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Items: 1 to 50 of 116

1.

TRPC channels: Regulation, dysregulation and contributions to chronic kidney disease.

Dryer SE, Roshanravan H, Kim EY.

Biochim Biophys Acta Mol Basis Dis. 2019 Jun 1;1865(6):1041-1066. doi: 10.1016/j.bbadis.2019.04.001. Epub 2019 Apr 4. Review.

PMID:
30953689
2.

TRPC6 inactivation does not affect loss of renal function in nephrotoxic serum glomerulonephritis in rats, but reduces severity of glomerular lesions.

Kim EY, Shotorbani PY, Dryer SE.

Biochem Biophys Rep. 2019 Jan 8;17:139-150. doi: 10.1016/j.bbrep.2018.12.006. eCollection 2019 Mar.

3.

Mechanisms underlying modulation of podocyte TRPC6 channels by suPAR: Role of NADPH oxidases and Src family tyrosine kinases.

Kim EY, Hassanzadeh Khayyat N, Dryer SE.

Biochim Biophys Acta Mol Basis Dis. 2018 Oct;1864(10):3527-3536. doi: 10.1016/j.bbadis.2018.08.007. Epub 2018 Aug 8.

4.

Permeation and Rectification in Canonical Transient Receptor Potential-6 (TRPC6) Channels.

Dryer SE, Kim EY.

Front Physiol. 2018 Aug 3;9:1055. doi: 10.3389/fphys.2018.01055. eCollection 2018. Review.

5.

Trpc6 inactivation confers protection in a model of severe nephrosis in rats.

Kim EY, Yazdizadeh Shotorbani P, Dryer SE.

J Mol Med (Berl). 2018 Jul;96(7):631-644. doi: 10.1007/s00109-018-1648-3. Epub 2018 May 22.

6.

Changes in podocyte TRPC channels evoked by plasma and sera from patients with recurrent FSGS and by putative glomerular permeability factors.

Kim EY, Roshanravan H, Dryer SE.

Biochim Biophys Acta Mol Basis Dis. 2017 Sep;1863(9):2342-2354. doi: 10.1016/j.bbadis.2017.06.010. Epub 2017 Jun 16.

7.

20-Hydroxyeicosatetraenoic Acid (20-HETE) Modulates Canonical Transient Receptor Potential-6 (TRPC6) Channels in Podocytes.

Roshanravan H, Kim EY, Dryer SE.

Front Physiol. 2016 Aug 31;7:351. doi: 10.3389/fphys.2016.00351. eCollection 2016.

9.
10.

Syndecan-4 ectodomain evokes mobilization of podocyte TRPC6 channels and their associated pathways: An essential role for integrin signaling.

Kim EY, Roshanravan H, Dryer SE.

Biochim Biophys Acta. 2015 Oct;1853(10 Pt A):2610-20. doi: 10.1016/j.bbamcr.2015.07.011. Epub 2015 Jul 17. Erratum in: Biochim Biophys Acta. 2015 Dec;1853(12):3294. Biochim Biophys Acta. 2015 Dec;1853(12):3294.

11.

Pleiotropic signaling evoked by tumor necrosis factor in podocytes.

Abkhezr M, Kim EY, Roshanravan H, Nikolos F, Thomas C, Hagmann H, Benzing T, Dryer SE.

Am J Physiol Renal Physiol. 2015 Jul 15;309(2):F98-108. doi: 10.1152/ajprenal.00146.2015. Epub 2015 May 27. Retraction in: Am J Physiol Renal Physiol. 2016 Jun 1;310(11):F1423.

12.

Glutamate receptors in the kidney.

Dryer SE.

Nephrol Dial Transplant. 2015 Oct;30(10):1630-8. doi: 10.1093/ndt/gfv028. Epub 2015 Mar 31. Review.

PMID:
25829324
13.

Loss of PTEN promotes podocyte cytoskeletal rearrangement, aggravating diabetic nephropathy.

Lin J, Shi Y, Peng H, Shen X, Thomas S, Wang Y, Truong LD, Dryer SE, Hu Z, Xu J.

J Pathol. 2015 May;236(1):30-40. doi: 10.1002/path.4508. Epub 2015 Feb 19.

14.

STAT3 regulates steady-state expression of synaptopodin in cultured mouse podocytes.

