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Items: 29

1.

Subcellular localization of the chemotherapeutic agent doxorubicin in renal epithelial cells and in tumor cells using correlative light and electron microscopy.

Asam C, Buerger K, Felthaus O, Rachel R, Prantl L, Witzgall R, Aung T, Haerteis S.

Clin Hemorheol Microcirc. 2019 Sep 18. doi: 10.3233/CH-199212. [Epub ahead of print]

PMID:
31561340
2.

Ion channels in sarcoma: pathophysiology and treatment options.

Aung T, Asam C, Haerteis S.

Pflugers Arch. 2019 Sep;471(9):1163-1171. doi: 10.1007/s00424-019-02299-8. Epub 2019 Aug 3. Review.

PMID:
31377822
3.

Urokinase-type plasminogen activator (uPA) is not essential for epithelial sodium channel (ENaC)-mediated sodium retention in experimental nephrotic syndrome.

Bohnert BN, Daiminger S, Wörn M, Sure F, Staudner T, Ilyaskin AV, Batbouta F, Janessa A, Schneider JC, Essigke D, Kanse S, Haerteis S, Korbmacher C, Artunc F.

Acta Physiol (Oxf). 2019 Dec;227(4):e13286. doi: 10.1111/apha.13286. Epub 2019 May 20.

PMID:
31006168
4.

Bile acids inhibit human purinergic receptor P2X4 in a heterologous expression system.

Ilyaskin AV, Sure F, Nesterov V, Haerteis S, Korbmacher C.

J Gen Physiol. 2019 Jun 3;151(6):820-833. doi: 10.1085/jgp.201812291. Epub 2019 Apr 15.

PMID:
30988062
5.

The degenerin region of the human bile acid-sensitive ion channel (BASIC) is involved in channel inhibition by calcium and activation by bile acids.

Ilyaskin AV, Kirsch SA, Böckmann RA, Sticht H, Korbmacher C, Haerteis S, Diakov A.

Pflugers Arch. 2018 Jul;470(7):1087-1102. doi: 10.1007/s00424-018-2142-z. Epub 2018 Mar 27.

PMID:
29589117
6.

Plasma kallikrein activates the epithelial sodium channel in vitro but is not essential for volume retention in nephrotic mice.

Haerteis S, Schork A, Dörffel T, Bohnert BN, Nacken R, Wörn M, Xiao M, Essigke D, Janessa A, Schmaier AH, Feener EP, Häring HU, Bertog M, Korbmacher C, Artunc F.

Acta Physiol (Oxf). 2018 Sep;224(1):e13060. doi: 10.1111/apha.13060. Epub 2018 Mar 25.

PMID:
29489063
7.

Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome.

Bohnert BN, Menacher M, Janessa A, Wörn M, Schork A, Daiminger S, Kalbacher H, Häring HU, Daniel C, Amann K, Sure F, Bertog M, Haerteis S, Korbmacher C, Artunc F.

Kidney Int. 2018 Jan;93(1):159-172. doi: 10.1016/j.kint.2017.07.023. Epub 2017 Oct 14.

PMID:
29042083
8.

Erratum to: Sensitisation of TRPV4 by PAR2 is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase.

Sostegni S, Diakov A, McIntyre P, Bunnett N, Korbmacher C, Haerteis S.

Pflugers Arch. 2017 Jun;469(5-6):845-846. doi: 10.1007/s00424-017-1973-3. No abstract available.

PMID:
28389775
9.

Bile acids potentiate proton-activated currents in Xenopus laevis oocytes expressing human acid-sensing ion channel (ASIC1a).

Ilyaskin AV, Diakov A, Korbmacher C, Haerteis S.

Physiol Rep. 2017 Feb;5(3). pii: e13132. doi: 10.14814/phy2.13132.

10.

Activation of the Human Epithelial Sodium Channel (ENaC) by Bile Acids Involves the Degenerin Site.

Ilyaskin AV, Diakov A, Korbmacher C, Haerteis S.

J Biol Chem. 2016 Sep 16;291(38):19835-47. doi: 10.1074/jbc.M116.726471. Epub 2016 Aug 3.

