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Items: 7


TLR-independent type I interferon induction in response to an extracellular bacterial pathogen via intracellular recognition of its DNA.

Charrel-Dennis M, Latz E, Halmen KA, Trieu-Cuot P, Fitzgerald KA, Kasper DL, Golenbock DT.

Cell Host Microbe. 2008 Dec 11;4(6):543-54. doi: 10.1016/j.chom.2008.11.002.


Phagocytosis and intracellular killing of MD-2 opsonized gram-negative bacteria depend on TLR4 signaling.

Jain V, Halle A, Halmen KA, Lien E, Charrel-Dennis M, Ram S, Golenbock DT, Visintin A.

Blood. 2008 May 1;111(9):4637-45. doi: 10.1182/blood-2007-11-126862. Epub 2008 Jan 18.


Malaria hemozoin is immunologically inert but radically enhances innate responses by presenting malaria DNA to Toll-like receptor 9.

Parroche P, Lauw FN, Goutagny N, Latz E, Monks BG, Visintin A, Halmen KA, Lamphier M, Olivier M, Bartholomeu DC, Gazzinelli RT, Golenbock DT.

Proc Natl Acad Sci U S A. 2007 Feb 6;104(6):1919-24. Epub 2007 Jan 29.


MD-2 expression is not required for cell surface targeting of Toll-like receptor 4 (TLR4).

Visintin A, Halmen KA, Khan N, Monks BG, Golenbock DT, Lien E.

J Leukoc Biol. 2006 Dec;80(6):1584-92. Epub 2006 Aug 31.



Visintin A, Iliev DB, Monks BG, Halmen KA, Golenbock DT.

Immunobiology. 2006;211(6-8):437-47. Epub 2006 Jul 18. Review.


Meningococcal porin PorB binds to TLR2 and requires TLR1 for signaling.

Massari P, Visintin A, Gunawardana J, Halmen KA, King CA, Golenbock DT, Wetzler LM.

J Immunol. 2006 Feb 15;176(4):2373-80.


Pharmacological inhibition of endotoxin responses is achieved by targeting the TLR4 coreceptor, MD-2.

Visintin A, Halmen KA, Latz E, Monks BG, Golenbock DT.

J Immunol. 2005 Nov 15;175(10):6465-72.

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