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Items: 6

1.

Non-random integration of the HPV genome in cervical cancer.

Schmitz M, Driesch C, Jansen L, Runnebaum IB, Dürst M.

PLoS One. 2012;7(6):e39632. doi: 10.1371/journal.pone.0039632. Epub 2012 Jun 27.

2.

Loss of gene function as a consequence of human papillomavirus DNA integration.

Schmitz M, Driesch C, Beer-Grondke K, Jansen L, Runnebaum IB, Dürst M.

Int J Cancer. 2012 Sep 1;131(5):E593-602. doi: 10.1002/ijc.27433. Epub 2012 Feb 22.

3.

The majority of viral-cellular fusion transcripts in cervical carcinomas cotranscribe cellular sequences of known or predicted genes.

Kraus I, Driesch C, Vinokurova S, Hovig E, Schneider A, von Knebel Doeberitz M, Dürst M.

Cancer Res. 2008 Apr 1;68(7):2514-22. doi: 10.1158/0008-5472.CAN-07-2776.

4.

Type-dependent integration frequency of human papillomavirus genomes in cervical lesions.

Vinokurova S, Wentzensen N, Kraus I, Klaes R, Driesch C, Melsheimer P, Kisseljov F, Dürst M, Schneider A, von Knebel Doeberitz M.

Cancer Res. 2008 Jan 1;68(1):307-13. doi: 10.1158/0008-5472.CAN-07-2754.

5.

Integration of the HPV16 genome does not invariably result in high levels of viral oncogene transcripts.

Häfner N, Driesch C, Gajda M, Jansen L, Kirchmayr R, Runnebaum IB, Dürst M.

Oncogene. 2008 Mar 6;27(11):1610-7. Epub 2007 Sep 10.

PMID:
17828299
6.

[Necessity of increasing autopsy frequency following the introduction of DRGs].

Krukemeyer MG, v d Driesch C, Dankof A, Krenn V, Hansen D, Dietel M.

Pathologe. 2007 Jul;28(4):294-8. German.

PMID:
16838174

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