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Items: 5

1.

Fetal exposure to maternal inflammation interrupts murine intestinal development and increases susceptibility to neonatal intestinal injury.

Elgin TG, Fricke EM, Gong H, Reese J, Mills DA, Kalantera KM, Underwood MA, McElroy SJ.

Dis Model Mech. 2019 Oct 21;12(10). pii: dmm040808. doi: 10.1242/dmm.040808.

2.

Dichotomizing the spectrum of ductal shunt places long-term outcomes research at risk.

Elgin TG, McNamara PJ, Giesinger RE.

J Pediatr. 2019 Jun;209:257. doi: 10.1016/j.jpeds.2019.02.006. Epub 2019 Mar 8. No abstract available.

PMID:
30853193
3.

Loss of murine Paneth cell function alters the immature intestinal microbiome and mimics changes seen in neonatal necrotizing enterocolitis.

Lueschow SR, Stumphy J, Gong H, Kern SL, Elgin TG, Underwood MA, Kalanetra KM, Mills DA, Wong MH, Meyerholz DK, Good M, McElroy SJ.

PLoS One. 2018 Oct 1;13(10):e0204967. doi: 10.1371/journal.pone.0204967. eCollection 2018.

4.

Lipopolysaccharide-induced maternal inflammation induces direct placental injury without alteration in placental blood flow and induces a secondary fetal intestinal injury that persists into adulthood.

Fricke EM, Elgin TG, Gong H, Reese J, Gibson-Corley KN, Weiss RM, Zimmerman K, Bowdler NC, Kalantera KM, Mills DA, Underwood MA, McElroy SJ.

Am J Reprod Immunol. 2018 May;79(5):e12816. doi: 10.1111/aji.12816. Epub 2018 Jan 25.

5.

Development of the Neonatal Intestinal Microbiome and Its Association With Necrotizing Enterocolitis.

Elgin TG, Kern SL, McElroy SJ.

Clin Ther. 2016 Apr;38(4):706-15. doi: 10.1016/j.clinthera.2016.01.005. Epub 2016 Feb 3. Review.

PMID:
26852144

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