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Items: 21

1.

Lack of muscle mTOR kinase activity causes early onset myopathy and compromises whole-body homeostasis.

Zhang Q, Duplany A, Moncollin V, Mouradian S, Goillot E, Mazelin L, Gauthier K, Streichenberger N, Angleraux C, Chen J, Ding S, Schaeffer L, Gangloff YG.

J Cachexia Sarcopenia Muscle. 2019 Feb;10(1):35-53. doi: 10.1002/jcsm.12336. Epub 2018 Nov 21.

2.

Increased Serpina3n release into circulation during glucocorticoid-mediated muscle atrophy.

Gueugneau M, d'Hose D, Barbé C, de Barsy M, Lause P, Maiter D, Bindels LB, Delzenne NM, Schaeffer L, Gangloff YG, Chambon C, Coudy-Gandilhon C, Béchet D, Thissen JP.

J Cachexia Sarcopenia Muscle. 2018 Oct;9(5):929-946. doi: 10.1002/jcsm.12315. Epub 2018 Jul 10.

3.

Resistance exercise initiates mechanistic target of rapamycin (mTOR) translocation and protein complex co-localisation in human skeletal muscle.

Song Z, Moore DR, Hodson N, Ward C, Dent JR, O'Leary MF, Shaw AM, Hamilton DL, Sarkar S, Gangloff YG, Hornberger TA, Spriet LL, Heigenhauser GJ, Philp A.

Sci Rep. 2017 Jul 10;7(1):5028. doi: 10.1038/s41598-017-05483-x.

4.

mTOR inactivation in myocardium from infant mice rapidly leads to dilated cardiomyopathy due to translation defects and p53/JNK-mediated apoptosis.

Mazelin L, Panthu B, Nicot AS, Belotti E, Tintignac L, Teixeira G, Zhang Q, Risson V, Baas D, Delaune E, Derumeaux G, Taillandier D, Ohlmann T, Ovize M, Gangloff YG, Schaeffer L.

J Mol Cell Cardiol. 2016 Aug;97:213-25. doi: 10.1016/j.yjmcc.2016.04.011. Epub 2016 Apr 28.

PMID:
27133769
5.

S6K1 controls pancreatic β cell size independently of intrauterine growth restriction.

Um SH, Sticker-Jantscheff M, Chau GC, Vintersten K, Mueller M, Gangloff YG, Adams RH, Spetz JF, Elghazi L, Pfluger PT, Pende M, Bernal-Mizrachi E, Tauler A, Tschöp MH, Thomas G, Kozma SC.

J Clin Invest. 2015 Jul 1;125(7):2736-47. doi: 10.1172/JCI77030. Epub 2015 Jun 15.

6.

The metabolic checkpoint kinase mTOR is essential for IL-15 signaling during the development and activation of NK cells.

Marçais A, Cherfils-Vicini J, Viant C, Degouve S, Viel S, Fenis A, Rabilloud J, Mayol K, Tavares A, Bienvenu J, Gangloff YG, Gilson E, Vivier E, Walzer T.

Nat Immunol. 2014 Aug;15(8):749-757. doi: 10.1038/ni.2936. Epub 2014 Jun 29.

7.

Arrest of myelination and reduced axon growth when Schwann cells lack mTOR.

Sherman DL, Krols M, Wu LM, Grove M, Nave KA, Gangloff YG, Brophy PJ.

J Neurosci. 2012 Feb 1;32(5):1817-25. doi: 10.1523/JNEUROSCI.4814-11.2012.

8.

Myopathy caused by mammalian target of rapamycin complex 1 (mTORC1) inactivation is not reversed by restoring mitochondrial function.

Romanino K, Mazelin L, Albert V, Conjard-Duplany A, Lin S, Bentzinger CF, Handschin C, Puigserver P, Zorzato F, Schaeffer L, Gangloff YG, Rüegg MA.

Proc Natl Acad Sci U S A. 2011 Dec 20;108(51):20808-13. doi: 10.1073/pnas.1111448109. Epub 2011 Dec 5.

9.

Muscle inactivation of mTOR causes metabolic and dystrophin defects leading to severe myopathy.

