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Items: 49

1.

Porphyromonas gingivalis Sphingolipid Synthesis Limits the Host Inflammatory Response.

Rocha FG, Moye ZD, Ottenberg G, Tang P, Campopiano DJ, Gibson FC 3rd, Davey ME.

J Dent Res. 2020 Feb 27:22034520908784. doi: 10.1177/0022034520908784. [Epub ahead of print]

PMID:
32105543
2.

Cell Wall Hydrolytic Enzymes Enhance Antimicrobial Drug Activity Against Mycobacterium.

Gustine JN, Au MB, Haserick JR, Hett EC, Rubin EJ, Gibson FC 3rd, Deng LL.

Curr Microbiol. 2019 Apr;76(4):398-409. doi: 10.1007/s00284-018-1620-z. Epub 2019 Jan 2.

PMID:
30603964
3.

Immunization with cell-free-generated vaccine protects from Porphyromonas gingivalis-induced alveolar bone loss.

Huang N, Shimomura E, Yin G, Tran C, Sato A, Steiner A, Heibeck T, Tam M, Fairman J, Gibson FC 3rd.

J Clin Periodontol. 2019 Feb;46(2):197-205. doi: 10.1111/jcpe.13047. Epub 2019 Jan 31.

PMID:
30578564
4.

Linkage of Infection to Adverse Systemic Complications: Periodontal Disease, Toll-Like Receptors, and Other Pattern Recognition Systems.

Wallet SM, Puri V, Gibson FC.

Vaccines (Basel). 2018 Apr 5;6(2). pii: E21. doi: 10.3390/vaccines6020021. Review.

5.

Porphyromonas gingivalis-mediated signaling through TLR4 mediates persistent HIV infection of primary macrophages.

Agosto LM, Hirnet JB, Michaels DH, Shaik-Dasthagirisaheb YB, Gibson FC, Viglianti G, Henderson AJ.

Virology. 2016 Dec;499:72-81. doi: 10.1016/j.virol.2016.09.007. Epub 2016 Sep 16.

6.

Signaling events in pathogen-induced macrophage foam cell formation.

Shaik-Dasthagirisaheb YB, Mekasha S, He X, Gibson FC 3rd, Ingalls RR.

Pathog Dis. 2016 Aug;74(6). pii: ftw074. doi: 10.1093/femspd/ftw074. Epub 2016 Jul 31.

7.

Immunologic environment influences macrophage response to Porphyromonas gingivalis.

Papadopoulos G, Shaik-Dasthagirisaheb YB, Huang N, Viglianti GA, Henderson AJ, Kantarci A, Gibson FC 3rd.

Mol Oral Microbiol. 2017 Jun;32(3):250-261. doi: 10.1111/omi.12168. Epub 2016 Aug 26.

8.

Liver X receptors contribute to periodontal pathogen-elicited inflammation and oral bone loss.

Huang N, Shaik-Dasthagirisaheb YB, LaValley MP, Gibson FC 3rd.

Mol Oral Microbiol. 2015 Dec;30(6):438-50. doi: 10.1111/omi.12103. Epub 2015 Jun 9.

9.

Distinct gene signatures in aortic tissue from ApoE-/- mice exposed to pathogens or Western diet.

Kramer CD, Weinberg EO, Gower AC, He X, Mekasha S, Slocum C, Beaulieu LM, Wetzler L, Alekseyev Y, Gibson FC 3rd, Freedman JE, Ingalls RR, Genco CA.

BMC Genomics. 2014 Dec 24;15:1176. doi: 10.1186/1471-2164-15-1176.

10.

Aging and contribution of MyD88 and TRIF to expression of TLR pathway-associated genes following stimulation with Porphyromonas gingivalis.

Shaik-Dasthagirisaheb YB, Huang N, Weinberg EO, Shen SS, Genco CA, Gibson FC 3rd.

J Periodontal Res. 2015 Feb;50(1):89-102. doi: 10.1111/jre.12185. Epub 2014 May 24.

11.

Immuno-pathogenesis of Periodontal Disease: Current and Emerging Paradigms.