11.

Association of Plasminuria with Overhydration in Patients with CKD.

Schork A, Woern M, Kalbacher H, Voelter W, Nacken R, Bertog M, Haerteis S, Korbmacher C, Heyne N, Peter A, Häring HU, Artunc F.

Clin J Am Soc Nephrol. 2016 May 6;11(5):761-9. doi: 10.2215/CJN.12261115. Epub 2016 Mar 1.

12.

Atomic force microscopy imaging reveals the formation of ASIC/ENaC cross-clade ion channels.

Jeggle P, Smith ES, Stewart AP, Haerteis S, Korbmacher C, Edwardson JM.

Biochem Biophys Res Commun. 2015 Aug 14;464(1):38-44. doi: 10.1016/j.bbrc.2015.05.091. Epub 2015 May 29.

PMID:
26032502
13.

Neutrophil Elastase Activates Protease-activated Receptor-2 (PAR2) and Transient Receptor Potential Vanilloid 4 (TRPV4) to Cause Inflammation and Pain.

Zhao P, Lieu T, Barlow N, Sostegni S, Haerteis S, Korbmacher C, Liedtke W, Jimenez-Vargas NN, Vanner SJ, Bunnett NW.

J Biol Chem. 2015 May 29;290(22):13875-87. doi: 10.1074/jbc.M115.642736. Epub 2015 Apr 15.

14.

The bile acid receptor TGR5 activates the TRPA1 channel to induce itch in mice.

Lieu T, Jayaweera G, Zhao P, Poole DP, Jensen D, Grace M, McIntyre P, Bron R, Wilson YM, Krappitz M, Haerteis S, Korbmacher C, Steinhoff MS, Nassini R, Materazzi S, Geppetti P, Corvera CU, Bunnett NW.

Gastroenterology. 2014 Dec;147(6):1417-28. doi: 10.1053/j.gastro.2014.08.042. Epub 2014 Sep 3.

15.

Cathepsin S causes inflammatory pain via biased agonism of PAR2 and TRPV4.

Zhao P, Lieu T, Barlow N, Metcalf M, Veldhuis NA, Jensen DD, Kocan M, Sostegni S, Haerteis S, Baraznenok V, Henderson I, Lindström E, Guerrero-Alba R, Valdez-Morales EE, Liedtke W, McIntyre P, Vanner SJ, Korbmacher C, Bunnett NW.

J Biol Chem. 2014 Sep 26;289(39):27215-34. doi: 10.1074/jbc.M114.599712. Epub 2014 Aug 12. Erratum in: J Biol Chem. 2014 Dec 26;289(52):35858.

16.
17.

Sensitisation of TRPV4 by PAR2 is independent of intracellular calcium signalling and can be mediated by the biased agonist neutrophil elastase.

Sostegni S, Diakov A, McIntyre P, Bunnett N, Korbmacher C, Haerteis S.

Pflugers Arch. 2015 Apr;467(4):687-701. doi: 10.1007/s00424-014-1539-6. Epub 2014 Jun 8. Erratum in: Pflugers Arch. 2017 Jun;469(5-6):845-846.

PMID:
24906497
18.

Proteolytic activation of the human epithelial sodium channel by trypsin IV and trypsin I involves distinct cleavage sites.

Haerteis S, Krappitz A, Krappitz M, Murphy JE, Bertog M, Krueger B, Nacken R, Chung H, Hollenberg MD, Knecht W, Bunnett NW, Korbmacher C.

J Biol Chem. 2014 Jul 4;289(27):19067-78. doi: 10.1074/jbc.M113.538470. Epub 2014 May 19.

19.

Pharmacological and electrophysiological characterization of the human bile acid-sensitive ion channel (hBASIC).

Lefèvre CM, Diakov A, Haerteis S, Korbmacher C, Gründer S, Wiemuth D.

Pflugers Arch. 2014 Feb;466(2):253-63. doi: 10.1007/s00424-013-1310-4. Epub 2013 Jul 11.

PMID:
23842738
20.

