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Items: 6


A conserved and immunodominant lipoprotein of Francisella tularensis is proinflammatory but not essential for virulence.

Forestal CA, Gil H, Monfett M, Noah CE, Platz GJ, Thanassi DG, Benach JL, Furie MB.

Microb Pathog. 2008 Jun;44(6):512-23. doi: 10.1016/j.micpath.2008.01.003. Epub 2008 Jan 17.


Francisella tularensis has a significant extracellular phase in infected mice.

Forestal CA, Malik M, Catlett SV, Savitt AG, Benach JL, Sellati TJ, Furie MB.

J Infect Dis. 2007 Jul 1;196(1):134-7. Epub 2007 May 23.


Mac-1+ cells are the predominant subset in the early hepatic lesions of mice infected with Francisella tularensis.

Rasmussen JW, Cello J, Gil H, Forestal CA, Furie MB, Thanassi DG, Benach JL.

Infect Immun. 2006 Dec;74(12):6590-8. Epub 2006 Sep 25.


Deletion of TolC orthologs in Francisella tularensis identifies roles in multidrug resistance and virulence.

Gil H, Platz GJ, Forestal CA, Monfett M, Bakshi CS, Sellati TJ, Furie MB, Benach JL, Thanassi DG.

Proc Natl Acad Sci U S A. 2006 Aug 22;103(34):12897-902. Epub 2006 Aug 14.


The live vaccine strain of Francisella tularensis replicates in human and murine macrophages but induces only the human cells to secrete proinflammatory cytokines.

Bolger CE, Forestal CA, Italo JK, Benach JL, Furie MB.

J Leukoc Biol. 2005 Jun;77(6):893-7. Epub 2005 Mar 9. Review.


Francisella tularensis selectively induces proinflammatory changes in endothelial cells.

Forestal CA, Benach JL, Carbonara C, Italo JK, Lisinski TJ, Furie MB.

J Immunol. 2003 Sep 1;171(5):2563-70.

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