Abkhezr M, Dryer SE.

Mol Pharmacol. 2015 Feb;87(2):231-9. doi: 10.1124/mol.114.094508. Epub 2014 Nov 25. Retraction in: Mol Pharmacol. 2016 Sep;90(3):161.

PMID:
25425624
15.
16.

A mutation in TRPC6 channels abolishes their activation by hypoosmotic stretch but does not affect activation by diacylglycerol or G protein signaling cascades.

Wilson C, Dryer SE.

Am J Physiol Renal Physiol. 2014 May 1;306(9):F1018-25. doi: 10.1152/ajprenal.00662.2013. Epub 2014 Mar 5.

17.

ATP acting through P2Y receptors causes activation of podocyte TRPC6 channels: role of podocin and reactive oxygen species.

Roshanravan H, Dryer SE.

Am J Physiol Renal Physiol. 2014 May 1;306(9):F1088-97. doi: 10.1152/ajprenal.00661.2013. Epub 2014 Feb 19.

18.

Angiotensin II activation of TRPC6 channels in rat podocytes requires generation of reactive oxygen species.

Anderson M, Roshanravan H, Khine J, Dryer SE.

J Cell Physiol. 2014 Apr;229(4):434-42. doi: 10.1002/jcp.24461.

PMID:
24037962
19.

NOX2 interacts with podocyte TRPC6 channels and contributes to their activation by diacylglycerol: essential role of podocin in formation of this complex.

Kim EY, Anderson M, Wilson C, Hagmann H, Benzing T, Dryer SE.

Am J Physiol Cell Physiol. 2013 Nov 1;305(9):C960-71. doi: 10.1152/ajpcell.00191.2013. Epub 2013 Aug 15.

20.

Opposing effects of podocin on the gating of podocyte TRPC6 channels evoked by membrane stretch or diacylglycerol.

Anderson M, Kim EY, Hagmann H, Benzing T, Dryer SE.

Am J Physiol Cell Physiol. 2013 Aug 1;305(3):C276-89. doi: 10.1152/ajpcell.00095.2013. Epub 2013 May 8.

22.

Insulin increases surface expression of TRPC6 channels in podocytes: role of NADPH oxidases and reactive oxygen species.

Kim EY, Anderson M, Dryer SE.

Am J Physiol Renal Physiol. 2012 Feb 1;302(3):F298-307. doi: 10.1152/ajprenal.00423.2011. Epub 2011 Oct 26.

23.

The two-pore domain potassium channel KCNK5: induction by estrogen receptor alpha and role in proliferation of breast cancer cells.

Alvarez-Baron CP, Jonsson P, Thomas C, Dryer SE, Williams C.

Mol Endocrinol. 2011 Aug;25(8):1326-36. doi: 10.1210/me.2011-0045. Epub 2011 Jun 16.

24.

Effects of insulin and high glucose on mobilization of slo1 BKCa channels in podocytes.

Kim EY, Dryer SE.

J Cell Physiol. 2011 Sep;226(9):2307-15. doi: 10.1002/jcp.22567.

25.

Functional NMDA receptors with atypical properties are expressed in podocytes.

Anderson M, Suh JM, Kim EY, Dryer SE.

Am J Physiol Cell Physiol. 2011 Jan;300(1):C22-32. doi: 10.1152/ajpcell.00268.2010. Epub 2010 Aug 25. Erratum in: Am J Physiol Cell Physiol. 2012 Mar;302(5):C834.

26.

TRPC6 channels and their binding partners in podocytes: role in glomerular filtration and pathophysiology.

Dryer SE, Reiser J.

Am J Physiol Renal Physiol. 2010 Oct;299(4):F689-701. doi: 10.1152/ajprenal.00298.2010. Epub 2010 Aug 4. Review.

27.

Regulation of podocyte BK(Ca) channels by synaptopodin, Rho, and actin microfilaments.

Kim EY, Suh JM, Chiu YH, Dryer SE.

Am J Physiol Renal Physiol. 2010 Sep;299(3):F594-604. doi: 10.1152/ajprenal.00206.2010. Epub 2010 Jul 14.

28.

Dominant-negative regulation of cell surface expression by a pentapeptide motif at the extreme COOH terminus of an Slo1 calcium-activated potassium channel splice variant.