A mutation in the β-subunit of ENaC identified in a patient with cystic fibrosis-like symptoms has a gain-of-function effect.

Rauh R, Soell D, Haerteis S, Diakov A, Nesterov V, Krueger B, Sticht H, Korbmacher C.

Am J Physiol Lung Cell Mol Physiol. 2013 Jan 1;304(1):L43-55. doi: 10.1152/ajplung.00093.2012. Epub 2012 Oct 19.

21.

Plasmin and chymotrypsin have distinct preferences for channel activating cleavage sites in the γ subunit of the human epithelial sodium channel.

Haerteis S, Krappitz M, Diakov A, Krappitz A, Rauh R, Korbmacher C.

J Gen Physiol. 2012 Oct;140(4):375-89. Epub 2012 Sep 10.

22.

Proteolytic activation of the epithelial sodium channel (ENaC) by the cysteine protease cathepsin-S.

Haerteis S, Krappitz M, Bertog M, Krappitz A, Baraznenok V, Henderson I, Lindström E, Murphy JE, Bunnett NW, Korbmacher C.

Pflugers Arch. 2012 Oct;464(4):353-65. Epub 2012 Aug 5.

23.

An inhibitory peptide derived from the α-subunit of the epithelial sodium channel (ENaC) shows a helical conformation.

Haerteis S, Schaal D, Brauer F, Brüschke S, Schweimer K, Rauh R, Sticht H, Rösch P, Schwarzinger S, Korbmacher C.

Cell Physiol Biochem. 2012;29(5-6):761-74. doi: 10.1159/000312590. Epub 2012 May 11.

24.

Four subunits (αβγδ) of the epithelial sodium channel (ENaC) are expressed in the human eye in various locations.

Krueger B, Schlötzer-Schrehardt U, Haerteis S, Zenkel M, Chankiewitz VE, Amann KU, Kruse FE, Korbmacher C.

Invest Ophthalmol Vis Sci. 2012 Feb 2;53(2):596-604. doi: 10.1167/iovs.11-8581.

PMID:
22167092
25.

Atomic force microscopy reveals the architecture of the epithelial sodium channel (ENaC).

Stewart AP, Haerteis S, Diakov A, Korbmacher C, Edwardson JM.

J Biol Chem. 2011 Sep 16;286(37):31944-52. doi: 10.1074/jbc.M111.275289. Epub 2011 Jul 20.

26.

Functional characterization of a partial loss-of-function mutation of the epithelial sodium channel (ENaC) associated with atypical cystic fibrosis.

Huber R, Krueger B, Diakov A, Korbmacher J, Haerteis S, Einsiedel J, Gmeiner P, Azad AK, Cuppens H, Cassiman JJ, Korbmacher C, Rauh R.

Cell Physiol Biochem. 2010;25(1):145-58. doi: 10.1159/000272059. Epub 2009 Dec 22.

27.

Cholesterol depletion of the plasma membrane prevents activation of the epithelial sodium channel (ENaC) by SGK1.

Krueger B, Haerteis S, Yang L, Hartner A, Rauh R, Korbmacher C, Diakov A.

Cell Physiol Biochem. 2009;24(5-6):605-18. doi: 10.1159/000257516. Epub 2009 Nov 4.

28.

The delta-subunit of the epithelial sodium channel (ENaC) enhances channel activity and alters proteolytic ENaC activation.

Haerteis S, Krueger B, Korbmacher C, Rauh R.

J Biol Chem. 2009 Oct 16;284(42):29024-40. doi: 10.1074/jbc.M109.018945. Epub 2009 Aug 28.

29.

Plasmin in nephrotic urine activates the epithelial sodium channel.

Svenningsen P, Bistrup C, Friis UG, Bertog M, Haerteis S, Krueger B, Stubbe J, Jensen ON, Thiesson HC, Uhrenholt TR, Jespersen B, Jensen BL, Korbmacher C, Skøtt O.

J Am Soc Nephrol. 2009 Feb;20(2):299-310. doi: 10.1681/ASN.2008040364. Epub 2008 Dec 10.

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