Huang N, Gibson FC 3rd.

Curr Oral Health Rep. 2014 Jun 1;1(2):124-132.

12.

Inflammatory response to Porphyromonas gingivalis partially requires interferon regulatory factor (IRF) 3.

Shaik-Dasthagirisaheb YB, Huang N, Gibson FC 3rd.

Innate Immun. 2014 Apr;20(3):312-9. doi: 10.1177/1753425913492180. Epub 2013 Jun 26.

13.

Macrophage-specific TLR2 signaling mediates pathogen-induced TNF-dependent inflammatory oral bone loss.

Papadopoulos G, Weinberg EO, Massari P, Gibson FC 3rd, Wetzler LM, Morgan EF, Genco CA.

J Immunol. 2013 Feb 1;190(3):1148-57. doi: 10.4049/jimmunol.1202511. Epub 2012 Dec 21.

14.

Role of MyD88-dependent and MyD88-independent signaling in Porphyromonas gingivalis-elicited macrophage foam cell formation.

Shaik-Dasthagirisaheb YB, Huang N, Baer MT, Gibson FC 3rd.

Mol Oral Microbiol. 2013 Feb;28(1):28-39. doi: 10.1111/omi.12003. Epub 2012 Oct 8.

15.

Protective role for TLR4 signaling in atherosclerosis progression as revealed by infection with a common oral pathogen.

Hayashi C, Papadopoulos G, Gudino CV, Weinberg EO, Barth KR, Madrigal AG, Chen Y, Ning H, LaValley M, Gibson FC 3rd, Hamilton JA, Genco CA.

J Immunol. 2012 Oct 1;189(7):3681-8. Epub 2012 Sep 5.

16.

Porphyromonas gingivalis accelerates inflammatory atherosclerosis in the innominate artery of ApoE deficient mice.

Hayashi C, Viereck J, Hua N, Phinikaridou A, Madrigal AG, Gibson FC 3rd, Hamilton JA, Genco CA.

Atherosclerosis. 2011 Mar;215(1):52-9. doi: 10.1016/j.atherosclerosis.2010.12.009. Epub 2010 Dec 22.

17.

Immune response of macrophages from young and aged mice to the oral pathogenic bacterium Porphyromonas gingivalis.

Shaik-Dasthagirisaheb YB, Kantarci A, Gibson FC 3rd.

Immun Ageing. 2010 Nov 29;7:15. doi: 10.1186/1742-4933-7-15.

18.

Review: Pathogen-induced inflammation at sites distant from oral infection: bacterial persistence and induction of cell-specific innate immune inflammatory pathways.

Hayashi C, Gudino CV, Gibson FC 3rd, Genco CA.

Mol Oral Microbiol. 2010 Oct;25(5):305-16. doi: 10.1111/j.2041-1014.2010.00582.x. Review.

19.

Pathogen-mediated inflammatory atherosclerosis is mediated in part via Toll-like receptor 2-induced inflammatory responses.

Hayashi C, Madrigal AG, Liu X, Ukai T, Goswami S, Gudino CV, Gibson FC 3rd, Genco CA.

J Innate Immun. 2010;2(4):334-43. doi: 10.1159/000314686. Epub 2010 May 10.

20.

Scavenger receptor A is expressed by macrophages in response to Porphyromonas gingivalis, and participates in TNF-alpha expression.

Baer MT, Huang N, Gibson FC 3rd.

Oral Microbiol Immunol. 2009 Dec;24(6):456-63. doi: 10.1111/j.1399-302X.2009.00538.x.

21.

Bacterial fimbriae stimulate proinflammatory activation in the endothelium through distinct TLRs.

Davey M, Liu X, Ukai T, Jain V, Gudino C, Gibson FC 3rd, Golenbock D, Visintin A, Genco CA.

J Immunol. 2008 Feb 15;180(4):2187-95.

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Toll-like receptor 2 plays a critical role in the progression of atherosclerosis that is independent of dietary lipids.

Liu X, Ukai T, Yumoto H, Davey M, Goswami S, Gibson FC 3rd, Genco CA.