Chiu YH, Alvarez-Baron C, Kim EY, Dryer SE.

Mol Pharmacol. 2010 Apr;77(4):497-507. doi: 10.1124/mol.109.061929. Epub 2010 Jan 5.

29.

AF17 competes with AF9 for binding to Dot1a to up-regulate transcription of epithelial Na+ channel alpha.

Reisenauer MR, Anderson M, Huang L, Zhang Z, Zhou Q, Kone BC, Morris AP, Lesage GD, Dryer SE, Zhang W.

J Biol Chem. 2009 Dec 18;284(51):35659-69. doi: 10.1074/jbc.M109.038448.

30.

Neph1 regulates steady-state surface expression of Slo1 Ca(2+)-activated K(+) channels: different effects in embryonic neurons and podocytes.

Kim EY, Chiu YH, Dryer SE.

Am J Physiol Cell Physiol. 2009 Dec;297(6):C1379-88. doi: 10.1152/ajpcell.00354.2009. Epub 2009 Sep 30.

31.

The beta1 subunit of Na+/K+-ATPase interacts with BKCa channels and affects their steady-state expression on the cell surface.

Jha S, Dryer SE.

FEBS Lett. 2009 Oct 6;583(19):3109-14. doi: 10.1016/j.febslet.2009.08.039. Epub 2009 Sep 1.

32.

MAGI-1 interacts with Slo1 channel proteins and suppresses Slo1 expression on the cell surface.

Ridgway LD, Kim EY, Dryer SE.

Am J Physiol Cell Physiol. 2009 Jul;297(1):C55-65. doi: 10.1152/ajpcell.00073.2009. Epub 2009 Apr 29.

33.
34.

Nephrin binds to the COOH terminus of a large-conductance Ca2+-activated K+ channel isoform and regulates its expression on the cell surface.

Kim EY, Choi KJ, Dryer SE.

Am J Physiol Renal Physiol. 2008 Jul;295(1):F235-46. doi: 10.1152/ajprenal.00140.2008. Epub 2008 May 14.

35.

A novel actin-binding domain on Slo1 calcium-activated potassium channels is necessary for their expression in the plasma membrane.

Zou S, Jha S, Kim EY, Dryer SE.

Mol Pharmacol. 2008 Feb;73(2):359-68. Epub 2007 Nov 7.

PMID:
17989352
36.
38.

The expression of L-type voltage-gated calcium channels in retinal photoreceptors is under circadian control.

Ko ML, Liu Y, Dryer SE, Ko GY.

J Neurochem. 2007 Oct;103(2):784-92. Epub 2007 Aug 7.

39.
40.
41.

Alternatively spliced C-terminal domains regulate the surface expression of large conductance calcium-activated potassium channels.

Kim EY, Ridgway LD, Zou S, Chiu YH, Dryer SE.

Neuroscience. 2007 Jun 8;146(4):1652-61. Epub 2007 May 2.

42.

Beta1-subunits increase surface expression of a large-conductance Ca2+-activated K+ channel isoform.

Kim EY, Zou S, Ridgway LD, Dryer SE.

J Neurophysiol. 2007 May;97(5):3508-16. Epub 2007 Feb 28.

43.
44.

Delayed synapsing muscles are more severely affected in an experimental model of MuSK-induced myasthenia gravis.

Xu K, Jha S, Hoch W, Dryer SE.

Neuroscience. 2006 Dec;143(3):655-9. Epub 2006 Nov 1.

PMID:
17081697
45.

Evidence for two-pore domain potassium channels in rat cerebral arteries.

Bryan RM Jr, You J, Phillips SC, Andresen JJ, Lloyd EE, Rogers PA, Dryer SE, Marrelli SP.

Am J Physiol Heart Circ Physiol. 2006 Aug;291(2):H770-80. Epub 2006 Mar 24.

48.
49.

Measuring circadian rhythms in olfaction using electroantennograms.

Krishnan P, Dryer SE, Hardin PE.

Methods Enzymol. 2005;393:495-508.

PMID:
15817308
50.

Akt activation is necessary for growth factor-induced trafficking of functional K(Ca) channels in developing parasympathetic neurons.

Chae KS, Martin-Caraballo M, Anderson M, Dryer SE.

J Neurophysiol. 2005 Mar;93(3):1174-82. Epub 2004 Oct 27.

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