Sort by
Items per page

Send to

Choose Destination

Search results

Items: 15


MAP3K19 Is Overexpressed in COPD and Is a Central Mediator of Cigarette Smoke-Induced Pulmonary Inflammation and Lower Airway Destruction.

Boehme SA, Franz-Bacon K, Ludka J, DiTirro DN, Ly TW, Bacon KB.

PLoS One. 2016 Dec 9;11(12):e0167169. doi: 10.1371/journal.pone.0167169. eCollection 2016.


MAP3K19 Is a Novel Regulator of TGF-β Signaling That Impacts Bleomycin-Induced Lung Injury and Pulmonary Fibrosis.

Boehme SA, Franz-Bacon K, DiTirro DN, Ly TW, Bacon KB.

PLoS One. 2016 May 4;11(5):e0154874. doi: 10.1371/journal.pone.0154874. eCollection 2016.


Murine bone marrow-derived mast cells express chemoattractant receptor-homologous molecule expressed on T-helper class 2 cells (CRTh2).

Boehme SA, Franz-Bacon K, Chen EP, Ly TW, Kawakami Y, Bacon KB.

Int Immunol. 2009 Jun;21(6):621-32. doi: 10.1093/intimm/dxp031. Epub 2009 Apr 3.


Antagonism of CRTH2 ameliorates chronic epicutaneous sensitization-induced inflammation by multiple mechanisms.

Boehme SA, Chen EP, Franz-Bacon K, Sásik R, Sprague LJ, Ly TW, Hardiman G, Bacon KB.

Int Immunol. 2009 Jan;21(1):1-17. doi: 10.1093/intimm/dxn118. Epub 2008 Dec 9.


A small molecule CRTH2 antagonist inhibits FITC-induced allergic cutaneous inflammation.

Boehme SA, Franz-Bacon K, Chen EP, Sásik R, Sprague LJ, Ly TW, Hardiman G, Bacon KB.

Int Immunol. 2009 Jan;21(1):81-93. doi: 10.1093/intimm/dxn127. Epub 2008 Dec 9.


CRTH2 antagonism significantly ameliorates airway hyperreactivity and downregulates inflammation-induced genes in a mouse model of airway inflammation.

Lukacs NW, Berlin AA, Franz-Bacon K, Sásik R, Sprague LJ, Ly TW, Hardiman G, Boehme SA, Bacon KB.

Am J Physiol Lung Cell Mol Physiol. 2008 Nov;295(5):L767-79. doi: 10.1152/ajplung.90351.2008. Epub 2008 Aug 29.


Human thymocytes express CCR-3 and are activated by eotaxin.

Franz-Bacon K, Dairaghi DJ, Boehme SA, Sullivan SK, Schall TJ, Conlon PJ, Taylor N, Bacon KB.

Blood. 1999 May 15;93(10):3233-40.


Macrophage inflammatory protein-1beta induces migration and activation of human thymocytes.

Dairaghi DJ, Franz-Bacon K, Callas E, Cupp J, Schall TJ, Tamraz SA, Boehme SA, Taylor N, Bacon KB.

Blood. 1998 Apr 15;91(8):2905-13.


CCR6, a CC chemokine receptor that interacts with macrophage inflammatory protein 3alpha and is highly expressed in human dendritic cells.

Greaves DR, Wang W, Dairaghi DJ, Dieu MC, Saint-Vis B, Franz-Bacon K, Rossi D, Caux C, McClanahan T, Gordon S, Zlotnik A, Schall TJ.

J Exp Med. 1997 Sep 15;186(6):837-44.


HNMP-1: a novel hematopoietic and neural membrane protein differentially regulated in neural development and injury.

Bolin LM, McNeil T, Lucian LA, DeVaux B, Franz-Bacon K, Gorman DM, Zurawski S, Murray R, McClanahan TK.

J Neurosci. 1997 Jul 15;17(14):5493-502.


Identification through bioinformatics of two new macrophage proinflammatory human chemokines: MIP-3alpha and MIP-3beta.

Rossi DL, Vicari AP, Franz-Bacon K, McClanahan TK, Zlotnik A.

J Immunol. 1997 Feb 1;158(3):1033-6. Erratum in: J Immunol 1999 Jul 15;163(2):1091.


Biochemical and genetic characterization of multiple splice variants of the Flt3 ligand.

McClanahan T, Culpepper J, Campbell D, Wagner J, Franz-Bacon K, Mattson J, Tsai S, Luh J, Guimaraes MJ, Mattei MG, Rosnet O, Birnbaum D, Hannum CH.

Blood. 1996 Nov 1;88(9):3371-82.


DNAM-1, a novel adhesion molecule involved in the cytolytic function of T lymphocytes.

Shibuya A, Campbell D, Hannum C, Yssel H, Franz-Bacon K, McClanahan T, Kitamura T, Nicholl J, Sutherland GR, Lanier LL, Phillips JH.

Immunity. 1996 Jun;4(6):573-81.


Molecular cloning of NKB1. A natural killer cell receptor for HLA-B allotypes.

D'Andrea A, Chang C, Franz-Bacon K, McClanahan T, Phillips JH, Lanier LL.

J Immunol. 1995 Sep 1;155(5):2306-10.


Motor deficit and impairment of synaptic plasticity in mice lacking mGluR1.

Conquet F, Bashir ZI, Davies CH, Daniel H, Ferraguti F, Bordi F, Franz-Bacon K, Reggiani A, Matarese V, Condé F, et al.

Nature. 1994 Nov 17;372(6503):237-43.


Supplemental Content

Loading ...
Support Center