Atherosclerosis. 2008 Jan;196(1):146-54. Epub 2007 Apr 26.

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Roles of the host oxidative immune response and bacterial antioxidant rubrerythrin during Porphyromonas gingivalis infection.

Mydel P, Takahashi Y, Yumoto H, Sztukowska M, Kubica M, Gibson FC 3rd, Kurtz DM Jr, Travis J, Collins LV, Nguyen KA, Genco CA, Potempa J.

PLoS Pathog. 2006 Jul;2(7):e76.

28.

Fimbria-dependent activation of pro-inflammatory molecules in Porphyromonas gingivalis infected human aortic endothelial cells.

Takahashi Y, Davey M, Yumoto H, Gibson FC 3rd, Genco CA.

Cell Microbiol. 2006 May;8(5):738-57.

PMID:
16611224
29.

Innate immune signaling and Porphyromonas gingivalis-accelerated atherosclerosis.

Gibson FC 3rd, Yumoto H, Takahashi Y, Chou HH, Genco CA.

J Dent Res. 2006 Feb;85(2):106-21. Review.

PMID:
16434728
30.

Pathogen-accelerated atherosclerosis occurs early after exposure and can be prevented via immunization.

Miyamoto T, Yumoto H, Takahashi Y, Davey M, Gibson FC 3rd, Genco CA.

Infect Immun. 2006 Feb;74(2):1376-80.

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Gingipain-specific IgG in the sera of patients with periodontal disease is necessary for opsonophagocytosis of Porphyromonas gingivalis.

Gibson FC 3rd, Savelli J, Van Dyke TE, Genco CA.

J Periodontol. 2005 Oct;76(10):1629-36.

PMID:
16253083
33.

Porphyromonas gingivalis fimbria-dependent activation of inflammatory genes in human aortic endothelial cells.

Chou HH, Yumoto H, Davey M, Takahashi Y, Miyamoto T, Gibson FC 3rd, Genco CA.

Infect Immun. 2005 Sep;73(9):5367-78.

34.

Inducible nitric oxide synthase mediates bone development and P. gingivalis-induced alveolar bone loss.

Gyurko R, Shoji H, Battaglino RA, Boustany G, Gibson FC 3rd, Genco CA, Stashenko P, Van Dyke TE.

Bone. 2005 Mar;36(3):472-9.

PMID:
15777672
35.

Innate immune recognition of invasive bacteria accelerates atherosclerosis in apolipoprotein E-deficient mice.

Gibson FC 3rd, Hong C, Chou HH, Yumoto H, Chen J, Lien E, Wong J, Genco CA.

Circulation. 2004 Jun 8;109(22):2801-6. Epub 2004 May 3.

PMID:
15123526
36.
37.

Mice lacking inducible nitric oxide synthase demonstrate impaired killing of Porphyromonas gingivalis.

Gyurko R, Boustany G, Huang PL, Kantarci A, Van Dyke TE, Genco CA, Gibson FC 3rd.

Infect Immun. 2003 Sep;71(9):4917-24.

38.
40.

Fimbria-dependent activation of cell adhesion molecule expression in Porphyromonas gingivalis-infected endothelial cells.

Khlgatian M, Nassar H, Chou HH, Gibson FC 3rd, Genco CA.

Infect Immun. 2002 Jan;70(1):257-67.

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IL-2 mediates protection against abscess formation in an experimental model of sepsis.

Tzianabos AO, Russell PR, Onderdonk AB, Gibson FC 3rd, Cywes C, Chan M, Finberg RW, Kasper DL.

J Immunol. 1999 Jul 15;163(2):893-7.

44.

Protection against experimental intraabdominal sepsis by two polysaccharide immunomodulators.

Tzianabos AO, Gibson FC 3rd, Cisneros RL, Kasper DL.

J Infect Dis. 1998 Jul;178(1):200-6.

PMID:
9652441
45.

Cellular mechanism of intraabdominal abscess formation by Bacteroides fragilis.

Gibson FC 3rd, Onderdonk AB, Kasper DL, Tzianabos AO.

J Immunol. 1998 May 15;160(10):5000-6.

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