Risson V, Mazelin L, Roceri M, Sanchez H, Moncollin V, Corneloup C, Richard-Bulteau H, Vignaud A, Baas D, Defour A, Freyssenet D, Tanti JF, Le-Marchand-Brustel Y, Ferrier B, Conjard-Duplany A, Romanino K, Bauché S, Hantaï D, Mueller M, Kozma SC, Thomas G, Rüegg MA, Ferry A, Pende M, Bigard X, Koulmann N, Schaeffer L, Gangloff YG.

J Cell Biol. 2009 Dec 14;187(6):859-74. doi: 10.1083/jcb.200903131.

10.

Disruption of the mouse mTOR gene leads to early postimplantation lethality and prohibits embryonic stem cell development.

Gangloff YG, Mueller M, Dann SG, Svoboda P, Sticker M, Spetz JF, Um SH, Brown EJ, Cereghini S, Thomas G, Kozma SC.

Mol Cell Biol. 2004 Nov;24(21):9508-16.

11.

Crystal structure of a subcomplex of human transcription factor TFIID formed by TATA binding protein-associated factors hTAF4 (hTAF(II)135) and hTAF12 (hTAF(II)20).

Werten S, Mitschler A, Romier C, Gangloff YG, Thuault S, Davidson I, Moras D.

J Biol Chem. 2002 Nov 22;277(47):45502-9. Epub 2002 Sep 16.

12.

Functional analysis of the TFIID-specific yeast TAF4 (yTAF(II)48) reveals an unexpected organization of its histone-fold domain.

Thuault S, Gangloff YG, Kirchner J, Sanders S, Werten S, Romier C, Weil PA, Davidson I.

J Biol Chem. 2002 Nov 22;277(47):45510-7. Epub 2002 Sep 16.

13.

Distinct mutations in yeast TAF(II)25 differentially affect the composition of TFIID and SAGA complexes as well as global gene expression patterns.

Kirschner DB, vom Baur E, Thibault C, Sanders SL, Gangloff YG, Davidson I, Weil PA, Tora L.

Mol Cell Biol. 2002 May;22(9):3178-93.

14.

The TFIID components human TAF(II)140 and Drosophila BIP2 (TAF(II)155) are novel metazoan homologues of yeast TAF(II)47 containing a histone fold and a PHD finger.

Gangloff YG, Pointud JC, Thuault S, Carré L, Romier C, Muratoglu S, Brand M, Tora L, Couderc JL, Davidson I.

Mol Cell Biol. 2001 Aug;21(15):5109-21.

15.

The histone fold is a key structural motif of transcription factor TFIID.

Gangloff YG, Romier C, Thuault S, Werten S, Davidson I.

Trends Biochem Sci. 2001 Apr;26(4):250-7. Review.

PMID:
11295558
16.

Histone folds mediate selective heterodimerization of yeast TAF(II)25 with TFIID components yTAF(II)47 and yTAF(II)65 and with SAGA component ySPT7.

Gangloff YG, Sanders SL, Romier C, Kirschner D, Weil PA, Tora L, Davidson I.

Mol Cell Biol. 2001 Mar;21(5):1841-53.

17.

Dissecting the interaction network of multiprotein complexes by pairwise coexpression of subunits in E. coli.

Fribourg S, Romier C, Werten S, Gangloff YG, Poterszman A, Moras D.

J Mol Biol. 2001 Feb 16;306(2):363-73.

PMID:
11237605
19.

The human TFIID components TAF(II)135 and TAF(II)20 and the yeast SAGA components ADA1 and TAF(II)68 heterodimerize to form histone-like pairs.

Gangloff YG, Werten S, Romier C, Carré L, Poch O, Moras D, Davidson I.

Mol Cell Biol. 2000 Jan;20(1):340-51.

21.

Synergistic transcriptional activation by TATA-binding protein and hTAFII28 requires specific amino acids of the hTAFII28 histone fold.

Lavigne AC, Gangloff YG, Carré L, Mengus G, Birck C, Poch O, Romier C, Moras D, Davidson I.

Mol Cell Biol. 1999 Jul;19(7):5050-60.

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