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Items: 6

1.

Conversion of the LIN-1 ETS protein of Caenorhabditis elegans from a SUMOylated transcriptional repressor to a phosphorylated transcriptional activator.

Leight ER, Murphy JT, Fantz DA, Pepin D, Schneider DL, Ratliff TM, Mohammad DH, Herman MA, Kornfeld K.

Genetics. 2015 Mar;199(3):761-75. doi: 10.1534/genetics.114.172668. Epub 2015 Jan 7.

2.

Docking sites on substrate proteins direct extracellular signal-regulated kinase to phosphorylate specific residues.

Fantz DA, Jacobs D, Glossip D, Kornfeld K.

J Biol Chem. 2001 Jul 20;276(29):27256-65. Epub 2001 May 22.

3.

Mice with a targeted disruption of the H1t gene are fertile and undergo normal changes in structural chromosomal proteins during spermiogenesis.

Fantz DA, Hatfield WR, Horvath G, Kistler MK, Kistler WS.

Biol Reprod. 2001 Feb;64(2):425-31.

PMID:
11159343
4.

Elimination of male germ cells in transgenic mice by the diphtheria toxin A chain gene directed by the histone H1t promoter.

Bartell JG, Fantz DA, Davis T, Dewey MJ, Kistler MK, Kistler WS.

Biol Reprod. 2000 Aug;63(2):409-16.

PMID:
10906044
5.

The testis-specific histone H1t gene is strongly repressed by a G/C-rich region just downstream of the TATA Box.

Clare SE, Fantz DA, Kistler WS, Kistler MK.

J Biol Chem. 1997 Dec 26;272(52):33028-36.

6.

Characterization of the promoter region of the rat testis-specific histone H1t gene.

Clare SE, Hatfield WR, Fantz DA, Kistler WS, Kistler MK.

Biol Reprod. 1997 Jan;56(1):73-82.

PMID:
9002635

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