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Items: 13

1.

Potentially pathogenic circulating autoantibodies to cardiac troponin are present in hemodialysis patients.

Jefferies HJ, Tertti R, Wittfooth S, Burton JO, Metsärinne K, Pettersson K, McIntyre CW.

Hemodial Int. 2017 Oct;21(4):519-523. doi: 10.1111/hdi.12514. Epub 2016 Nov 20.

PMID:
27866393
2.

Elevated serum free pregnancy-associated plasma protein-A independently predicts mortality in haemodialysis patients but is not associated with recurrent haemodialysis-induced ischaemic myocardial injury.

Jefferies HJ, Tertti R, Wittfooth S, Burton JO, Metsärinne K, Pettersson K, McIntyre CW.

Nephron. 2015;129(3):171-8. doi: 10.1159/000369557. Epub 2015 Mar 6.

PMID:
25766334
3.

Circulating endotoxaemia and frequent haemodialysis schedules.

Jefferies HJ, Crowley LE, Harrison LE, Szeto CC, Li PK, Schiller B, Moran J, McIntyre CW.

Nephron Clin Pract. 2014;128(1-2):141-6. doi: 10.1159/000366519. Epub 2014 Nov 12.

PMID:
25401768
4.

The impact of hemodialysis on segmental and global longitudinal myocardial strain.

Huang SH, Crowley LE, Jefferies HJ, Eldehni MT, Odudu A, McIntyre CW.

Can J Cardiol. 2014 Nov;30(11):1422-8. doi: 10.1016/j.cjca.2014.05.012. Epub 2014 Sep 15.

PMID:
25228129
5.

Troponin T for the detection of dialysis-induced myocardial stunning in hemodialysis patients.

Breidthardt T, Burton JO, Odudu A, Eldehni MT, Jefferies HJ, McIntyre CW.

Clin J Am Soc Nephrol. 2012 Aug;7(8):1285-92. doi: 10.2215/CJN.00460112. Epub 2012 Jul 19.

6.

Angiopoietin-2 levels predict mortality in CKD patients.

David S, John SG, Jefferies HJ, Sigrist MK, Kümpers P, Kielstein JT, Haller H, McIntyre CW.

Nephrol Dial Transplant. 2012 May;27(5):1867-72. doi: 10.1093/ndt/gfr551. Epub 2011 Oct 4.

PMID:
21976741
7.

Frequent hemodialysis schedules are associated with reduced levels of dialysis-induced cardiac injury (myocardial stunning).

Jefferies HJ, Virk B, Schiller B, Moran J, McIntyre CW.

Clin J Am Soc Nephrol. 2011 Jun;6(6):1326-32. doi: 10.2215/CJN.05200610. Epub 2011 May 19.

8.

Individualised dialysate temperature improves intradialytic haemodynamics and abrogates haemodialysis-induced myocardial stunning, without compromising tolerability.

Jefferies HJ, Burton JO, McIntyre CW.

Blood Purif. 2011;32(1):63-8. doi: 10.1159/000324199. Epub 2011 Feb 24.

PMID:
21346338
9.

Circulating endotoxemia: a novel factor in systemic inflammation and cardiovascular disease in chronic kidney disease.

McIntyre CW, Harrison LE, Eldehni MT, Jefferies HJ, Szeto CC, John SG, Sigrist MK, Burton JO, Hothi D, Korsheed S, Owen PJ, Lai KB, Li PK.

Clin J Am Soc Nephrol. 2011 Jan;6(1):133-41. doi: 10.2215/CJN.04610510. Epub 2010 Sep 28.

10.

Tissue-advanced glycation end product concentration in dialysis patients.

McIntyre NJ, Chesterton LJ, John SG, Jefferies HJ, Burton JO, Taal MW, Fluck RJ, McIntyre CW.

Clin J Am Soc Nephrol. 2010 Jan;5(1):51-5. doi: 10.2215/CJN.05350709. Epub 2009 Nov 5.

11.

Hemodialysis-induced repetitive myocardial injury results in global and segmental reduction in systolic cardiac function.

Burton JO, Jefferies HJ, Selby NM, McIntyre CW.

Clin J Am Soc Nephrol. 2009 Dec;4(12):1925-31. doi: 10.2215/CJN.04470709. Epub 2009 Oct 1.

12.

Hemodialysis-induced cardiac injury: determinants and associated outcomes.

Burton JO, Jefferies HJ, Selby NM, McIntyre CW.

Clin J Am Soc Nephrol. 2009 May;4(5):914-20. doi: 10.2215/CJN.03900808. Epub 2009 Apr 8.

13.

Advances in the cardiovascular assessment of patients with chronic kidney disease.

McIntyre CW, John SG, Jefferies HJ.

NDT Plus. 2008 Dec;1(6):383-391. doi: 10.1093/ndtplus/sfn146. Epub 2008 Sep 8.

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