Livneh Y, Ramesh RN, Burgess CR, Levandowski KM, Madara JC, Fenselau H, Goldey GJ, Diaz VE, Jikomes N, Resch JM, Lowell BB, Andermann ML.

Nature. 2017 Jun 29;546(7660):611-616. doi: 10.1038/nature22375. Epub 2017 Jun 14.

4.

Hunger-Dependent Enhancement of Food Cue Responses in Mouse Postrhinal Cortex and Lateral Amygdala.

Burgess CR, Ramesh RN, Sugden AU, Levandowski KM, Minnig MA, Fenselau H, Lowell BB, Andermann ML.

Neuron. 2016 Sep 7;91(5):1154-1169. doi: 10.1016/j.neuron.2016.07.032. Epub 2016 Aug 11.

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