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Items: 9

1.

A recurrent cancer-associated substitution in DNA polymerase ε produces a hyperactive enzyme.

Xing X, Kane DP, Bulock CR, Moore EA, Sharma S, Chabes A, Shcherbakova PV.

Nat Commun. 2019 Jan 22;10(1):374. doi: 10.1038/s41467-018-08145-2.

2.

Functional Analysis of Cancer-Associated DNA Polymerase ε Variants in Saccharomyces cerevisiae.

Barbari SR, Kane DP, Moore EA, Shcherbakova PV.

G3 (Bethesda). 2018 Mar 2;8(3):1019-1029. doi: 10.1534/g3.118.200042.

3.

A common cancer-associated DNA polymerase ε mutation causes an exceptionally strong mutator phenotype, indicating fidelity defects distinct from loss of proofreading.

Kane DP, Shcherbakova PV.

Cancer Res. 2014 Apr 1;74(7):1895-901. doi: 10.1158/0008-5472.CAN-13-2892. Epub 2014 Feb 13.

4.

Competition between replicative and translesion polymerases during homologous recombination repair in Drosophila.

Kane DP, Shusterman M, Rong Y, McVey M.

PLoS Genet. 2012;8(4):e1002659. doi: 10.1371/journal.pgen.1002659. Epub 2012 Apr 19.

5.

Super-sized deletions: improved transposon excision screens using a mus309 mutant background.

Witsell A, Kane DP, McVey M.

Fly (Austin). 2010 Apr-Jun;4(2):137-40. Epub 2010 Apr 9.

PMID:
20081362
6.

Removal of the bloom syndrome DNA helicase extends the utility of imprecise transposon excision for making null mutations in Drosophila.

Witsell A, Kane DP, Rubin S, McVey M.

Genetics. 2009 Nov;183(3):1187-93. doi: 10.1534/genetics.109.108472. Epub 2009 Aug 17.

7.

State II dissociation element formation following activator excision in maize.

Conrad LJ, Bai L, Ahern K, Dusinberre K, Kane DP, Brutnell TP.

Genetics. 2007 Oct;177(2):737-47. Epub 2007 Aug 24.

8.
9.

Identification of a novel oligosaccharide backbone structure with a galactose residue monosubstituted at C-6 in human foetal gastrointestinal mucins.

Hounsell EF, Lawson AM, Feeney J, Cashmore GC, Kane DP, Stoll M, Feizi T.

Biochem J. 1988 Dec 1;256(2):397-401.

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