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Items: 5

1.

MuLV-related endogenous retroviral elements and Flt3 participate in aberrant end-joining events that promote B-cell leukemogenesis.

Johnson RM, Papp E, Grandal I, Kowalski PE, Nutter L, Wong RC, Joseph-George AM, Danska JS, Guidos CJ.

Genes Dev. 2014 Jun 1;28(11):1179-90. doi: 10.1101/gad.240820.114.

2.

Therapeutic potential of spleen tyrosine kinase inhibition for treating high-risk precursor B cell acute lymphoblastic leukemia.

Perova T, Grandal I, Nutter LM, Papp E, Matei IR, Beyene J, Kowalski PE, Hitzler JK, Minden MD, Guidos CJ, Danska JS.

Sci Transl Med. 2014 May 14;6(236):236ra62. doi: 10.1126/scitranslmed.3008661.

3.

The RAG-1/2 endonuclease causes genomic instability and controls CNS complications of lymphoblastic leukemia in p53/Prkdc-deficient mice.

Gladdy RA, Taylor MD, Williams CJ, Grandal I, Karaskova J, Squire JA, Rutka JT, Guidos CJ, Danska JS.

Cancer Cell. 2003 Jan;3(1):37-50.

4.

Irradiation promotes V(D)J joining and RAG-dependent neoplastic transformation in SCID T-cell precursors.

Williams CJ, Grandal I, Vesprini DJ, Wojtyra U, Danska JS, Guidos CJ.

Mol Cell Biol. 2001 Jan;21(2):400-13.

5.

V(D)J recombination activates a p53-dependent DNA damage checkpoint in scid lymphocyte precursors.

Guidos CJ, Williams CJ, Grandal I, Knowles G, Huang MT, Danska JS.

Genes Dev. 1996 Aug 15;10(16):2038-54.

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