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Items: 6

1.

Non-spa-typeable clinical Staphylococcus aureus strains are naturally occurring protein A mutants.

Baum C, Haslinger-Löffler B, Westh H, Boye K, Peters G, Neumann C, Kahl BC.

J Clin Microbiol. 2009 Nov;47(11):3624-9. doi: 10.1128/JCM.00941-09. Epub 2009 Sep 16.

2.

Expression of Pls (plasmin sensitive) in Staphylococcus aureus negative for pls reduces adherence and cellular invasion and acts by steric hindrance.

Hussain M, Schäfer D, Juuti KM, Peters G, Haslinger-Löffler B, Kuusela PI, Sinha B.

J Infect Dis. 2009 Jul 1;200(1):107-17. doi: 10.1086/599359.

PMID:
19473097
3.

Multiple effects of HMG-CoA reductase inhibitors (statins) besides their lipid-lowering function.

Haslinger-Löffler B.

Kidney Int. 2008 Sep;74(5):553-5. doi: 10.1038/ki.2008.323.

4.

Staphylococcal infections impair the mesothelial fibrinolytic system: the role of cell death and cytokine release.

Haslinger-Löffler B, Brück M, Grundmeier M, Peters G, Sinha B.

Thromb Haemost. 2007 Oct;98(4):813-22. Erratum in: Thromb Haemost. 2008 Feb;99(2):455.

PMID:
17938806
5.

Staphylococcus aureus induces caspase-independent cell death in human peritoneal mesothelial cells.

Haslinger-Löffler B, Wagner B, Brück M, Strangfeld K, Grundmeier M, Fischer U, Völker W, Peters G, Schulze-Osthoff K, Sinha B.

Kidney Int. 2006 Sep;70(6):1089-98. Epub 2006 Jul 26.

6.

Multiple virulence factors are required for Staphylococcus aureus-induced apoptosis in endothelial cells.

Haslinger-Löffler B, Kahl BC, Grundmeier M, Strangfeld K, Wagner B, Fischer U, Cheung AL, Peters G, Schulze-Osthoff K, Sinha B.

Cell Microbiol. 2005 Aug;7(8):1087-97.

PMID:
16008576

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