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Items: 4

1.

Pathogenicity and transmission of triple reassortant H3N2 swine influenza A viruses is attenuated following Turkey embryo propagation.

Raghunath S, Pudupakam RS, Deventhiran J, Tevatia R, Leroith T.

Vet Microbiol. 2017 Mar;201:208-215. doi: 10.1016/j.vetmic.2017.01.030. Epub 2017 Feb 1.

PMID:
28284612
2.

PB1-F2 Protein Does Not Impact the Virulence of Triple-Reassortant H3N2 Swine Influenza Virus in Pigs but Alters Pathogenicity and Transmission in Turkeys.

Deventhiran J, Kumar SR, Raghunath S, Leroith T, Elankumaran S.

J Virol. 2015 Oct 14;90(1):222-31. doi: 10.1128/JVI.01551-15. Print 2016 Jan 1.

3.

A novel pathogenic Mammalian orthoreovirus from diarrheic pigs and Swine blood meal in the United States.

Thimmasandra Narayanappa A, Sooryanarain H, Deventhiran J, Cao D, Ammayappan Venkatachalam B, Kambiranda D, LeRoith T, Heffron CL, Lindstrom N, Hall K, Jobst P, Sexton C, Meng XJ, Elankumaran S.

MBio. 2015 May 19;6(3):e00593-15. doi: 10.1128/mBio.00593-15.

4.

A Time Course for Susceptibility to Staphylococcus aureus Respiratory Infection during Influenza in a Swine Model.

Smith EA, Kumar SR, Deventhiran J, Cecere TE, Leroith T, McGilliard M, Elankumaran S, Mullarky IK.

Influenza Res Treat. 2011;2011:846910. doi: 10.1155/2011/846910. Epub 2012 Jan 11.

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