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Items: 8

1.

Xist Deletional Analysis Reveals an Interdependency between Xist RNA and Polycomb Complexes for Spreading along the Inactive X.

Colognori D, Sunwoo H, Kriz AJ, Wang CY, Lee JT.

Mol Cell. 2019 Feb 28. pii: S1097-2765(19)30035-8. doi: 10.1016/j.molcel.2019.01.015. [Epub ahead of print]

PMID:
30827740
2.

Exploration of CTCF post-translation modifications uncovers Serine-224 phosphorylation by PLK1 at pericentric regions during the G2/M transition.

Del Rosario BC, Kriz AJ, Del Rosario AM, Anselmo A, Fry CJ, White FM, Sadreyev RI, Lee JT.

Elife. 2019 Jan 24;8. pii: e42341. doi: 10.7554/eLife.42341.

3.

Megadomains and superloops form dynamically but are dispensable for X-chromosome inactivation and gene escape.

Froberg JE, Pinter SF, Kriz AJ, J├ęgu T, Lee JT.

Nat Commun. 2018 Nov 27;9(1):5004. doi: 10.1038/s41467-018-07446-w.

4.

Transcriptional Pause Sites Delineate Stable Nucleosome-Associated Premature Polyadenylation Suppressed by U1 snRNP.

Chiu AC, Suzuki HI, Wu X, Mahat DB, Kriz AJ, Sharp PA.

Mol Cell. 2018 Feb 15;69(4):648-663.e7. doi: 10.1016/j.molcel.2018.01.006. Epub 2018 Feb 1.

5.

In vivo genome editing using Staphylococcus aureus Cas9.

Ran FA, Cong L, Yan WX, Scott DA, Gootenberg JS, Kriz AJ, Zetsche B, Shalem O, Wu X, Makarova KS, Koonin EV, Sharp PA, Zhang F.

Nature. 2015 Apr 9;520(7546):186-91. doi: 10.1038/nature14299. Epub 2015 Apr 1.

6.

Target specificity of the CRISPR-Cas9 system.

Wu X, Kriz AJ, Sharp PA.

Quant Biol. 2014 Jun;2(2):59-70.

7.

Genome-wide binding of the CRISPR endonuclease Cas9 in mammalian cells.

Wu X, Scott DA, Kriz AJ, Chiu AC, Hsu PD, Dadon DB, Cheng AW, Trevino AE, Konermann S, Chen S, Jaenisch R, Zhang F, Sharp PA.

Nat Biotechnol. 2014 Jul;32(7):670-6. doi: 10.1038/nbt.2889. Epub 2014 Apr 20.

8.

Promoter directionality is controlled by U1 snRNP and polyadenylation signals.

Almada AE, Wu X, Kriz AJ, Burge CB, Sharp PA.

Nature. 2013 Jul 18;499(7458):360-3. doi: 10.1038/nature12349. Epub 2013 Jun 23.

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