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Items: 11


The endoplasmic reticulum-mitochondria interface is perturbed in PARK2 knockout mice and patients with PARK2 mutations.

Gautier CA, Erpapazoglou Z, Mouton-Liger F, Muriel MP, Cormier F, Bigou S, Duffaure S, Girard M, Foret B, Iannielli A, Broccoli V, Dalle C, Bohl D, Michel PP, Corvol JC, Brice A, Corti O.

Hum Mol Genet. 2016 Jul 15;25(14):2972-2984. Epub 2016 May 19.


Tissue- and cell-specific mitochondrial defect in Parkin-deficient mice.

Damiano M, Gautier CA, Bulteau AL, Ferrando-Miguel R, Gouarne C, Paoli MG, Pruss R, Auchère F, L'Hermitte-Stead C, Bouillaud F, Brice A, Corti O, Lombès A.

PLoS One. 2014 Jun 24;9(6):e99898. doi: 10.1371/journal.pone.0099898. eCollection 2014.


Mitochondrial dysfunctions in Parkinson's disease.

Gautier CA, Corti O, Brice A.

Rev Neurol (Paris). 2014 May;170(5):339-43. doi: 10.1016/j.neurol.2013.06.003. Epub 2013 Oct 9. Review.


Parkin differently regulates presenilin-1 and presenilin-2 functions by direct control of their promoter transcription.

Duplan E, Sevalle J, Viotti J, Goiran T, Bauer C, Renbaum P, Levy-Lahad E, Gautier CA, Corti O, Leroudier N, Checler F, da Costa CA.

J Mol Cell Biol. 2013 Apr;5(2):132-42. doi: 10.1093/jmcb/mjt003. Epub 2013 Jan 28.


Loss of DJ-1 does not affect mitochondrial respiration but increases ROS production and mitochondrial permeability transition pore opening.

Giaime E, Yamaguchi H, Gautier CA, Kitada T, Shen J.

PLoS One. 2012;7(7):e40501. doi: 10.1371/journal.pone.0040501. Epub 2012 Jul 9.


Regulation of mitochondrial permeability transition pore by PINK1.

Gautier CA, Giaime E, Caballero E, Núñez L, Song Z, Chan D, Villalobos C, Shen J.

Mol Neurodegener. 2012 May 25;7:22. doi: 10.1186/1750-1326-7-22.


Mitochondrial membrane potential decrease caused by loss of PINK1 is not due to proton leak, but to respiratory chain defects.

Amo T, Sato S, Saiki S, Wolf AM, Toyomizu M, Gautier CA, Shen J, Ohta S, Hattori N.

Neurobiol Dis. 2011 Jan;41(1):111-8. doi: 10.1016/j.nbd.2010.08.027. Epub 2010 Sep 15.


PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy.

Matsuda N, Sato S, Shiba K, Okatsu K, Saisho K, Gautier CA, Sou YS, Saiki S, Kawajiri S, Sato F, Kimura M, Komatsu M, Hattori N, Tanaka K.

J Cell Biol. 2010 Apr 19;189(2):211-21. doi: 10.1083/jcb.200910140.


PINK1 is selectively stabilized on impaired mitochondria to activate Parkin.

Narendra DP, Jin SM, Tanaka A, Suen DF, Gautier CA, Shen J, Cookson MR, Youle RJ.

PLoS Biol. 2010 Jan 26;8(1):e1000298. doi: 10.1371/journal.pbio.1000298.


Absence of nigral degeneration in aged parkin/DJ-1/PINK1 triple knockout mice.

Kitada T, Tong Y, Gautier CA, Shen J.

J Neurochem. 2009 Nov;111(3):696-702. doi: 10.1111/j.1471-4159.2009.06350.x. Epub 2009 Aug 19.


Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress.

Gautier CA, Kitada T, Shen J.

Proc Natl Acad Sci U S A. 2008 Aug 12;105(32):11364-9. doi: 10.1073/pnas.0802076105. Epub 2008 Aug 7.

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