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Items: 5

1.

Different monocyte phenotypes result in proresolving macrophages in conjugated linoleic acid-induced attenuated progression and regression of atherosclerosis.

Bruen R, Curley S, Kajani S, Lynch G, O'Reilly ME, Dillon ET, Fitzsimons S, Mthunzi L, McGillicuddy FC, Belton O.

FASEB J. 2019 Jul 5:fj201900922R. doi: 10.1096/fj.201900922R. [Epub ahead of print]

PMID:
31284764
2.

Liraglutide attenuates pre-established atherosclerosis in apolipoprotein E deficient mice via regulation of immune cell phenotypes and pro-inflammatory mediators.

Bruen R, Curley S, Kajani S, Lynch G, O'Reilly ME, Dillon ET, Brennan EP, Barry M, Sheehan S, McGillicuddy F, Belton O.

J Pharmacol Exp Ther. 2019 Jul 3. pii: jpet.119.258343. doi: 10.1124/jpet.119.258343. [Epub ahead of print]

3.

Nutritionally Derived Metabolic Cues Typical of the Obese Microenvironment Increase Cholesterol Efflux Capacity of Adipose Tissue Macrophages.

O'Reilly ME, Kajani S, Ralston JC, Lenighan YM, Roche HM, McGillicuddy FC.

Mol Nutr Food Res. 2019 Jan;63(2):e1800713. doi: 10.1002/mnfr.201800713. Epub 2018 Nov 20.

4.

Unravelling HDL-Looking beyond the Cholesterol Surface to the Quality Within.

Kajani S, Curley S, McGillicuddy FC.

Int J Mol Sci. 2018 Jul 6;19(7). pii: E1971. doi: 10.3390/ijms19071971. Review.

5.

Liraglutide dictates macrophage phenotype in apolipoprotein E null mice during early atherosclerosis.

Bruen R, Curley S, Kajani S, Crean D, O'Reilly ME, Lucitt MB, Godson CG, McGillicuddy FC, Belton O.

Cardiovasc Diabetol. 2017 Nov 6;16(1):143. doi: 10.1186/s12933-017-0626-3.

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