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Items: 26


Unexpected Transcripts in Tn7 orf19.2646 C. albicans Mutant Lead to Low Fungal Burden Phenotype In vivo.

Pierrehumbert A, Ischer F, Coste AT.

Front Microbiol. 2017 May 16;8:873. doi: 10.3389/fmicb.2017.00873. eCollection 2017.


Adaptation of a Gaussia princeps Luciferase reporter system in Candida albicans for in vivo detection in the Galleria mellonella infection model.

Delarze E, Ischer F, Sanglard D, Coste AT.

Virulence. 2015;6(7):684-93. doi: 10.1080/21505594.2015.1081330.


Examining the virulence of Candida albicans transcription factor mutants using Galleria mellonella and mouse infection models.

Amorim-Vaz S, Delarze E, Ischer F, Sanglard D, Coste AT.

Front Microbiol. 2015 May 5;6:367. doi: 10.3389/fmicb.2015.00367. eCollection 2015.


Molecular mechanisms of drug resistance in clinical Candida species isolated from Tunisian hospitals.

Eddouzi J, Parker JE, Vale-Silva LA, Coste A, Ischer F, Kelly S, Manai M, Sanglard D.

Antimicrob Agents Chemother. 2013 Jul;57(7):3182-93. doi: 10.1128/AAC.00555-13. Epub 2013 Apr 29.


Gain-of-function mutations in PDR1, a regulator of antifungal drug resistance in Candida glabrata, control adherence to host cells.

Vale-Silva L, Ischer F, Leibundgut-Landmann S, Sanglard D.

Infect Immun. 2013 May;81(5):1709-20. doi: 10.1128/IAI.00074-13. Epub 2013 Mar 4.


Identification and functional characterization of Rca1, a transcription factor involved in both antifungal susceptibility and host response in Candida albicans.

Vandeputte P, Pradervand S, Ischer F, Coste AT, Ferrari S, Harshman K, Sanglard D.

Eukaryot Cell. 2012 Jul;11(7):916-31. doi: 10.1128/EC.00134-12. Epub 2012 May 11.


Azole resistance by loss of function of the sterol Δ⁵,⁶-desaturase gene (ERG3) in Candida albicans does not necessarily decrease virulence.

Vale-Silva LA, Coste AT, Ischer F, Parker JE, Kelly SL, Pinto E, Sanglard D.

Antimicrob Agents Chemother. 2012 Apr;56(4):1960-8. doi: 10.1128/AAC.05720-11. Epub 2012 Jan 17.


In vivo systematic analysis of Candida albicans Zn2-Cys6 transcription factors mutants for mice organ colonization.

Vandeputte P, Ischer F, Sanglard D, Coste AT.

PLoS One. 2011;6(10):e26962. doi: 10.1371/journal.pone.0026962. Epub 2011 Oct 31.


Genetic dissection of azole resistance mechanisms in Candida albicans and their validation in a mouse model of disseminated infection.

MacCallum DM, Coste A, Ischer F, Jacobsen MD, Odds FC, Sanglard D.

Antimicrob Agents Chemother. 2010 Apr;54(4):1476-83. doi: 10.1128/AAC.01645-09. Epub 2010 Jan 19.


Gain of function mutations in CgPDR1 of Candida glabrata not only mediate antifungal resistance but also enhance virulence.

Ferrari S, Ischer F, Calabrese D, Posteraro B, Sanguinetti M, Fadda G, Rohde B, Bauser C, Bader O, Sanglard D.

PLoS Pathog. 2009 Jan;5(1):e1000268. doi: 10.1371/journal.ppat.1000268. Epub 2009 Jan 16.


Divergent functions of three Candida albicans zinc-cluster transcription factors (CTA4, ASG1 and CTF1) complementing pleiotropic drug resistance in Saccharomyces cerevisiae.

Coste AT, Ramsdale M, Ischer F, Sanglard D.

Microbiology. 2008 May;154(Pt 5):1491-1501. doi: 10.1099/mic.0.2007/016063-0.


Multiple resistance mechanisms to azole antifungals in yeast clinical isolates.

Sanglard D, Ischer F, Calabrese D, Micheli M, Bille J.

Drug Resist Updat. 1998;1(4):255-65.


A mutation in Tac1p, a transcription factor regulating CDR1 and CDR2, is coupled with loss of heterozygosity at chromosome 5 to mediate antifungal resistance in Candida albicans.

Coste A, Turner V, Ischer F, Morschhäuser J, Forche A, Selmecki A, Berman J, Bille J, Sanglard D.

Genetics. 2006 Apr;172(4):2139-56. Epub 2006 Feb 1.


Candida albicans mutations in the ergosterol biosynthetic pathway and resistance to several antifungal agents.

Sanglard D, Ischer F, Parkinson T, Falconer D, Bille J.

Antimicrob Agents Chemother. 2003 Aug;47(8):2404-12.


Calcineurin A of Candida albicans: involvement in antifungal tolerance, cell morphogenesis and virulence.

Sanglard D, Ischer F, Marchetti O, Entenza J, Bille J.

Mol Microbiol. 2003 May;48(4):959-76.


The ATP binding cassette transporter gene CgCDR1 from Candida glabrata is involved in the resistance of clinical isolates to azole antifungal agents.

Sanglard D, Ischer F, Calabrese D, Majcherczyk PA, Bille J.

Antimicrob Agents Chemother. 1999 Nov;43(11):2753-65.


High-frequency endonuclease (REA) typing: results from the WHO collaborative study group on subtyping of Listeria monocytogenes.

Gerner-Smidt P, Boerlin P, Ischer F, Schmidt J.

Int J Food Microbiol. 1996 Oct;32(3):313-24.


Susceptibilities of Candida albicans multidrug transporter mutants to various antifungal agents and other metabolic inhibitors.

Sanglard D, Ischer F, Monod M, Bille J.

Antimicrob Agents Chemother. 1996 Oct;40(10):2300-5.


Mechanisms of resistance to azole antifungal agents in Candida albicans isolates from AIDS patients involve specific multidrug transporters.

Sanglard D, Kuchler K, Ischer F, Pagani JL, Monod M, Bille J.

Antimicrob Agents Chemother. 1995 Nov;39(11):2378-86.


Typing Listeria monocytogenes: a comparison of random amplification of polymorphic DNA with 5 other methods.

Boerlin P, Bannerman E, Ischer F, Rocourt J, Bille J.

Res Microbiol. 1995 Jan;146(1):35-49.


Characterization of Listeria strains from a foodborne listeriosis outbreak by rDNA gene restriction patterns compared to four other typing methods.

Nocera D, Altwegg M, Martinetti Lucchini G, Bannerman E, Ischer F, Rocourt J, Bille J.

Eur J Clin Microbiol Infect Dis. 1993 Mar;12(3):162-9.


Problems related to the administration of ACTH.

Hamburger C, Sprechler M, Brøchner-Mortensen K, ischer F, Videbaek A.

Acta Endocrinol (Copenh). 1952 Aug;10(4):357-72. No abstract available.


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